يعرض 1 - 3 نتائج من 3 نتيجة بحث عن '"Damien Dos-Santos-Luis"', وقت الاستعلام: 0.77s تنقيح النتائج
  1. 1
    Decreased Expression of Vascular Endothelial Growth Factor Receptor 1 Contributes to the Pathogenesis of Hereditary Hemorrhagic Telangiectasia Type 2

    المؤلفون: Franck Lebrin, Simon Tual-Chalot, Helen M. Arthur, Steven Kroon, Frans Disch, Noël Lamandé, Christine L. Mummery, Damien Dos-Santos-Luis, Jérémy H. Thalgott, Johannes J. Mager, Ton J. Rabelink, Repke J. Snijder, Georgios Galaris, Karine Raymond, Anna E Hosman, Yihai Cao, Samly Srun, Sabrina Martin, Hetty C. de Boer, Diane Bracquart

    المساهمون: Institut de Recherche Interdisciplinaire de Grenoble (IRIG), Direction de Recherche Fondamentale (CEA) (DRF (CEA)), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA)

    المصدر: Circulation
    Circulation, American Heart Association, 2018, 138 (23), pp.2698-2712. ⟨10.1161/CIRCULATIONAHA.117.033062⟩
    Circulation, 138(23), 2698-2712
    Circulation, 2018, 138 (23), pp.2698-2712. ⟨10.1161/CIRCULATIONAHA.117.033062⟩

    مصطلحات موضوعية: Adult, Male, 0301 basic medicine, Heterozygote, Activin Receptors, Type II, [SDV]Life Sciences [q-bio], arteriovenous malformation, Neovascularization, Physiologic, Antibodies, Mycoplasma pulmonis, Arteriovenous Malformations, Pathogenesis, Mice, angiogenesis, 03 medical and health sciences, 0302 clinical medicine, Physiology (medical), Animals, Humans, hereditary hemorrhagic telangiectasia, Medicine, vascular endothelial growth factors, Telangiectasia, Skin, Vascular Endothelial Growth Factor Receptor-1, business.industry, Genetic disorder, Retinal Vessels, Mouse Embryonic Stem Cells, ACVRL1, Arteriovenous malformation, Middle Aged, medicine.disease, Mice, Inbred C57BL, Platelet Endothelial Cell Adhesion Molecule-1, Disease Models, Animal, 030104 developmental biology, 030220 oncology & carcinogenesis, cardiovascular system, Cancer research, Female, Telangiectasia, Hereditary Hemorrhagic, pathological, medicine.symptom, Cardiology and Cardiovascular Medicine, business, Haploinsufficiency, Activin Receptors, Type I, Signal Transduction

    URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c863f2798fab803c6c38e525cf96af34
    https://doi.org/10.1161/circulationaha.117.033062

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  2. 2
    Interaction Between ALK1 Signaling and Connexin40 in the Development of Arteriovenous Malformations

    المؤلفون: Konstantinos Gkatzis, Jérémy Thalgott, Damien Dos-Santos-Luis, Sabrina Martin, Noël Lamandé, Marie France Carette, Frans Disch, Repke J. Snijder, Cornelius J. Westermann, Johannes J. Mager, S. Paul Oh, Lucile Miquerol, Helen M. Arthur, Christine L. Mummery, Franck Lebrin

    المساهمون: Institut de Biologie du Développement de Marseille (IBDM), Aix Marseille Université (AMU)-Collège de France (CdF (institution))-Centre National de la Recherche Scientifique (CNRS)

    المصدر: Arteriosclerosis, Thrombosis, and Vascular Biology
    Arteriosclerosis, Thrombosis, and Vascular Biology, American Heart Association, 2016, 36 (4), pp.707-17. ⟨10.1161/ATVBAHA.115.306719.Epub2016Jan28⟩
    Arteriosclerosis, Thrombosis, and Vascular Biology, 36(4), 707-717
    None
    Arteriosclerosis, Thrombosis, and Vascular Biology, 2016, 36 (4), pp.707-17. ⟨10.1161/ATVBAHA.115.306719.Epub2016Jan28⟩

    مصطلحات موضوعية: 0301 basic medicine, Pathology, medicine.medical_specialty, Activin Receptors, Type II, Mutant, Mice, Transgenic, Vasodilation, Haploinsufficiency, [SDV.BC]Life Sciences [q-bio]/Cellular Biology, Vascular Remodeling, Biology, Transfection, Connexins, alpha-smooth muscle actin, 03 medical and health sciences, connexin 40, medicine, Animals, Humans, hereditary hemorrhagic telangiectasia, Genetic Predisposition to Disease, arteriovenous malformations, Cells, Cultured, mouse, chemistry.chemical_classification, Reactive oxygen species, Neovascularization, Pathologic, Kinase, Endothelial Cells, Retinal Vessels, ACVRL1, Molecular biology, Mice, Mutant Strains, Disease Models, Animal, Phenotype, 030104 developmental biology, chemistry, RNA Interference, Telangiectasia, Hereditary Hemorrhagic, erythrocyte, Signal transduction, Reactive Oxygen Species, Cardiology and Cardiovascular Medicine, Activin Receptors, Type I, Signal Transduction

    URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::de8be4b756bdf01a95877028d9d73e94
    https://hal.archives-ouvertes.fr/hal-01444444

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  3. 3
    Pericytes as targets in hereditary hemorrhagic telangiectasia

    المؤلفون: Damien Dos-Santos-Luis, Franck Lebrin, Jérémy Thalgott

    المصدر: Frontiers in Genetics, Vol 6 (2015)
    Frontiers in Genetics

    مصطلحات موضوعية: transforming growth factor-β signaling, lcsh:QH426-470, Angiogenesis, Cell, Review Article, Bioinformatics, Pediatrics, Mural cell, Paracrine signalling, angiogenesis, pericyte, thalidomide, Genetics, Medicine, Telangiectasia, rare vascular disease, Genetics (clinical), business.industry, Thalidomide, lcsh:Genetics, medicine.anatomical_structure, Cancer research, cardiovascular system, Molecular Medicine, Pericyte, medicine.symptom, business, medicine.drug, Transforming growth factor

    URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eac98a4f4f31d0c452e9b71793b763d0

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