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1دورية أكاديمية
المؤلفون: Shimura T; a Department of Environmental Health ; National Institute of Public Health 2-3-6 Minami ; Wako , Saitama , Japan., Sasatani M; b Department of Experimental Oncology ; Research Center for Radiation Genome Medicine ; Research Institute for Radiation Biology and Medicine (RIRBM) ; Hiroshima University , Hiroshima , Japan ., Kawai H; b Department of Experimental Oncology ; Research Center for Radiation Genome Medicine ; Research Institute for Radiation Biology and Medicine (RIRBM) ; Hiroshima University , Hiroshima , Japan ., Kamiya K; b Department of Experimental Oncology ; Research Center for Radiation Genome Medicine ; Research Institute for Radiation Biology and Medicine (RIRBM) ; Hiroshima University , Hiroshima , Japan ., Kobayashi J; c Department of Genome Dynamics , Radiation Biology Center ; Kyoto University ; Kyoto , Japan., Komatsu K; c Department of Genome Dynamics , Radiation Biology Center ; Kyoto University ; Kyoto , Japan., Kunugita N; a Department of Environmental Health ; National Institute of Public Health 2-3-6 Minami ; Wako , Saitama , Japan.
المصدر: Cell cycle (Georgetown, Tex.) [Cell Cycle] 2017; Vol. 16 (24), pp. 2345-2354. Date of Electronic Publication: 2017 Nov 29.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Taylor & Francis Country of Publication: United States NLM ID: 101137841 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1551-4005 (Electronic) Linking ISSN: 15514005 NLM ISO Abbreviation: Cell Cycle Subsets: MEDLINE
مواضيع طبية MeSH: DNA Damage*/radiation effects, Ataxia Telangiectasia Mutated Proteins/*metabolism , Mitochondria/*metabolism, Adenosine Triphosphate/analysis ; Adenosine Triphosphate/metabolism ; Cell Line ; Cell Nucleus/genetics ; Cell Nucleus/metabolism ; DNA Repair/radiation effects ; Electron Transport Complex I/metabolism ; Fibroblasts/cytology ; Fibroblasts/metabolism ; Fibroblasts/radiation effects ; Histones/metabolism ; Humans ; Lung ; Membrane Potential, Mitochondrial/radiation effects ; Mitochondria/radiation effects ; Oxidative Phosphorylation/radiation effects ; Rad51 Recombinase/metabolism ; Radiation, Ionizing ; Reactive Oxygen Species/metabolism
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2دورية أكاديمية
المؤلفون: Shimura T; a Department of Environmental Health , National Institute of Public Health , Wako , Saitama , Japan., Kobayashi J; b Department of Genome Dynamics , Radiation Biology Center, Kyoto University , Kyoto , Japan., Komatsu K; b Department of Genome Dynamics , Radiation Biology Center, Kyoto University , Kyoto , Japan., Kunugita N; a Department of Environmental Health , National Institute of Public Health , Wako , Saitama , Japan.
المصدر: Cell cycle (Georgetown, Tex.) [Cell Cycle] 2016; Vol. 15 (8), pp. 1099-107.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Taylor & Francis Country of Publication: United States NLM ID: 101137841 Publication Model: Print Cited Medium: Internet ISSN: 1551-4005 (Electronic) Linking ISSN: 15514005 NLM ISO Abbreviation: Cell Cycle Subsets: MEDLINE
مواضيع طبية MeSH: Radiation Tolerance*/drug effects , Radiation Tolerance*/radiation effects, Ataxia Telangiectasia Mutated Proteins/*deficiency , Cell Cycle Proteins/*deficiency , Mitochondria/*pathology , Nuclear Proteins/*deficiency, Acetylcysteine/pharmacology ; Apoptosis/drug effects ; Apoptosis/radiation effects ; Ataxia Telangiectasia Mutated Proteins/metabolism ; Cell Cycle Proteins/metabolism ; Cell Nucleus/drug effects ; Cell Nucleus/metabolism ; Cell Nucleus/radiation effects ; Cell Survival/drug effects ; Cell Survival/radiation effects ; DNA Damage ; Dose-Response Relationship, Radiation ; Histones/metabolism ; Humans ; Membrane Potential, Mitochondrial/drug effects ; Membrane Potential, Mitochondrial/radiation effects ; Mitochondria/drug effects ; Mitochondria/metabolism ; Mitochondria/radiation effects ; Mitophagy/drug effects ; Mitophagy/radiation effects ; Models, Biological ; Nuclear Proteins/metabolism ; Reactive Oxygen Species/metabolism
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3دورية أكاديمية
المؤلفون: Kobayashi J; Department of Genome Repair Dynamics, Radiation Biology Center, Kyoto University, Kyoto 606-8501, Japan. Electronic address: jkobayashi@house.rbc.kyoto-u.ac.jp., Saito Y; Department of Genome Repair Dynamics, Radiation Biology Center, Kyoto University, Kyoto 606-8501, Japan., Okui M; Biomedical Engineering Center, Toin University of Yokohama, Yokohama 225-8503, Japan., Miwa N; Department of Genome Repair Dynamics, Radiation Biology Center, Kyoto University, Kyoto 606-8501, Japan., Komatsu K; Department of Genome Repair Dynamics, Radiation Biology Center, Kyoto University, Kyoto 606-8501, Japan.
