نتائج البحث - "Angels Díaz-Ramos"

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دورية أكاديمية

Decreased expression of mitochondrial aminoacyl-tRNA synthetases causes downregulation of OXPHOS subunits in type 2 diabetic muscle

المؤلفون: Iliana López-Soldado, Adrian Gabriel Torres, Raúl Ventura, Inma Martínez-Ruiz, Angels Díaz-Ramos, Evarist Planet, Diane Cooper, Agnieszka Pazderska, Krzysztof Wanic, Declan O'Hanlon, Donal J. O'Gorman, Teresa Carbonell, Lluís Ribas de Pouplana, John J. Nolan, Antonio Zorzano, María Isabel Hernández-Alvarez

المصدر: Redox Biology, Vol 61, Iss , Pp 102630- (2023)

مصطلحات موضوعية: Type 2 diabetes, Mitochondrial aminoacyl tRNA synthetases, Nitrosative stress, OXPHOS, Skeletal muscle, Nitric oxide, Medicine (General), R5-920, Biology (General), QH301-705.5

وصف الملف: electronic resource

Relation: http://www.sciencedirect.com/science/article/pii/S2213231723000319; https://doaj.org/toc/2213-2317

URL الوصول: https://doaj.org/article/c53b67421abe4c1cbd8be5c09028244b

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Decreased expression of mitochondrial aminoacyl-tRNA synthetases causes downregulation of mitochondrial OXPHOS subunits in type 2 diabetic skeletal muscle

المؤلفون: Iliana López-Soldado, Adrian Gabriel Torres, Raúl Ventura, Inma Martínez-Ruiz, Angels Díaz-Ramos, Evarist Planet, Diane Cooper, Agnieszka Pazderska, Krzysztof Wanic, Declan O’Hanlon, Donal J. O’Gorman, Teresa Carbonell, Lluís Ribas de Pouplana, John J. Nolan, María Isabel Hernández-Alvarez

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::c3182bef2361ceb51443116b484375b2
https://doi.org/10.1101/2022.12.30.22283970

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TP53INP2 regulates adiposity by activating β-catenin through autophagy-dependent sequestration of GSK3β

المؤلفون: Víctor A. Francis, M. Romero, Ann Hammarstedt, Joan Vendrell, José Manuel Fernández-Real, Ignacio Castrillon-Rodríguez, Xavier Duran, Ulf Smith, Manuela Sánchez-Feutrie, Antonio Zorzano, José María Moreno-Navarrete, Angels Díaz-Ramos, Manuel Palacín, Birgit Gustafson, Alba Sabaté-Pérez

المصدر: Nature Cell Biology. 20:443-454

مصطلحات موضوعية: Adult, Male, Transcriptional Activation, 0301 basic medicine, Cell signaling, Time Factors, Endosome, 030209 endocrinology & metabolism, Endosomes, ESCRT, Mice, 03 medical and health sciences, 0302 clinical medicine, 3T3-L1 Cells, Adipocytes, Autophagy, Animals, Humans, Wnt Signaling Pathway, beta Catenin, Adiposity, Mice, Knockout, Sweden, Adipogenesis, Glycogen Synthase Kinase 3 beta, Hyperplasia, Endosomal Sorting Complexes Required for Transport, Activator (genetics), Chemistry, Wnt signaling pathway, Nuclear Proteins, Cell Biology, Middle Aged, Cell biology, Mice, Inbred C57BL, Protein Transport, 030104 developmental biology, Adipose Tissue, Spain, Catenin, Female, TCF Transcription Factors

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8818c76ff55fc3d6a884f0991eeda5d7
https://doi.org/10.1038/s41556-018-0072-9

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4

Adipose tissue mitochondrial dysfunction in human obesity is linked to a specific DNA methylation signature in adipose-derived stem cells

المؤلفون: Jeffrey M. Gimble, Pablo M. Garcia-Roves, Catalina Núñez-Roa, Xevi Duran, Victoria Ceperuelo-Mallafré, Miquel A. Peinado, Pau Gama, Elsa Maymó-Masip, Antonio Zorzano, Monica Millan-Scheiding, Angels Díaz-Ramos, Miriam Ejarque, Carolina Serena, Sonia Fernández-Veledo, Joan Vendrell, Yaiza Núñez-Álvarez

المصدر: International Journal of Obesity
r-IGTP. Repositorio Institucional de Producción Científica del Instituto de Investigación Germans Trias i Pujol
instname
Dipòsit Digital de la UB
Universidad de Barcelona
International Journal of Obesity (2005)

مصطلحات موضوعية: Adult, medicine.medical_specialty, Endocrinology, Diabetes and Metabolism, Population, Medicine (miscellaneous), Adipose tissue, 030209 endocrinology & metabolism, Mitochondrion, Biology, Article, 03 medical and health sciences, chemistry.chemical_compound, 0302 clinical medicine, Thinness, Internal medicine, Adipocyte, Adipocytes, medicine, Humans, Obesity, 030212 general & internal medicine, education, Human obesity, Inflammation, education.field_of_study, Adipogenesis, Cèl·lules animals, Nutrition and Dietetics, Stem Cells, Adipose tissues, DNA Methylation, Translational research, Lipids, Cell biology, Mitochondria, Oxidative Stress, Teixit adipós, Endocrinology, Adipose Tissue, Animal cells, Lipotoxicity, chemistry, Lípids, DNA methylation, Female, Stem cell

