يعرض 1 - 10 نتائج من 12 نتيجة بحث عن '"Signal Transduction -- immunology."', وقت الاستعلام: 1.75s تنقيح النتائج
  1. 1

    المصدر: Frontiers in Immunology, Vol 11 (2020)
    Frontiers in Immunology
    Frontiers in immunology, Vol. 11 (2020) P. 580974

    مصطلحات موضوعية: 0301 basic medicine, follicular T helper cells, Signal Transduction / immunology, Th1 Cells / immunology, Receptor, Interferon alpha-beta, ddc:616.07, Receptor, Interferon alpha-beta / genetics, Ligands, B-Lymphocytes / immunology, Monocytes, NF-kappa B / immunology, chemistry.chemical_compound, Mice, 0302 clinical medicine, germinal centers, Immunology and Allergy, Cationic liposome, Receptor, Original Research, Mice, Knockout, B-Lymphocytes, ddc:618, Membrane Glycoproteins, TLR7/8 agonist, NF-kappa B, T helper cell, Cell biology, Liposome, medicine.anatomical_structure, Interferon Type I, liposome, Phosphatidylcholines, Germinal centers, Adjuvants, Immunologic / administration & dosage, Follicular T helper cells, Heterocyclic Compounds, 3-Ring, Stearic Acids, Signal Transduction, lcsh:Immunologic diseases. Allergy, Liposomes / administration & dosage, Stearic Acids / administration & dosage, Interferon Type I / immunology, Drug Compounding, NF-kappa B / deficiency, Immunology, Membrane Glycoproteins / agonists, Phosphatidylcholines / administration & dosage, Toll-Like Receptor 7 / agonists, 03 medical and health sciences, Adjuvants, Immunologic, Immunity, Adjuvanticity, medicine, Dendritic Cells / immunology, Animals, adjuvants for vaccine, Receptor, Interferon alpha-beta / immunology, Adjuvants for vaccine, Heterocyclic Compounds, 3-Ring / administration & dosage, Toll-Like Receptor 8 / agonists, Germinal center, NF-κB, TLR7, Dendritic Cells, Th1 Cells, Monocytes / immunology, Germinal Center, Immunity, Innate, NF-kappa B / genetics, Mice, Inbred C57BL, Germinal Center / immunology, 030104 developmental biology, Receptor, Interferon alpha-beta / deficiency, chemistry, Toll-Like Receptor 7, Toll-Like Receptor 8, Liposomes, lcsh:RC581-607, 030215 immunology

  2. 2

    المساهمون: Groupe Interdisciplinaire de Génoprotéomique Appliquée (GIGA-Research), Université de Liège, Department of Pathology [Stanford], Stanford Medicine, Stanford University-Stanford University, Sean N. Parker Center for Allergy and Asthma Research [Stanford], Institut de Génétique et de Biologie Moléculaire et Cellulaire (IGBMC), Université de Strasbourg (UNISTRA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Technische Universität Dresden = Dresden University of Technology (TU Dresden), Cell and Developmental Biology [San Diego], School of Biological Sciences [Univ California San Diego] (UC San Diego), University of California [San Diego] (UC San Diego), University of California (UC)-University of California (UC)-University of California [San Diego] (UC San Diego), University of California (UC)-University of California (UC), Environmental Molecular Biology Laboratory (RIKEN), RIKEN - Institute of Physical and Chemical Research [Japon] (RIKEN), Walloon Excellence in Life sciences and BIOtechnology [Liège] (WELBIO), Stanford School of Medicine [Stanford], Department of Microbiology and Immunology [Stanford], Pistre, Karine

    المصدر: Journal of Clinical Investigation
    Journal of Clinical Investigation, 2016, 126 (12), pp.4497-4515. ⟨10.1172/JCI86359⟩

    مصطلحات موضوعية: Keratinocytes, 0301 basic medicine, MESH: Guanine Nucleotide Exchange Factors / genetics, [SDV]Life Sciences [q-bio], MESH: Myeloid Differentiation Factor 88 / immunology, MESH: Dermatitis, Contact / immunology, Dermatitis, Contact, MESH: Mice, Knockout, Mice, 0302 clinical medicine, MESH: Humans Keratinocytes / immunology, Guanine Nucleotide Exchange Factors, MESH: Animals, MESH: NF-kappa B / genetics, Mice, Knockout, MESH: Dermatitis, Contact / pathology, integumentary system, MESH: Signal Transduction / immunology, MESH: Guanine Nucleotide Exchange Factors / immunology, Chemistry, NF-kappa B, MESH: Epidermis / pathology, General Medicine, Atopic dermatitis, Phenotype, Cell biology, [SDV] Life Sciences [q-bio], medicine.anatomical_structure, MESH: NF-kappa B / immunology, 030220 oncology & carcinogenesis, MESH: Dermatitis, Contact / genetics, [SDV.IMM]Life Sciences [q-bio]/Immunology, Female, Guanine nucleotide exchange factor, MESH: Signal Transduction / genetics Signal Transduction / immunology, Signal transduction, medicine.symptom, Keratinocyte, Signal Transduction, Research Article, [SDV.IMM] Life Sciences [q-bio]/Immunology, Inflammation, [SDV.BC]Life Sciences [q-bio]/Cellular Biology, 03 medical and health sciences, MESH: Keratinocytes / pathology, medicine, Animals, Humans, MESH: Receptors, Interleukin-1 Type I / immunology, MESH: Mice, [SDV.BC] Life Sciences [q-bio]/Cellular Biology, Allergic contact dermatitis, Receptors, Interleukin-1 Type I, medicine.disease, 030104 developmental biology, Myeloid Differentiation Factor 88, Rab, Epidermis, MESH: Receptors, Interleukin-1 Type I / genetics, MESH: Female, MESH: Myeloid Differentiation Factor 88 / genetics

