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المؤلفون: Jun O. Liu, Curtis R. Chong, Hyunki Kim, Jeon Han Park, S. Diane Hayward, Richard F. Ambinder, Hyun Gyu Lee, Hyemi Kim, Jae Myun Lee, Pil Gu Park, Seung Myung Dong, Eun Jung Kim, Tae Hyun Choi, Jianmeng Chen
المصدر: Oncotarget
مصطلحات موضوعية: p53, Ganciclovir, Antimetabolites, Antineoplastic, Epstein-Barr Virus Infections, Herpesvirus 4, Human, Time Factors, viruses, Mice, SCID, Biology, Fialuridine, Transfection, Antiviral Agents, Deoxycytidine, Thymidine Kinase, Virus, Viral Proteins, Epstein-Barr virus associated gastric carcinoma, EBVaGC mouse model, Mice, Inbred NOD, Stomach Neoplasms, In vivo, Cell Line, Tumor, Antineoplastic Combined Chemotherapy Protocols, medicine, Animals, Humans, Molecular Targeted Therapy, Dose-Response Relationship, Drug, Carcinoma, Drug Repositioning, gemcitabine, ataxia telangiectasia-mutated, Xenograft Model Antitumor Assays, Gemcitabine, Tumor Burden, Epstein-Barr virus-associated gastric carcinoma, Oncology, Lytic cycle, Thymidine kinase, Enzyme Induction, Cancer research, Female, RNA Interference, Virus Activation, Protein Kinases, Signal Transduction, Research Paper, medicine.drug
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c110e895a2cde1817a884763c073239
https://doi.org/10.18632/oncotarget.5041 -
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