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المؤلفون: Margaret E. Tome, Ines Batinic-Haberle
المصدر: Redox Biology
Redox Biology, Vol 25, Iss, Pp-(2019)مصطلحات موضوعية: GSSG, glutathione disulfide, S-glutathionylation, MnTE-3-PyP5+, Mn(III) meso-tetrakis(N-ethylpyridinium-3-yl)porphyrin, CuZnSOD, copper, zinc superoxide dismutase (SOD1), Biochemistry, MnTSPP3-, Mn(III) meso-tetrakis(4-sulfonatophenyl)porphyrin, 0302 clinical medicine, SOD mimics, GSH, glutathione, NF-кB, Nuclear factor кB, ProtCys-SH, reduced protein cysteine, Redox biology, lcsh:QH301-705.5, chemistry.chemical_classification, MnT-2-PyP+, Mn(III) meso-tetrakis(N-pyridinium-2-yl)porphyrin, Keap1, Kelch-like ECH-associated protein 1, iNOS, inducible nitric oxide synthase, MnTMOE-3-PyP5+, Mn(III) meso-tetrakis(N-(2′-methoxyethyl)-3-yl)porphyrin, HSP, heat shock protein, lcsh:Medicine (General), MCAO, middle cerebral artery occlusion: MEKK, mitogen-activated protein kinase kinase kinase 1, MnTnOct-2-PyP5+, Mn(III) meso-tetrakis(N-n-octylpyridinium-2-yl)porphyrin, IDH, isocitrate dehydrogenase, MnCl4TE-2-PyP5+, Mn(III) β-tetrachloro-meso-tetrakis(N-ethylpyridinium-2-yl)porphyrin, EC-SOD, extracellular superoxide dismutase (SOD3), MnTM-3-PyP5+, Mn(III) meso-tetrakis(N-methylpyridinium-3-yl)porphyrin, Article, MnTnOct-3-PyP5+, Mn(III) meso-tetrakis(N-n-octylpyridinium-3-yl)porphyrin, Txndc 5&9, Thioredoxin domain containing proteins 5&9, MnTBAP3-, Mn(III) meso-tetrakis(4-carboxylatophenyl)porphyrin, 03 medical and health sciences, In vivo, MnTnHex-2-PyP5+, Mn(III) meso-tetrakis(N-n-hexylpyridinium-2-yl)porphyrin, BMX-001, Humans, MnTE-2-PyPhP5+, Mn(III) meso-tetrakis(phenyl-4′-(N-ethylpyridinium-2-yl))porphyrin, •NO, nitric oxide, MnBr8TM-3 or 4-PyP4+, Mn(II) beta-octabromo-meso-terakis(N-methylpyridium-3(or 4)-yl)porphyrin, MnCl5TE-2-PyP5+, Mn(III) β-pentachloro-meso-tetrakis(N-ethylpyridinium-2-yl)porphyrin, Mn porphyrins, MnSOD, manganese superoxide dismutase (SOD2), LPx, lipid peroxidase, O2•-, superoxide, 030104 developmental biology, chemistry, Protein cysteines, 030217 neurology & neurosurgery, MAPK, mitogen-activated protein kinase, 0301 basic medicine, MnTnBu-2-PyP5+, Mn(III) meso-tetrakis(N-n-butylpyridinium-2-yl)porphyrin, Clinical Biochemistry, MnTF3Pen-2-PyP5+, Mn(III) meso-tetrakis(N-5′5′,5′-trifluoropentylpyridinium-2-yl)porphyrin, BMX-003, MnTM-4-PyP5+, Mn(III) meso-tetrakis(N-methylpyridinium-4-yl)porphyrin, GST, glutathione S-transferase, polycyclic compounds, S-Glutathionylation, MnTnPr-2-PyP5+, Mn(III) meso-tetrakis(N-n-propylpyridinium-2-yl)porphyrin, lcsh:R5-920, biology, GPx, glutathione peroxidase, MnSOD, Mn superoxide dismutase (SOD2), NOX, NADPH oxidase, PKC, Protein kinase C, MnCl3TE-2-PyP5+, Mn(III) β-trichloro-meso-tetrakis(N-ethylpyridinium-2-yl)porphyrin, H2O2, hydrogen peroxide, Thiol, Thermodynamics, MnTE-4-PyP5+, Mn(III) meso-tetrakis(N-ethylpyridinium-4-yl)porphyrin, MnTnBuOE-2-PyP5+, MnBuOE (BMX-001), Mn(III) meso-tetrakis(N-(2′-n-butoxyethyl)pyridinium-2-yl)porphyrin (BMX-001), Oxidation-Reduction, SO32-, sulfite, Cys, Cysteine, TrxR, thioredoxin reductase, MnT-4-PyP+, Mn(III) meso-tetrakis(N-pyridinium-4-yl)porphyrin, Porphyrins, ONOO-, peroxynitrite (stands for both ONOO- and ONOOH), Redox, Catalysis, MnP, manganese porphyrins, Superoxide dismutase, MnTnHex-3-PyP5+, Mn(III) meso-tetrakis(N-n-hexylpyridinium-3-yl)porphyrin, Thiol signaling, ClO-, hypochlorite, Animals, In patient, Sulfhydryl Compounds, MnTnHex-4-PyP5+, Mn(III) meso-tetrakis(N-n-hexylpyridinium-4-yl)porphyrin, Manganese, MnTPhE-2-PyP5+, Mn(III) meso-tetrakis(N-(2′-phenylethyl)pyridinium-2-yl)porphyrin, Superoxide Dismutase, Organic Chemistry, Nrf2, nuclear factor E2-related factor 2, lcsh:Biology (General), MnTE-2-PyP5+, Mn(III) meso-tetrakis(N-ethylpyridinium-2-yl)porphyrin (AEOL10113, BMX-010, MnE), biology.protein, Prx, peroxiredoxin, NHE, normal hydrogen electrode, MnTFE-2-PyP5+, Mn(III) meso-tetrakis(N-fluoroethylpyridinium-2-yl)porphyrn, BMX-002, MnTnHexOE-2-PyP5+, Mn(III) meso-tetrakis(N-(2′-n-hexoxyethyl)pyridinium-2-yl)porphyrin