  3. 3

    المصدر: Nature immunology, vol. 21, no. 3, pp. 298-308
    Nature Immunology, Vol. 21, No 3 (2020) pp. 298-308
    Nature Immunology

    وصف الملف: application/pdf; Print-Electronic

  4. 4

    المصدر: Infection and Immunity
    Infection and immunity, vol. 86, no. 12, pp. e00710-18

    وصف الملف: application/pdf

  5. 5

    المساهمون: Pathogénie des Staphylocoques – Staphylococcal Pathogenesis (StaPath), Centre International de Recherche en Infectiologie - UMR (CIRI), École normale supérieure - Lyon (ENS Lyon)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-École normale supérieure - Lyon (ENS Lyon)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Microbiologie moléculaire et biochimie structurale / Molecular Microbiology and Structural Biochemistry (MMSB), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Centre National de la Recherche Scientifique (CNRS), Centre National de Reference des Staphylocoques, Université de Lyon, University Medical Center [Utrecht], Wageningen University and Research [Wageningen] (WUR), Hôpitaux Universitaires de Genève (HUG), Centre International de Recherche en Infectiologie (CIRI), École normale supérieure de Lyon (ENS de Lyon)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Université Jean Monnet - Saint-Étienne (UJM)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)-École normale supérieure de Lyon (ENS de Lyon)-Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Université Jean Monnet - Saint-Étienne (UJM)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)

    المصدر: PLoS Pathogens
    PLoS Pathogens, Public Library of Science, 2017, 13 (1), pp.e1006092. ⟨10.1371/journal.ppat.1006092⟩
    PLoS Pathogens 13 (2017) 1
    PLOS Pathogens, Vol. 13, No 1 (2017) P. e1006092
    PLoS Pathogens, Vol 13, Iss 1, p e1006092 (2017)
    PLoS Pathogens, 2017, 13 (1), pp.e1006092. ⟨10.1371/journal.ppat.1006092⟩
    PLoS Pathogens, 13(1)

    مصطلحات موضوعية: Staphylococcus, Pathology and Laboratory Medicine, Biochemistry, Toll-Like Receptors/genetics, Mice, Bacterial Proteins/genetics/immunology, Biology (General), Immune Response, ddc:616, Membrane Glycoproteins, Effector, Toll-Like Receptors, 3. Good health, Bacterial Pathogens, Staphylococcus aureus, Medical Microbiology, [SDV.MP.VIR]Life Sciences [q-bio]/Microbiology and Parasitology/Virology, QH301-705.5, Virulence Factors, Knockout, 030106 microbiology, Immunology, Virulence, Microbiology, digestive system, 03 medical and health sciences, Signs and Symptoms, Bacterial Proteins, Genetics, Humans, Penicillin-Binding Proteins, Molecular Biology Techniques, Microbial Pathogens, Molecular Biology, Staphylococcal Skin Infections/drug therapy/microbiology, Signal Transduction/immunology, Bacteria, Animal, Macrophages, Neutrophils/immunology, Organisms, Biology and Life Sciences, Proteins, Receptors, Interleukin-1/genetics/immunology, Receptors, Interleukin-1, Correction, biochemical phenomena, metabolism, and nutrition, [SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, Laboratorium voor Phytopathologie, Mice, Inbred C57BL, TLR2, 030104 developmental biology, Virulence Factors/genetics/immunology, Laboratory of Phytopathology, Lesions, Parasitology, Immunologic diseases. Allergy, EPS, Fusidic Acid, Cloning, 0301 basic medicine, Penicillin-Binding Proteins/genetics, Neutrophils, Macrophages/immunology, medicine.disease_cause, Inbred C57BL, Immune Receptors, Receptors, Medicine and Health Sciences, Mice, Knockout, Immune System Proteins, Animal Models, Membrane Glycoproteins/genetics/immunology, Infectious Diseases, Experimental Organism Systems, Fusidic Acid/pharmacology, [SDV.IMM]Life Sciences [q-bio]/Immunology, Methicillin-resistant Staphylococcus aureus, Staphylococcal Skin Infections, Signal transduction, Pathogens, Research Article, Signal Transduction, Skin Infections, Mouse Models, Dermatology, Biology, Research and Analysis Methods, Cell Line, Model Organisms, Diagnostic Medicine, Virology, parasitic diseases, medicine, Life Science, Animals, Myeloid Differentiation Factor 88/deficiency/genetics, Inflammation, Innate immune system, SCCmec, Staphylococcus aureus/genetics/immunology/pathogenicity, Cell Biology, RC581-607, Disease Models, Animal, HEK293 Cells, Disease Models, Myeloid Differentiation Factor 88, Ectopic expression, Interleukin-1

