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1دورية أكاديمية
المؤلفون: Marjuki H; Division of Virology, Department of Infectious Diseases, St. Jude Children's Research Hospital, Memphis, TN 38105, USA., Gornitzky A, Marathe BM, Ilyushina NA, Aldridge JR, Desai G, Webby RJ, Webster RG
المصدر: Cellular microbiology [Cell Microbiol] 2011 Apr; Vol. 13 (4), pp. 587-601. Date of Electronic Publication: 2010 Dec 29.
نوع المنشور: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Hindawi Country of Publication: India NLM ID: 100883691 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1462-5822 (Electronic) Linking ISSN: 14625814 NLM ISO Abbreviation: Cell Microbiol Subsets: MEDLINE
مواضيع طبية MeSH: Virus Internalization*, Extracellular Signal-Regulated MAP Kinases/*metabolism , Influenza A virus/*enzymology , Influenza A virus/*physiology , Phosphatidylinositol 3-Kinase/*metabolism , Vacuolar Proton-Translocating ATPases/*metabolism, Animals ; Cell Line ; Enzyme Activation ; Humans ; Hydrogen-Ion Concentration ; Influenza A virus/genetics ; MAP Kinase Kinase Kinases/metabolism ; Protein Isoforms/genetics ; Protein Isoforms/metabolism ; Protein Subunits/genetics ; Protein Subunits/metabolism ; Proto-Oncogene Proteins c-akt/metabolism ; RNA, Small Interfering/metabolism ; Signal Transduction/physiology ; Vacuolar Proton-Translocating ATPases/genetics ; raf Kinases/metabolism
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2دورية أكاديمية
المؤلفون: Ehrhardt C; Institute of Molecular Virology (IMV), ZMBE, Westfaelische-Wilhelms-University, Von-Esmarch-Str. 56, D-48149 Muenster, Germany., Marjuki H, Wolff T, Nürnberg B, Planz O, Pleschka S, Ludwig S
المصدر: Cellular microbiology [Cell Microbiol] 2006 Aug; Vol. 8 (8), pp. 1336-48.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Hindawi Country of Publication: India NLM ID: 100883691 Publication Model: Print Cited Medium: Print ISSN: 1462-5814 (Print) Linking ISSN: 14625814 NLM ISO Abbreviation: Cell Microbiol Subsets: MEDLINE
مواضيع طبية MeSH: Influenza A virus/*pathogenicity , Phosphatidylinositol 3-Kinases/*metabolism, Animals ; Base Sequence ; Cell Line ; DNA, Viral/genetics ; Dogs ; Enhancer Elements, Genetic ; Enzyme Activation ; Humans ; Influenza A virus/immunology ; Influenza A virus/physiology ; Interferon Regulatory Factor-3/genetics ; Interferon Regulatory Factor-3/metabolism ; Phosphatidylinositol Phosphates/metabolism ; Promoter Regions, Genetic ; Protein Serine-Threonine Kinases/metabolism ; Signal Transduction ; Viral Proteins/biosynthesis ; Virus Replication