نتائج البحث - "Peng-fei Zhang"

1

Long non-coding RNA PSMA3-AS1 promotes malignant phenotypes of esophageal cancer by modulating the miR-101/EZH2 axis as a ceRNA

المؤلفون: Jian-Jun Xu, Xia-Hui Lin, Xiao-qiang Zhang, Shu-Qiang Zhu, Xu-Dong Ye, Xu Pei, Lu-Lu Sheng, Bai-Quan Qiu, Peng-Fei Zhang, Kun Lin, Wei Huang, Dian Xiong, Xiang Long, Xue-Mei Zhang, Yong-Bing Wu, Feng Lu

المصدر: Aging (Albany NY)

مصطلحات موضوعية: Adult, Male, Proteasome Endopeptidase Complex, Aging, Esophageal Neoplasms, proliferation, enhancer of zeste homolog 2, Biology, Metastasis, Cell Movement, Cell Line, Tumor, medicine, metastasis, Humans, Enhancer of Zeste Homolog 2 Protein, Neoplasm Metastasis, 3' Untranslated Regions, neoplasms, Aged, Cell Proliferation, Neoplasm Staging, long non-coding RNA, medicine.diagnostic_test, Competing endogenous RNA, EZH2, Cell Biology, Middle Aged, medicine.disease, Phenotype, digestive system diseases, Long non-coding RNA, Gene Expression Regulation, Neoplastic, MicroRNAs, esophageal squamous cell carcinoma (ESCC), Cell culture, Cancer research, Immunohistochemistry, Female, RNA Interference, RNA, Long Noncoding, Research Paper, Fluorescence in situ hybridization

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0fbb76617a06b8a6f8c6fba5863e1186
https://doi.org/10.18632/aging.102716

2

CTLA-4 Synergizes With PD1/PD-L1 in the Inhibitory Tumor Microenvironment of Intrahepatic Cholangiocarcinoma

المؤلفون: Xiao-Jun Guo, Jia-Cheng Lu, Hai-Ying Zeng, Rong Zhou, Qi-Man Sun, Guo-Huan Yang, Yan-Zi Pei, Xian-Long Meng, Ying-Hao Shen, Peng-Fei Zhang, Jia-Bin Cai, Pei-Xin Huang, Ai-Wu Ke, Ying-Hong Shi, Jian Zhou, Jia Fan, Yi Chen, Liu-Xiao Yang, Guo-Ming Shi, Xiao-Yong Huang

المصدر: Frontiers in Immunology, Vol 12 (2021)
Frontiers in Immunology

مصطلحات موضوعية: Male, Oncology, Lymphocyte, medicine.medical_treatment, Programmed Cell Death 1 Receptor, Kaplan-Meier Estimate, Lithiasis, B7-H1 Antigen, Targeted therapy, Cholangiocarcinoma, intrahepatic cholangiocarcinoma, Tumor Microenvironment, Immunology and Allergy, CTLA-4 Antigen, Intrahepatic Cholangiocarcinoma, Original Research, biology, Liver Diseases, hepatolithiasis, FOXP3, Forkhead Transcription Factors, hemic and immune systems, Middle Aged, cytotoxic T-lymphocyte-associated antigen-4, Neoplasm Proteins, Up-Regulation, medicine.anatomical_structure, Immunohistochemistry, Female, programmed death ligand-1, medicine.medical_specialty, Immunology, chemical and pharmacologic phenomena, Lymphocytes, Tumor-Infiltrating, Internal medicine, PD-L1, medicine, Humans, Aged, Proportional Hazards Models, Tumor microenvironment, business.industry, Proportional hazards model, Gene Expression Profiling, RC581-607, Bile Duct Neoplasms, biology.protein, prognosis, Immunologic diseases. Allergy, business

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5bd7e564602ecefdd1f9b41f591dda7c
https://doi.org/10.3389/fimmu.2021.705378

3

Distinct PD-L1/PD1 Profiles and Clinical Implications in Intrahepatic Cholangiocarcinoma Patients with Different Risk Factors

المؤلفون: Peng-Fei Zhang, Guo-Ming Shi, Qing-Nan Meng, Jian Zhou, Yujiang Geno Shi, Ying-Hao Shen, Xiao-Yong Huang, Jia Fan, Ai-Wu Ke, Ying-Hong Shi, Rui-Zhao Dong, Hui-Chuan Sun, Haiying Zeng, Chao Gao, Jia-Bing Cai, Liu-Xiao Yang, Jia-Cheng Lu, Chuan-Yuan Wei, Xuan Yang, Rui Peng, Qiman Sun