    وصف الملف: application/pdf; application/octet-stream

  6. 6

    المساهمون: Key Laboratory of Molecular Virology and Immunology [Shanghai, China], Institut Pasteur de Shanghai, Académie des Sciences de Chine - Chinese Academy of Sciences (IPS-CAS), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), Division of Experimental Animal Immunology, Center for Animal Disease Models [Japan], Tokyo University of Science [Tokyo]-Research Institute for Biomedical Sciences [Japan], Parasitologie moléculaire et Signalisation / Molecular Parasitology and Signaling, Institut Pasteur [Paris]-Institut National de la Santé et de la Recherche Médicale (INSERM), This work was supported by grants from Natural Science Foundation of China (31370892, 31300712, 91429307, 31570895), National Key Basic Research Programs (2014CB541905, 2015CB554302) and National Major Projects for Science and Technology (2014ZX0801011B-001)., Parasitologie moléculaire et Signalisation, Ricard Andraos, Christel, Institut Pasteur [Paris] (IP)-Institut National de la Santé et de la Recherche Médicale (INSERM)

    المصدر: Cytokine
    Cytokine, Elsevier, 2016, 81, pp.101-108. ⟨10.1016/j.cyto.2016.03.007⟩
    Cytokine, 2016, 81, pp.101-108. ⟨10.1016/j.cyto.2016.03.007⟩

    مصطلحات موضوعية: 0301 basic medicine, medicine.medical_treatment, MESH: Cytokines/genetics, MESH: Enzyme-Linked Immunosorbent Assay/methods, Interleukin-1beta, medicine.disease_cause, Biochemistry, Cross-reactivity, MESH: Mice, Knockout, Inflammasome, Immunology and Allergy, Medicine, MESH: Animals, MESH: Tumor Necrosis Factor-alpha/immunology, MESH: Signal Transduction/genetics, Cells, Cultured, Mice, Knockout, biology, MESH: Macrophages/immunology, Cross reactivity, MESH: Macrophages/microbiology, Hematology, MESH: Staphylococcus aureus/classification, MESH: Staphylococcus aureus/genetics, 3. Good health, Cytokine, [SDV.MP]Life Sciences [q-bio]/Microbiology and Parasitology, Staphylococcus aureus, IL-1β, MESH: Host-Pathogen Interactions/immunology, MESH: Cross Reactions/immunology, Host-Pathogen Interactions, Cytokines, Antibody, medicine.symptom, medicine.drug, MESH: Cells, Cultured, Signal Transduction, MESH: Interleukin-1beta/immunology, MESH: Interleukin-1beta/genetics, Gram-positive bacteria, Immunology, Blotting, Western, Inflammation, Enzyme-Linked Immunosorbent Assay, Cross Reactions, 03 medical and health sciences, Immune system, Species Specificity, MESH: Mice, Inbred C57BL, MESH: Blotting, Western, MESH: Species Specificity, Animals, MESH: Macrophages/metabolism, [SDV.MP] Life Sciences [q-bio]/Microbiology and Parasitology, Molecular Biology, business.industry, Tumor Necrosis Factor-alpha, Macrophages, MESH: Cytokines/immunology, MESH: Signal Transduction/immunology, biology.organism_classification, S. aureus, Mice, Inbred C57BL, 030104 developmental biology, TNF-α, biology.protein, business, MESH: Staphylococcus aureus/immunology

    وصف الملف: application/pdf

  7. 7

    المصدر: Nature immunology
    Nature Immunology, vol. 13, no. 6, pp. 569-578

    وصف الملف: application/pdf

  8. 8

    المساهمون: Cellular and Molecular Immunology, Department of Bio-engineering Sciences

    المصدر: PLoS Pathogens, Vol 6, Iss 8, p e1001045 (2010)
    PLoS pathogens 6(8), e1001045 (2010). doi:10.1371/journal.ppat.1001045
    P L o S Pathogens, 6 (8
    Vrije Universiteit Brussel
    PLoS Pathogens

    وصف الملف: 1 full-text file(s): application/pdf

  9. 9

    المصدر: PLoS Pathogens
    PLoS Pathogens, Vol 6, Iss 1, p e1000714 (2010)
    Galès, A, Conduché, A, Bernad, J, Lefevre, L, Olagnier, D, Béraud, M, Martin-Blondel, G, Linas, M-D, Auwerx, J, Coste, A & Pipy, B 2010, ' PPARγ controls Dectin-1 expression required for host antifungal defense against Candida albicans ' P L o S Pathogens, vol. 6, no. 1, pp. e1000714 . https://doi.org/10.1371/journal.ppat.1000714

  10. 10

    المصدر: International immunology, Vol. 20, No 7 (2008) pp. 881-91