نتائج البحث - "Cisplatin pharmacology"

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دورية أكاديمية

The transcription factor c-Jun inhibits RBM39 to reprogram pre-mRNA splicing during genotoxic stress.
Le facteur de transcription c-Jun inhibe RBM39 pour reprogrammer l'épissage des ARNs pré-messager lors d'un stress génotoxique

المؤلفون: Lemaitre, Florence, Chakrama, Fatima, O'Grady, Tina, Peulen, Olivier, Rademaker, Gilles, Deward, Adeline, Chabot, Benoit, Piette, Jacques, Colige, Alain, Lambert, Charles, Dequiedt, Franck, Habraken, Yvette

المصدر: Nucleic Acids Research, 50 (22), 12768 - 12789 (2022-12-09)

Relation: https://academic.oup.com/nar/article-pdf/50/22/12768/48488767/gkac1130.pdf; urn:issn:0305-1048; urn:issn:1362-4962

URL الوصول: https://orbi.uliege.be/handle/2268/298397

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Hsp90 inhibition sensitizes DLBCL cells to cisplatin

المؤلفون: Linnéa Schmidt, Issa Ismail Issa, Hulda Haraldsdóttir, Jonas Laugård Hald, Alexander Schmitz, Hanne Due, Karen Dybkær

المصدر: Schmidt, L, Issa, I I, Haraldsdóttir, H, Hald, J L, Schmitz, A, Due, H & Dybkær, K 2022, ' Hsp90 inhibition sensitizes DLBCL cells to cisplatin ', Cancer Chemotherapy and Pharmacology, vol. 89, no. 4, pp. 431-440 . https://doi.org/10.1007/s00280-022-04407-5
Schmidt, L, Issa, I I, Haraldsdóttir, H, Hald, J L, Due, H & Dybkær, K 2022, ' Hsp90 inhibition sensitizes DLBCL cells to cisplatin ', Cancer Chemotherapy and Pharmacology, vol. 89, no. 4, pp. 431-440 . https://doi.org/10.1007/s00280-022-04407-5

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::95c3d1875eb090fc0377fa24b24ad23b
https://vbn.aau.dk/ws/files/468426671/Schmidt_et_al._2022_._Hsp90_inhibition_sensitizes_DLBCL_cells_to_cisplatin.pdf

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كتاب

Cisplatin: chemistry and biochemistry of a leading anticancer drug

المساهمون: Lippert, Bernhard, 1946-

مصطلحات موضوعية: Cisplatin., Cancer -- Chemotherapy., Cisplatin -- chemistry., Cisplatin -- pharmacology.

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Molecular correlates of cisplatin-based chemotherapy response in muscle invasive bladder cancer by integrated multi-omics analysis

المؤلفون: Jørgen Bjerggaard Jensen, Frederik Prip, Emil Christensen, Michael Knudsen, Philippe Lamy, Trine Line Hauge Okholm, Ann Taber, Torben Steiniche, Jakob Skou Pedersen, Sia Viborg Lindskrog, Karin Birkenkamp-Demtröder, Iver Nordentoft, Lars Dyrskjøt, Mads Agerbæk

المصدر: Nature Communications, Vol 11, Iss 1, Pp 1-15 (2020)
Taber, A, Christensen, E, Lamy, P, Nordentoft, I, Prip, F, Lindskrog, S V, Birkenkamp-Demtröder, K, Okholm, T L H, Knudsen, M, Pedersen, J S, Steiniche, T, Agerbæk, M, Jensen, J B & Dyrskjøt, L 2020, ' Molecular correlates of cisplatin-based chemotherapy response in muscle invasive bladder cancer by integrated multi-omics analysis ', Nature Communications, vol. 11, no. 1, 4858 . https://doi.org/10.1038/s41467-020-18640-0
Nature Communications

مصطلحات موضوعية: 0301 basic medicine, Genome instability, Oncology, Cisplatin/pharmacology, Molecular biology, CYSTECTOMY, medicine.medical_treatment, Programmed Cell Death 1 Receptor, General Physics and Astronomy, Proteomics, Transcriptome, NEOADJUVANT CHEMOTHERAPY, 0302 clinical medicine, lcsh:Science, Neoadjuvant therapy, Cancer, Epigenomics, Multidisciplinary, Molecular medicine, Urinary Bladder Neoplasms/drug therapy, GEMCITABINE PLUS CISPLATIN, ASSOCIATION, CLONAL EVOLUTION, BRCA2 Protein/genetics, Neoadjuvant Therapy, Gene Expression Regulation, Neoplastic, Chemotherapy, Adjuvant, 030220 oncology & carcinogenesis, DNA methylation, Allelic Imbalance, SURVIVAL, SENSITIVITY, medicine.drug, medicine.medical_specialty, CARCINOMA, Urology, Science, SOMATIC ERCC2 MUTATIONS, Genomics, Context (language use), Article, General Biochemistry, Genetics and Molecular Biology, Genomic Instability, 03 medical and health sciences, Medical research, Drug Therapy, Programmed Cell Death 1 Receptor/genetics, Internal medicine, medicine, Biomarkers, Tumor, Humans, SIGNATURES, Cisplatin, BRCA2 Protein, Bladder cancer, business.industry, General Chemistry, DNA Methylation, medicine.disease, 030104 developmental biology, Urinary Bladder Neoplasms, Gene Expression Regulation, Neoplastic/drug effects, Mutation, lcsh:Q, business, Biomarkers