المصدر: Theranostics

مصطلحات موضوعية: 0301 basic medicine, Oncology, Adult, Male, medicine.medical_specialty, Hepatitis B virus, immune checkpoint blockage, medicine.medical_treatment, programmed cell death protein ligand 1, Programmed Cell Death 1 Receptor, Medicine (miscellaneous), medicine.disease_cause, Cholangiocarcinoma, 03 medical and health sciences, 0302 clinical medicine, intrahepatic cholangiocarcinoma, Risk Factors, PD-L1, Internal medicine, medicine, Humans, Clinical significance, Pharmacology, Toxicology and Pharmaceutics (miscellaneous), Lymph node, Intrahepatic Cholangiocarcinoma, Aged, Retrospective Studies, biology, business.industry, Liver Neoplasms, hepatolithiasis, Immunotherapy, programmed cell death protein 1, Middle Aged, medicine.disease, 030104 developmental biology, medicine.anatomical_structure, 030220 oncology & carcinogenesis, biology.protein, Biomarker (medicine), Female, Hepatolithiasis, business, Research Paper

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::35424ebc1e31334a015302c10eaf3ec8
http://europepmc.org/articles/PMC6643449

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4

Overexpression of RNF38 facilitates TGF-β signaling by Ubiquitinating and degrading AHNAK in hepatocellular carcinoma

المؤلفون: Jia-Bin Cai, Chuan-Yuan Wei, Qiang Gao, Chao Gao, Xuan Yang, Xiao-Yong Huang, Rui Peng, Hai-Xiang Sun, Jia Fan, Dou-Sheng Bai, Ai-Wu Ke, Jia-Cheng Lu, Peng-Fei Zhang, Guo-Ming Shi

المصدر: Journal of Experimental & Clinical Cancer Research, Vol 38, Iss 1, Pp 1-14 (2019)
Journal of Experimental & Clinical Cancer Research : CR

مصطلحات موضوعية: 0301 basic medicine, Male, Cancer Research, Carcinogenesis, Hepatocellular carcinoma, Cell, Receptor, Transforming Growth Factor-beta Type I, TGF-β signaling, Metastasis, Mice, 0302 clinical medicine, Transforming Growth Factor beta, Stable isotope labeling by amino acids in cell culture, Tissue microarray, Liver Neoplasms, Cell migration, Middle Aged, AHNAK, Prognosis, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Neoplasm Proteins, Gene Expression Regulation, Neoplastic, medicine.anatomical_structure, Oncology, 030220 oncology & carcinogenesis, Isotope Labeling, Female, Signal Transduction, Adult, Carcinoma, Hepatocellular, Epithelial-Mesenchymal Transition, Biology, lcsh:RC254-282, Disease-Free Survival, RNF38, 03 medical and health sciences, Downregulation and upregulation, medicine, Animals, Humans, Research, Membrane Proteins, Transforming growth factor beta, medicine.disease, Xenograft Model Antitumor Assays, digestive system diseases, 030104 developmental biology, biology.protein, Cancer research, Carrier Proteins, Transforming growth factor

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8f9b2e7d57ec24da690c382abdf0e86c
http://link.springer.com/article/10.1186/s13046-019-1113-3

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5

Bioinformatics-based analysis reveals elevated MFSD12 as a key promoter of cell proliferation and a potential therapeutic target in melanoma

المؤلفون: Rui Peng, Lu Wang, Jianying Gu, Chuan-Yuan Wei, Xuan Yang, Peng-Fei Zhang, Meng-Xuan Zhu, Nanhang Lu

المصدر: Oncogene

مصطلحات موضوعية: Adult, Male, 0301 basic medicine, MAPK/ERK pathway, Cancer Research, Skin Neoplasms, AKT1, Biology, Article, 03 medical and health sciences, 0302 clinical medicine, Downregulation and upregulation, Biomarkers, Tumor, Genetics, medicine, Humans, Molecular Targeted Therapy, Melanoma, neoplasms, Molecular Biology, Cells, Cultured, PI3K/AKT/mTOR pathway, Aged, Cell Proliferation, Aged, 80 and over, Regulation of gene expression, Cell growth, Gene Expression Profiling, Computational Biology, Membrane Proteins, Middle Aged, Prognosis, medicine.disease, Survival Analysis, Up-Regulation, Gene Expression Regulation, Neoplastic, Gene expression profiling, Cell Transformation, Neoplastic, 030104 developmental biology, 030220 oncology & carcinogenesis, Disease Progression, Cancer research, Female

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f22666448cf5747f5f2157a463606574
https://doi.org/10.1038/s41388-018-0531-6

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6

LncRNA SNHG3 induces EMT and sorafenib resistance by modulating the miR‐128/CD151 pathway in hepatocellular carcinoma

المؤلفون: Yin Wu, Ai-Wu Ke, Fei Wang, Jing Wu, Xue-Mei Zhang, Xiao-Yong Huang, Dong Liu, Peng-Fei Zhang, Wei Huang

المصدر: Journal of Cellular Physiology. 234:2788-2794

مصطلحات موضوعية: Male, 0301 basic medicine, Sorafenib, Carcinoma, Hepatocellular, Epithelial-Mesenchymal Transition, Carcinogenesis, Physiology, Clinical Biochemistry, Tetraspanin 24, medicine.disease_cause, Disease-Free Survival, 03 medical and health sciences, 0302 clinical medicine, Cell Movement, medicine, Humans, Neoplasm Invasiveness, Small nucleolar RNA, neoplasms, CD151, Cell Proliferation, Transition (genetics), business.industry, Liver Neoplasms, Hep G2 Cells, Cell Biology, Middle Aged, medicine.disease, digestive system diseases, Gene Expression Regulation, Neoplastic, MicroRNAs, 030104 developmental biology, Drug Resistance, Neoplasm, Tumor progression, 030220 oncology & carcinogenesis, Hepatocellular carcinoma, Cancer research, Biomarker (medicine), Female, RNA, Long Noncoding, business, medicine.drug

