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المؤلفون: N. T. Younger, Luke Boulter, David H. Wilson, Konstantinos Gournopanos, Juan Carlos Acosta, Peter Tennant, Duncan Sproul, Rachel V. Guest, Edward J. Jarman, Alison M. Meynert, Martin S. Taylor, Pleasantine Mill, Timothy J. Kendall, Stephen J. Wigmore, Graeme R. Grimes, Scott H. Waddell, Mollie L. Wilson
مصطلحات موضوعية: Genetic heterogeneity, medicine, Cancer research, Wnt signaling pathway, Cancer, Context (language use), Genetic variability, KRAS, Biology, Malignancy, medicine.disease, medicine.disease_cause, PI3K/AKT/mTOR pathway
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::f9ba9cbd144dbbbf6c53e1cb7b87d192
https://doi.org/10.1101/2021.07.05.449722 -
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المؤلفون: Kjetil Søreide, Ewen M Harrison, Stephen J. Wigmore, Rachel V. Guest, O. J. Garden, Timothy J. Kendall
المصدر: Soreide, K, Guest, R V, Harrison, E M, Kendall, T J, Garden, O J & Wigmore, S J 2019, ' Systematic review of management of incidental gallbladder cancer after cholecystectomy ', British Journal of Surgery . https://doi.org/10.1002/bjs.11035
مصطلحات موضوعية: Reoperation, medicine.medical_specialty, medicine.medical_treatment, Perforation (oil well), Disease, 030230 surgery, Risk Assessment, 03 medical and health sciences, Neoplasm Seeding, Postoperative Complications, 0302 clinical medicine, Biomarkers, Tumor, medicine, Humans, Cholecystectomy, Neoplasm Metastasis, Gallbladder cancer, Incidental Findings, business.industry, Gallbladder, General surgery, Cancer, Prognosis, medicine.disease, Conversion to Open Surgery, Systematic review, medicine.anatomical_structure, Chemotherapy, Adjuvant, 030220 oncology & carcinogenesis, Meta-analysis, Gallbladder Neoplasms, Laparoscopy, Surgery, business
وصف الملف: application/vnd.openxmlformats-officedocument.wordprocessingml.document
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المؤلفون: Benjamin J. Dwyer, Luke Boulter, Stuart J. Forbes, Rachel V. Guest
المصدر: NPJ Regenerative Medicine
npj Regenerative Medicine, Vol 2, Iss 1, Pp 1-8 (2017)مصطلحات موضوعية: 0301 basic medicine, Stromal cell, Regeneration (biology), Biomedical Engineering, Wnt signaling pathway, Medicine (miscellaneous), Cancer, Injury and repair, Cell Biology, Review Article, Biology, medicine.disease, Malignancy, Liver regeneration, 03 medical and health sciences, 030104 developmental biology, Immunology, medicine, Medicine, Primary liver cancer, Neuroscience, Developmental Biology
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المؤلفون: Luke Boulter, Tania Roskams, Mina Komuta, Sofia Ferreira Gonzalez, Owen J. Sansom, Rachel V. Guest, Stephen J. Wigmore, Davina Wojtacha, Andrew Robson, Wei-Yu Lu, Stuart J. Forbes, Timothy J. Kendall, Benjamin J. Dwyer, Sarah E. Minnis-Lyons, Tak-Yung Man, Alexander Raven, Jennifer P. Morton
المساهمون: UCL - SSS/IREC-Institut de recherche expérimentale et clinique, UCL - SSS/IREC/GAEN-Pôle d'Hépato-gastro-entérologie, UCL - (SLuc) Service d'anatomie pathologique
المصدر: Proceedings of the National academy of sciences of the United States of America, Vol. 113, no. 43, p. 12250-12255 (2016)
Guest, R V, Boulter, L, Dwyer, B J, Kendall, T J, Man, T, Minnis-Lyons, S E, Lu, W-Y, Robson, A J, Gonzalez, S F, Raven, A, Wojtacha, D, Morton, J P, Komuta, M, Roskams, T, Wigmore, S J, Sansom, O J & Forbes, S J 2016, ' Notch3 drives development and progression of cholangiocarcinoma ', Proceedings of the National Academy of Sciences, vol. 113, no. 43, pp. 12250-12255 . https://doi.org/10.1073/pnas.1600067113مصطلحات موضوعية: 0301 basic medicine, Genetically modified mouse, Notch, Carcinogenesis, Notch signaling pathway, Mice, Transgenic, Biology, medicine.disease_cause, noncanonica, Cholangiocarcinoma, 03 medical and health sciences, Mice, Phosphatidylinositol 3-Kinases, Mediator, medicine, Animals, Humans, Receptor, Receptor, Notch3, Cancer, Multidisciplinary, RBPJ, Neoplasms, Experimental, Biological Sciences, medicine.disease, Prognosis, 3. Good health, Rats, 030104 developmental biology, Immunology, Cancer research, Immunoglobulin Joining Region, Bile duct, Noncanonical, Signal transduction, Tumor Suppressor Protein p53, Signal Transduction
وصف الملف: application/pdf
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c5481771dc25e07cd40aa00ea257daf9
https://hdl.handle.net/2078.1/180106 -
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المؤلفون: Owen J. Sansom, Luke Boulter, Timothy J. Kendall, Sarah E. Minnis-Lyons, Stephen J. Wigmore, Robert S. Walker, Rachel V. Guest, Stuart J. Forbes
المصدر: Guest, R V, Boulter, L, Kendall, T J, Minnis-Lyons, S, Walker, R, Wigmore, S J, Sansom, O J & Forbes, S J 2014, ' Cell Lineage Tracing Reveals a Biliary Origin of Intrahepatic Cholangiocarcinoma ', Cancer Research, vol. 74, no. 4, pp. 1005-1010 . https://doi.org/10.1158/0008-5472.CAN-13-1911
مصطلحات موضوعية: Cancer Research, Pathology, medicine.medical_specialty, Notch, Lineage (genetic), Notch signaling pathway, Context (language use), Mice, Transgenic, Biology, Article, Cholangiocarcinoma, Mice, Bacterial Proteins, Genes, Reporter, origin, medicine, cancer, Animals, Cell Lineage, Progenitor cell, Biliary Tract, Intrahepatic Cholangiocarcinoma, Keratin-19, Lineage markers, Stem Cells, Liver Neoplasms, Genes, p53, 3. Good health, Luminescent Proteins, Bile Ducts, Intrahepatic, Cell Transformation, Neoplastic, Oncology, Bile Duct Neoplasms, Biliary tract, Cell Tracking, Hepatocytes, Stem cell, cholangiocarcinoma, cholangiocyte
وصف الملف: application/pdf
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::26fcd79845caa7c442194b34f03b6fff
https://pubmed.ncbi.nlm.nih.gov/24310400