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1دورية أكاديمية
المؤلفون: Liu Y; Institute of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China; Department of Gynecology, Hubei Women and Children's Hospital, Wuhan, Hubei 430070, China., Yan W; Institute of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China. Electronic address: ywm_net@qq.com., Yuan W; Institute of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China., Wang P; Institute of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China., Huang D; Institute of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China., Luo X; Department of Paediatrics, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 430030 Wuhan, Hubei, China., Ning Q; Institute of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, Wuhan, Hubei 430030, China; Department of Infectious Disease, Tongji Hospital of Tongji Medical College, Huazhong University of Science and Technology, 430030 Wuhan, Hubei, China. Electronic address: qning@vip.sina.com.
المصدر: Clinical immunology (Orlando, Fla.) [Clin Immunol] 2019 Jan; Vol. 198, pp. 79-88. Date of Electronic Publication: 2018 Nov 16.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Academic Press Country of Publication: United States NLM ID: 100883537 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1521-7035 (Electronic) Linking ISSN: 15216616 NLM ISO Abbreviation: Clin Immunol Subsets: MEDLINE
مواضيع طبية MeSH: Hepatitis, Autoimmune/*immunology , T-Lymphocytes, Regulatory/*immunology , Th17 Cells/*immunology, Adoptive Transfer ; Animals ; Disease Progression ; Female ; Humans ; Interleukin-17/physiology ; Male ; Mice ; Mice, Inbred C57BL ; Middle Aged
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2دورية أكاديمية
المؤلفون: Sun W; Department of Respiratory Medicine, the Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Research Unit of Respiratory Disease, Central South University, Changsha, Hunan 410011, China; Hunan Diagnosis and Treatment Center of Respiratory Disease, Changsha, Hunan 410011, China., Xiao B; Department of Emergency, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Institute of Emergency and Difficult Diseases, Central South University, Changsha, Hunan 410011, China., Jia A; Department of Respiratory Medicine, The Affiliated Hospital of Guilin Medical University, Guilin 541001, China., Qiu L; Department of Respiratory Medicine, the Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Research Unit of Respiratory Disease, Central South University, Changsha, Hunan 410011, China; Hunan Diagnosis and Treatment Center of Respiratory Disease, Changsha, Hunan 410011, China., Zeng Q; Department of Respiratory Medicine, the Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Research Unit of Respiratory Disease, Central South University, Changsha, Hunan 410011, China; Hunan Diagnosis and Treatment Center of Respiratory Disease, Changsha, Hunan 410011, China., Liu D; Department of Respiratory Medicine, the Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Research Unit of Respiratory Disease, Central South University, Changsha, Hunan 410011, China; Hunan Diagnosis and Treatment Center of Respiratory Disease, Changsha, Hunan 410011, China., Yuan Y; Department of Respiratory Medicine, the Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Research Unit of Respiratory Disease, Central South University, Changsha, Hunan 410011, China; Hunan Diagnosis and Treatment Center of Respiratory Disease, Changsha, Hunan 410011, China., Jia J; Department of Respiratory Medicine, the Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Research Unit of Respiratory Disease, Central South University, Changsha, Hunan 410011, China; Hunan Diagnosis and Treatment Center of Respiratory Disease, Changsha, Hunan 410011, China., Zhang X; Department of Respiratory Medicine, The Second Hospital, University of South China, 30 Jiefang Road, Hengyang, Hunan 421001, China. Electronic address: zxf96178@126.com., Xiang X; Department of Emergency, The Second Xiangya Hospital, Central South University, Changsha, Hunan 410011, China; Institute of Emergency and Difficult Diseases, Central South University, Changsha, Hunan 410011, China. Electronic address: xudongxiang@csu.edu.cn.
المصدر: Experimental cell research [Exp Cell Res] 2018 Oct 01; Vol. 371 (1), pp. 196-204. Date of Electronic Publication: 2018 Aug 09.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Academic Press Country of Publication: United States NLM ID: 0373226 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1090-2422 (Electronic) Linking ISSN: 00144827 NLM ISO Abbreviation: Exp Cell Res Subsets: MEDLINE
مواضيع طبية MeSH: Asthma/*genetics , DNA-Binding Proteins/*genetics , Lung/*immunology , Suppressor of Cytokine Signaling 3 Protein/*genetics , Th17 Cells/*immunology, Allergens/administration & dosage ; Animals ; Asthma/chemically induced ; Asthma/immunology ; Asthma/pathology ; Cell Differentiation ; DNA-Binding Proteins/immunology ; Disease Models, Animal ; Female ; Gene Expression Regulation ; Humans ; Lipopolysaccharides/administration & dosage ; Lung/pathology ; Mice ; Mice, Inbred C57BL ; Mice, Knockout ; Nuclear Receptor Subfamily 1, Group F, Member 3/genetics ; Nuclear Receptor Subfamily 1, Group F, Member 3/immunology ; Ovalbumin/administration & dosage ; Phosphorylation ; Pyroglyphidae/chemistry ; Pyroglyphidae/immunology ; STAT3 Transcription Factor/genetics ; STAT3 Transcription Factor/immunology ; Severity of Illness Index ; Signal Transduction ; Suppressor of Cytokine Signaling 3 Protein/immunology ; Th17 Cells/pathology