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::64c2755b54dc72c6d6ac691774549124
https://doi.org/10.1002/jcp.27095

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7

Circular RNA circMET drives immunosuppression and anti-PD1 therapy resistance in hepatocellular carcinoma via the miR-30-5p/snail/DPP4 axis

المؤلفون: Peng-Fei Zhang, Rui Peng, Chuan-Yuan Wei, Jian Zhou, Jia Fan, Ai-Wu Ke, Xiao-Yong Huang, Chao Gao, Jia-Bing Cai, Guo-Ming Shi, Jia-Cheng Lu, Xuan Yang

المصدر: Molecular Cancer, Vol 19, Iss 1, Pp 1-18 (2020)
Molecular Cancer

مصطلحات موضوعية: Male, 0301 basic medicine, Cancer Research, Programmed Cell Death 1 Receptor, Cell, Apoptosis, Immune tolerance, Mice, 0302 clinical medicine, Cell Movement, Tumor Cells, Cultured, circRNA, Immune Checkpoint Inhibitors, circMET, Liver Neoplasms, Middle Aged, Proto-Oncogene Proteins c-met, Prognosis, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Gene Expression Regulation, Neoplastic, Survival Rate, medicine.anatomical_structure, Oncology, 030220 oncology & carcinogenesis, Hepatocellular carcinoma, Chromosomal region, Molecular Medicine, Female, Carcinoma, Hepatocellular, Dipeptidyl Peptidase 4, Hepatocellular carcinogenesis, DPP4, Biology, lcsh:RC254-282, 03 medical and health sciences, Biomarkers, Tumor, medicine, Animals, Humans, CXCL10, Neoplasm Invasiveness, Epithelial–mesenchymal transition, neoplasms, Cell Proliferation, Tumor microenvironment, Research, RNA, Circular, miR-30-5p, medicine.disease, Xenograft Model Antitumor Assays, digestive system diseases, Mice, Inbred C57BL, MicroRNAs, 030104 developmental biology, Drug Resistance, Neoplasm, Cancer research, Snail Family Transcription Factors, CD8

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f4b15b4f7b4647f27e918e086d94a707
https://doi.org/10.1186/s12943-020-01213-6

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8

circMET promotes NSCLC cell proliferation, metastasis, and immune evasion by regulating the miR-145-5p/CXCL3 axis

المؤلفون: Shu-Qiang Zhu, Xu Pei, Feng Lu, Peng-Fei Zhang, Jian-Jun Xu, Kun Lin, Shi-Wei Chen, Xiang Long, Yong-Bing Wu, Bai-Quan Qiu

المصدر: Aging (Albany NY)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::102a8f81b7151eeb13a4f7ae0554e2db
https://pubmed.ncbi.nlm.nih.gov/32614785

9

Correction to: Elevated TRIP13 drives the AKT/mTOR pathway to induce the progression of hepatocellular carcinoma via interacting with ACTN4

المؤلفون: Dong-Mei Gao, Peng-Fei Zhang, Meng-Xuan Zhu, Binbin Liu, Shuang-Shuang Dong, Yan Zhao, Jie Chen, Chuan-Yuan Wei

المصدر: Journal of Experimental & Clinical Cancer Research : CR
Journal of Experimental & Clinical Cancer Research, Vol 38, Iss 1, Pp 1-2 (2019)

مصطلحات موضوعية: Male, Cancer Research, Carcinoma, Hepatocellular, Mice, Nude, Cell Cycle Proteins, lcsh:RC254-282, Mice, Cell Movement, Cell Line, Tumor, Medicine, Animals, Humans, Actinin, Protein kinase B, PI3K/AKT/mTOR pathway, Mice, Inbred BALB C, TRIP13, business.industry, TOR Serine-Threonine Kinases, Liver Neoplasms, Correction, Middle Aged, medicine.disease, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, Xenograft Model Antitumor Assays, Survival Rate, MicroRNAs, Oncology, Apoptosis, Hepatocellular carcinoma, Case-Control Studies, Cancer research, Disease Progression, ATPases Associated with Diverse Cellular Activities, Female, business, Proto-Oncogene Proteins c-akt, Signal Transduction

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6b707187852ab4c8ab185b8a2bffd4b9
http://europepmc.org/articles/PMC6822571

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10

Extracellular HMGB1 promotes CD44 expression in hepatocellular carcinoma via regulating miR-21

المؤلفون: Xiaolei Shi, Jun Li, Peng-Fei Zhang, Haozhen Ren, Jinglin Wang

المصدر: Aging (Albany NY)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d5b6e5bb908730fa36c16bd931710dd2
https://pubmed.ncbi.nlm.nih.gov/33661757

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