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    المساهمون: BRICS, Braunschweiger Zentrum für Systembiologie, Rebenring 56, 38106 Braunschweig, Germany., VU University medical center

    المصدر: Shinde, P V, Xu, H C, Maney, S K, Kloetgen, A, Namineni, S, Zhuang, Y, Honke, N, Shaabani, N, Bellora, N, Doerrenberg, M, Trilling, M, Pozdeev, V I, van Rooijen, N, Scheu, S, Pfeffer, K, Crocker, P R, Tanaka, M, Duggimpudi, S, Knolle, P, Heikenwalder, M, Ruland, J R, Mak, T W, Brenner, D, Pandyra, A A, Hoell, J I, Borkhardt, A, Häussinger, D, Lang, K S & Lang, P A 2018, ' Tumor necrosis factormediated survival of CD169+ cells promotes immune activation during vesicular stomatitis virus infection ', Journal of Virology, vol. 92, no. 3, e01637-17 . https://doi.org/10.1128/JVI.01637-17
    CONICET Digital (CONICET)
    Consejo Nacional de Investigaciones Científicas y Técnicas
    instacron:CONICET
    Shinde, P V, Xu, H C, Maney, S K, Kloetgen, A, Namineni, S, Zhuang, Y, Honke, N, Shaabani, N, Bellora, N, Doerrenberg, M, Trilling, M, Pozdeev, V I, van Rooijen, N, Scheu, S, Pfeffer, K, Crocker, P R, Tanaka, M, Duggimpudi, S, Knolle, P, Heikenwalder, M, Ruland, J, Mak, T W, Brenner, D, Pandyra, A A, Hoell, J I, Borkhardt, A, Häussinger, D, Lang, K S & Lang, P A 2018, ' Tumor Necrosis Factor-Mediated Survival of CD169+ Cells Promotes Immune Activation during Vesicular Stomatitis Virus Infection ', Journal of Virology, vol. 92, no. 3, e01637-17, pp. e01637-17 . https://doi.org/10.1128/JVI.01637-17
    Journal of virology
    J. Virol. 92, DOI: 10.1128/JVI.01637-17 (2017)
    Journal of Virology, 92(3):e01637-17. American Society for Microbiology
    Journal of Virology

    مصطلحات موضوعية: 0301 basic medicine, Interferon Type I/immunology, Sialic Acid Binding Ig-like Lectin 1, animal diseases, Transcription Factor RelA/metabolism, viruses, TNF, Medizin, Cellular Response to Infection, Macrophages/immunology, Adaptive Immunity, Virus Replication, INTERFERONS, NF-κB, purl.org/becyt/ford/1 [https], Mice, 0302 clinical medicine, Interferon, Vesicular Stomatitis/immunology, innate immunity, Vesiculovirus/physiology, biology, interferon, Bioquímica y Biología Molecular, Acquired immune system, 3. Good health, ddc, interferons, Receptors, Tumor Necrosis Factor, Type I, Vesicular stomatitis virus, 030220 oncology & carcinogenesis, Interferon Type I, Tumor necrosis factor alpha, Vesicular Stomatitis, NF-ΚB, CIENCIAS NATURALES Y EXACTAS, medicine.drug, INTERFERON, tumor necrosis factor, Immunology, chemical and pharmacologic phenomena, Microbiology, Virus, Ciencias Biológicas, 03 medical and health sciences, Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein/genetics, Virology, medicine, Animals, purl.org/becyt/ford/1.6 [https], Innate immune system, Tumor Necrosis Factor-alpha, TUMOR NECROSIS FACTOR, Macrophages, MALT1, Transcription Factor RelA, Vesiculovirus, biochemical phenomena, metabolism, and nutrition, biology.organism_classification, Tumor Necrosis Factor-alpha/immunology, Immunity, Innate, Mice, Inbred C57BL, 030104 developmental biology, Viral replication, Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein, Insect Science, INNATE IMMUNITY, bacteria, Tnf, Malt1, Innate Immunity, Nf-kappa B, Interferons, Tumor Necrosis Factor, Virología, Receptors, Tumor Necrosis Factor, Type I/immunology

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