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::84a7bb62342b24d4f3ce8b10b718a774
https://fundanet.igtp.cat/Publicaciones/ProdCientif/PublicacionFrw.aspx?id=2926

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5

Mitochondrial DNA disturbances and deregulated expression of oxidative phosphorylation and mitochondrial fusion proteins in sporadic inclusion body myositis

المؤلفون: Francesc Cardellach, Adriana Hernando, Marc Catalán-García, Angels Díaz-Ramos, Antonio Zorzano, Ester Tobías, Maria Bañó, Pedro Moreno, Sonia Emperador, Julio Montoya, Constanza Morén, José C. Milisenda, Ingrid González-Casacuberta, Mariona Guitart-Mampel, Josep M. Grau, Diana-Luz Juárez, Jennifer Enrich-Bengoa, Glòria Garrabou

المصدر: Clinical Science. 130:1741-1751

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::828e8491f24138ef96a9870cd94e7d49
https://doi.org/10.1042/cs20160080

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Mitochondrial DNA and TLR9 drive muscle inflammation upon Opa1 deficiency

المصدر: The EMBO journal. 37(10)

مصطلحات موضوعية: 0301 basic medicine, Male, Programmed cell death, endocrine system, Inflammation, Apoptosis, Mitochondrion, Biology, Systemic inflammation, DNA, Mitochondrial, General Biochemistry, Genetics and Molecular Biology, GTP Phosphohydrolases, 03 medical and health sciences, chemistry.chemical_compound, Necrosis, 0302 clinical medicine, Muscular Diseases, medicine, Myocyte, Animals, Regeneration, Inner mitochondrial membrane, Muscle, Skeletal, Molecular Biology, Sodium salicylate, Cells, Cultured, Mice, Knockout, General Immunology and Microbiology, General Neuroscience, Articles, eye diseases, Cell biology, 030104 developmental biology, chemistry, Toll-Like Receptor 9, Cytokines, Female, medicine.symptom, 030217 neurology & neurosurgery

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dcef05cbc7374927e607b3acd2455e31
https://pubmed.ncbi.nlm.nih.gov/29632021

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Early-onset and classical forms of type 2 diabetes show impaired expression of genes involved in muscle branched-chain amino acids metabolism

المؤلفون: Agnieszka Pazderska, Donal J. O’Gorman, Manel Esteller, María Isabel Hernández-Alvarez, María Berdasco, Antonio Gomez, Manuel Palacín, Krzystof Wanic, Jeff Cobb, Laura R. de la Ballina, Angels Díaz-Ramos, Diane Cooper, Evarist Planet, John J. Nolan, Declan O'Hanlon, Antonio Zorzano

المساهمون: Universitat de Barcelona

المصدر: Dipòsit Digital de la UB
Universidad de Barcelona
Scientific Reports, Vol 7, Iss 1, Pp 1-12 (2017)
Recercat. Dipósit de la Recerca de Catalunya
instname
Scientific Reports
Hernández-Alvarez, M I, Díaz-Ramos, A, Berdasco, M, Cobb, J, Planet, E, Cooper, D, Pazderska, A, Wanic, K, O'Hanlon, D, Gomez, A, de la Ballina, L R, Esteller, M, Palacin, M, O'Gorman, D J, Nolan, J J & Zorzano, A 2017, ' Early-onset and classical forms of type 2 diabetes show impaired expression of genes involved in muscle branched-chain amino acids metabolism ', Scientific Reports, vol. 7, 13850 . https://doi.org/10.1038/s41598-017-14120-6

مصطلحات موضوعية: 0301 basic medicine, Male, Mice, Obese, Muscle Proteins, lcsh:Medicine, Type 2 diabetes, Transcriptome, Mice, 0302 clinical medicine, Age of Onset, lcsh:Science, chemistry.chemical_classification, Multidisciplinary, Diabetis, Diabetes, Middle Aged, Pathophysiology, Amino acid, medicine.anatomical_structure, Female, Adult, medicine.medical_specialty, 030209 endocrinology & metabolism, Biology, Article, Diabetes Mellitus, Experimental, Amino acid sequence, 03 medical and health sciences, Young Adult, Insulin resistance, Internal medicine, Diabetes mellitus, Seqüència d'aminoàcids, medicine, Animals, Humans, Metabolomics, Muscle, Skeletal, Gene Expression Profiling, lcsh:R, Skeletal muscle, Metabolism, medicine.disease, Expressió gènica, 030104 developmental biology, Endocrinology, chemistry, Diabetes Mellitus, Type 2, Case-Control Studies, lcsh:Q, Gene expression, Insulin Resistance, Amino Acids, Branched-Chain, Biomarkers