المؤلفون: Vander Roest M; Biomedical Engineering, Vanderbilt University, Nashville, Tennessee., Krapp C; Epigenetics Institute, Department of Cell and Developmental Biology, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania., Thorvaldsen JL; Epigenetics Institute, Department of Cell and Developmental Biology, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania., Bartolomei MS; Epigenetics Institute, Department of Cell and Developmental Biology, University of Pennsylvania Perelman School of Medicine, Philadelphia, Pennsylvania., Merryman WD; Biomedical Engineering, Vanderbilt University, Nashville, Tennessee.

المصدر: Physiological reports [Physiol Rep] 2019 Oct; Vol. 7 (19), pp. e14244.

نوع المنشور: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

بيانات الدورية: Publisher: published by Wiley Periodicals, Inc. on behalf of the American Physiological Society and The Physiological Society Country of Publication: United States NLM ID: 101607800 Publication Model: Print Cited Medium: Internet ISSN: 2051-817X (Electronic) Linking ISSN: 2051817X NLM ISO Abbreviation: Physiol Rep Subsets: MEDLINE

مواضيع طبية MeSH: Aortic Valve/*pathology , Aortic Valve Stenosis/*genetics , Calcinosis/*genetics , DNA Methylation/*genetics , RNA, Long Noncoding/*genetics, Aging ; Animals ; Aortic Valve/metabolism ; Aortic Valve Stenosis/metabolism ; Calcinosis/metabolism ; Female ; Male ; Mice ; Mice, Inbred C57BL ; RNA, Long Noncoding/metabolism ; Sex Factors ; Up-Regulation

SCR Disease Name: Aortic Valve, Calcification of