المؤلفون: Daniel R, Carrasco, Tim, Fenton, Kumar, Sukhdeo, Marina, Protopopova, Miriam, Enos, Mingjian J, You, Dolores, Di Vizio, Dolores, Divicio, Cristina, Nogueira, Jayne, Stommel, Geraldine S, Pinkus, Christopher, Fletcher, Jason L, Hornick, Webster K, Cavenee, Frank B, Furnari, Ronald A, Depinho

المصدر: Cancer Cell. 9(5):379-390

مصطلحات موضوعية: Histiocytic Disorders, Malignant, Cancer Research, CELLCYCLE, 02 engineering and technology, Histiocytic sarcoma, medicine.disease_cause, Methylation, Immunophenotyping, law.invention, Pathogenesis, Mice, 03 medical and health sciences, 0302 clinical medicine, law, Ink4a arf, Tumor Suppressor Protein p14ARF, medicine, Animals, Homeostasis, Humans, PTEN, Myeloid Cells, Lymphocytes, Epigenetics, Extracellular Signal-Regulated MAP Kinases, Cyclin-Dependent Kinase Inhibitor p16, 030304 developmental biology, 0303 health sciences, Mutation, biology, PTEN Phosphohydrolase, Sarcoma, Cell Biology, Cell cycle, 021001 nanoscience & nanotechnology, medicine.disease, Phenotype, Enzyme Activation, Oncology, 030220 oncology & carcinogenesis, Immunology, biology.protein, Cancer research, Suppressor, 0210 nano-technology, Proto-Oncogene Proteins c-akt

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