نتائج البحث - "Damage-associated molecular pattern"

1

Acute Experimental Barrier Injury Triggers Ulcerative Colitis–Specific Innate Hyperresponsiveness and Ulcerative Colitis–Type Microbiome Changes in Humans

المؤلفون: Jakob Benedict Seidelin, Jens Vilstrup Johansen, Marina Ramírez Galera, Lene Riis, Tine Rask Licht, Benjamin E. Mead, Anders Woetmann, Martin Iain Bahl, Jeffrey M. Karp, Jacob Tveiten Bjerrum

المصدر: Seidelin, J B, Bahl, M I, Licht, T R, Mead, B E, Karp, J M, Johansen, J V, Riis, L B, Galera, M R, Woetmann, A & Bjerrum, J T 2021, ' Acute Experimental Barrier Injury Triggers Ulcerative Colitis–Specific Innate Hyperresponsiveness and Ulcerative Colitis–Type Microbiome Changes in Humans ', Cellular and Molecular Gastroenterology and Hepatology, vol. 12, no. 4, pp. 1281-1296 . https://doi.org/10.1016/j.jcmgh.2021.06.002
Cellular and Molecular Gastroenterology and Hepatology, Vol 12, Iss 4, Pp 1281-1296 (2021)
Cellular and Molecular Gastroenterology and Hepatology

مصطلحات موضوعية: Male, 0301 basic medicine, DAMP, damage-associated molecular pattern, ILC3, RC799-869, Flare Initiation, 0302 clinical medicine, AMP, antimicrobial peptide, Intestinal Mucosa, Original Research, IBD, inflammatory bowel disease, Gastroenterology, Sigmoid colon, ILC, innate lymphoid cell, Middle Aged, Diseases of the digestive system. Gastroenterology, Control subjects, Immunohistochemistry, Ulcerative colitis, rRNA, ribosomal RNA, medicine.anatomical_structure, MES, Mayo endoscopic subscore, Disease Progression, Innate Lymphoid Cells Type 3, qPCR, quantitative polymerase chain reaction, Cytokines, Female, 030211 gastroenterology & hepatology, Disease Susceptibility, Injury model, Inflammation Mediators, medicine.symptom, Acute Mucosal Injury, PAMP, pathogen-associated molecular pattern, Adult, Innate Intestinal Response, Inflammation, 03 medical and health sciences, GO, Gene Ontology, CD, Crohn’s disease, medicine, Humans, Ulcerative Colitis, Microbiome, Aged, Host Microbial Interactions, Hepatology, business.industry, Endoscopy, NCR, natural cytotoxicity receptor, medicine.disease, Immunity, Innate, Lymphocyte Subsets, IL, interleukin, Gastrointestinal Microbiome, UC, ulcerative colitis, 030104 developmental biology, Case-Control Studies, Immunology, Dysbiosis, Colitis, Ulcerative, business, Biomarkers

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e15deac621434858603c947222764fb6
https://doi.org/10.1016/j.jcmgh.2021.06.002

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2

Differential Regulation of Damage-Associated Molecular Pattern Release in a Mouse Model of Skeletal Muscle Ischemia/Reperfusion Injury

المؤلفون: Midori Kakuuchi, Hiroaki Furubeppu, Tomotsugu Yasuda, Takashi Ito, Yasuyuki Kakihana, Ikuro Maruyama, Chinatsu Kamikokuryo, Shingo Yamada

المصدر: Frontiers in Immunology, Vol 12 (2021)
Frontiers in Immunology

مصطلحات موضوعية: Male, 0301 basic medicine, medicine.medical_specialty, high mobility group box 1, Immunology, Ischemia, chemical and pharmacologic phenomena, Hindlimb, HMGB1, Histones, Mice, 03 medical and health sciences, Histone H3, Gastrocnemius muscle, 0302 clinical medicine, Internal medicine, medicine, Alarmins, Animals, Humans, Immunology and Allergy, HMGB1 Protein, skeletal muscle, Antibodies, Blocking, Muscle, Skeletal, damage-associated molecular patterns, Original Research, ischemia reperfusion injury, biology, business.industry, Damage-associated molecular pattern, Skeletal muscle, 030208 emergency & critical care medicine, RC581-607, medicine.disease, Mice, Inbred C57BL, Disease Models, Animal, Protein Transport, 030104 developmental biology, Endocrinology, medicine.anatomical_structure, Reperfusion Injury, biology.protein, Immunologic diseases. Allergy, business, Reperfusion injury, extracellular histones

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9b96bef52ca7c54b85b6988b7c3fce41
https://www.frontiersin.org/articles/10.3389/fimmu.2021.628822/full

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Elevated uric acid and adenosine triphosphate concentrations in bronchoalveolar lavage fluid of eosinophilic pneumonia

المؤلفون: Toru Noguchi, Kiyoko Kobayashi, Makoto Nagata, Hidetomo Nakamoto, Tomoyuki Soma, Kazuyuki Nakagome, Kenji Ikebuchi, Yutaka Ueda, Takehito Kobayashi

المصدر: Allergology International, Vol 66, Iss S, Pp S27-S34 (2017)

مصطلحات موضوعية: Male, 0301 basic medicine, Leukocyte Count, chemistry.chemical_compound, Adenosine Triphosphate, 0302 clinical medicine, Eosinophilic pneumonia, Alarmins, Immunology and Allergy, medicine.diagnostic_test, Interleukin, General Medicine, Middle Aged, respiratory system, medicine.anatomical_structure, Acute eosinophilic pneumonia, Cytokines, Female, Inflammation Mediators, Bronchoalveolar Lavage Fluid, Hypersensitivity pneumonitis, lcsh:Immunologic diseases. Allergy, Adult, Adolescent, government.form_of_government, Eosinophil-derived neurotoxin, Biology, Young Adult, 03 medical and health sciences, medicine, Humans, Pulmonary Eosinophilia, Aged, Damage-associated molecular pattern molecules, L-Lactate Dehydrogenase, Eosinophil, medicine.disease, Uric Acid, respiratory tract diseases, Eosinophils, 030104 developmental biology, Bronchoalveolar lavage, chemistry, Immunology, IL-33, government, Uric acid, lcsh:RC581-607, Biomarkers, 030215 immunology

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::fc4efe2581c499011359d2f75ec501c5
https://doi.org/10.1016/j.alit.2017.06.010

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Mitochondrial DNA Is a Pro-Inflammatory Damage-Associated Molecular Pattern Released During Active IBD

المؤلفون: Jack Satsangi, Ray Boyapati, Nicholas T. Ventham, Mary K. Doherty, Philip D. Whitfield, Gwo-Tzer Ho, Mohini Gray, Joseph Loane, Rahul Kalla, Arina Tamborska, Adriano G. Rossi, David A. Dorward

المصدر: Boyapati, R K, Dorward, D A, Tamborska, A, Kalla, R, Ventham, N T, Doherty, M K, Whitfield, P D, Gray, M, Loane, J, Rossi, A G, Satsangi, J & Ho, G-T 2018, ' Mitochondrial DNA Is a Pro-Inflammatory Damage-Associated Molecular Pattern Released During Active IBD ', Inflammatory Bowel Diseases . https://doi.org/10.1093/ibd/izy095, https://doi.org/10.1093/ibd/izy095
Inflamm Bowel Dis