المصدر: Mutation research. Genetic toxicology and environmental mutagenesis [Mutat Res Genet Toxicol Environ Mutagen] 2015 Apr; Vol. 782, pp. 42-50. Date of Electronic Publication: 2015 Mar 18.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Elsevier Country of Publication: Netherlands NLM ID: 101632149 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1879-3592 (Electronic) Linking ISSN: 13835718 NLM ISO Abbreviation: Mutat Res Genet Toxicol Environ Mutagen Subsets: MEDLINE
مواضيع طبية MeSH: DNA Damage* , Gamma Rays*, Ataxia Telangiectasia Mutated Proteins/*metabolism , Oxidative Stress/*radiation effects , Recombinational DNA Repair/*radiation effects, Blotting, Western ; Cell Cycle Checkpoints/drug effects ; Cell Cycle Checkpoints/radiation effects ; Cell Cycle Proteins/metabolism ; Cell Line, Transformed ; Cell Line, Tumor ; Cells, Cultured ; DNA Repair ; HeLa Cells ; Humans ; Hydrogen Peroxide/pharmacology ; Nuclear Proteins/metabolism ; Oxidants/pharmacology ; Oxidative Stress/drug effects ; Protein Binding/drug effects ; Protein Binding/radiation effects ; Pyocyanine/pharmacology ; Reactive Oxygen Species/metabolism ; Recombinational DNA Repair/drug effects ; Spinocerebellar Ataxias/congenital ; Spinocerebellar Degenerations/genetics ; Spinocerebellar Degenerations/metabolism ; Spinocerebellar Degenerations/pathology
SCR Disease Name: Spinocerebellar ataxia, autosomal recessive 1
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4دورية أكاديمية
المؤلفون: Iwahori S; Virology Division; National Cancer Center Research Institute; Chuohku, Tokyo, Japan., Kohmon D; Department of Cellular Biochemistry; Graduate School of Pharmaceutical Sciences; Kyushu University; Higashiku, Fukuoka, Japan., Kobayashi J; Radiation Biology Center; Kyoto University; Sakyo-ku, Kyoto, Japan., Tani Y; Department of Cellular Biochemistry; Graduate School of Pharmaceutical Sciences; Kyushu University; Higashiku, Fukuoka, Japan., Yugawa T; Virology Division; National Cancer Center Research Institute; Chuohku, Tokyo, Japan., Komatsu K; Radiation Biology Center; Kyoto University; Sakyo-ku, Kyoto, Japan., Kiyono T; Virology Division; National Cancer Center Research Institute; Chuohku, Tokyo, Japan., Sugimoto N; Department of Cellular Biochemistry; Graduate School of Pharmaceutical Sciences; Kyushu University; Higashiku, Fukuoka, Japan., Fujita M; Department of Cellular Biochemistry; Graduate School of Pharmaceutical Sciences; Kyushu University; Higashiku, Fukuoka, Japan.
المصدر: Cell cycle (Georgetown, Tex.) [Cell Cycle] 2014; Vol. 13 (3), pp. 471-81. Date of Electronic Publication: 2013 Nov 26.
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Taylor & Francis Country of Publication: United States NLM ID: 101137841 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1551-4005 (Electronic) Linking ISSN: 15514005 NLM ISO Abbreviation: Cell Cycle Subsets: MEDLINE
مواضيع طبية MeSH: DNA Replication* , S Phase*, Ataxia Telangiectasia Mutated Proteins/*metabolism , Cell Cycle Proteins/*metabolism , DNA/*metabolism, Cell Cycle Proteins/genetics ; Cell Line ; Gene Silencing ; Genomic Instability ; Humans ; Nuclear Proteins/metabolism ; Phosphorylation ; Proteolysis ; S-Phase Kinase-Associated Proteins/metabolism ; Signal Transduction