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المؤلفون: Lydia W.T. Cheung, Angel S N Ng, Abdullah Aldehaiman, Amy Y T Lau, Patrick Kwok Shing Ng, Stefan T. Arold, Yuan Zhou, Xinran Li
المصدر: Proceedings of the National Academy of Sciences of the United States of America
مصطلحات موضوعية: Receptor, ErbB-3, Class I Phosphatidylinositol 3-Kinases, Allosteric regulation, P110α, SH2 domain, medicine.disease_cause, Receptor tyrosine kinase, src Homology Domains, Transactivation, Phosphatidylinositol 3-Kinases, Protein Domains, Catalytic Domain, Cell Line, Tumor, Neoplasms, medicine, Humans, skin and connective tissue diseases, Protein kinase B, PI3K/AKT/mTOR pathway, Mutation, Multidisciplinary, biology, receptor tyrosine kinases, Chemistry, Receptor Protein-Tyrosine Kinases, p85α, Cell Biology, Biological Sciences, HCT116 Cells, Class Ia Phosphatidylinositol 3-Kinase, biology.protein, Cancer research, Proto-Oncogene Proteins c-akt, Signal Transduction
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::508557af3f0465bb61afc746140561dc
https://pubmed.ncbi.nlm.nih.gov/34507989