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المؤلفون: D. Neil Granger, Peter R. Kvietys
المصدر: Redox Biology
مصطلحات موضوعية: EC-SOD, extracellular superoxide dismutase, O2, molecular oxygen, Uncoupled nitric oxide synthase, nNOS, neuronal nitric oxide synthase, Biochemistry, TNF-α, tumor necrosis factor-α, IFN-γ, interferon-γ, DHE, dihydroethidine, Xanthine oxidase, IL-6, interleukin-6, NOS, nitric oxide synthase, chemistry.chemical_classification, Nox, NADPH oxidase, Ischemia-reperfusion, RBC, red blood cell, Duox, dual oxidase, IMAC, inner membrane anion channel, FAD, flavin adenine dinucleotide, Cell Hypoxia, Cell biology, iNOS, inducible nitric oxide synthase, NADPH, Nicotinamide adenine dinucleotide phosphate, UCP, uncoupling protein, AP-1, activator protein-1, MAO, monoamine oxidase, DPI, diphenyliodonium, CuZn SOD, copper–zinc superoxide dismutase, MPTP, mitochondrial permeability transition pore, H/R, hypoxia-reoxygenation, GAG, glycosaminoglycans, PKC, protein kinase C, Humans, PR-39, synthetic peptide inhibitor of Nox, XOR, xanthine oxidoreductase (XD+XO), PAF, platelet activating factor, NO, nitric oxide, NO2-, nitrite ion, XO, xanthine oxidase, NADPH oxidase, EC, endothelial cell, medicine.disease, HIF-1α, hypoxia inhibitory factor-1α, PDH, pyruvate dehydrogenase, XDH, xanthine dehydrogenase, DHFR, dihydrofolate reductase, mtROS, mitochondrial reactive oxygen species, chemistry, RET, reverse electron transport, mtNOS, mitochondrial nitric oxide synthase, α-GPD, α-glycerophosphate dehydrogenase, TCA, tricarboxyl acid, Reactive Oxygen Species, NNT, NADP-transhydrogenase, Clinical Biochemistry, A/R, anoxia-reoxygenation, ICAM-1, intercellular adhesion molecule-1, Mitochondrion, medicine.disease_cause, NFkB, nuclear factor kappa-B, RIRR, ROS-induced ROS release, chemistry.chemical_compound, Trx, thioredoxin, NAD+, Nicotinamide adenine dinucleotide (oxidized), GTPCH, guanosine triphosphate cyclohydrolase I, biology, GPx, glutathione peroxidase, eNOS, endothelial nitric oxide synthase, I/R, ischemia-reperfusion, DHR, dihyrdrorhodamine, Mitochondria, Nitric oxide synthase, ∆ψ, membrane potential, H2O2, hydrogen peroxide, Reperfusion Injury, ESR, electron spin resonance, Oxidation-Reduction, Research Paper, IL-1β, interleukin-1beta, FADH2, reduced FAD, α-KDH, α-ketoglutarate dehydrogenase, LTB4, leukotriene B4, ETC, electron transport chain, CoQ, coenzyme Q, ROS, reactive oxygen species, SOD, superoxide dismutase, medicine, Animals, MnSOD, manganese superoxide dismutase, O2·-, superoxide anion, Reactive oxygen species, Organic Chemistry, BH4, tetrahydrobiopterin, PEG-, polyethylene glycol conjugated, NADH, Nicotinamide adenine dinucleotide (reduced), Oxidative Stress, DCF, dichlorofluorescein, biology.protein, BM, bone marrow, Prx, peroxiredoxin, NAD+ kinase, Hypoxia-reoxygenation, Reperfusion injury, Oxidative stress
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المصدر: Redox Biology, Vol 1, Iss 1, Pp 24-31 (2013)
Gottfredsen, R H, Larsen, U G, Enghild, J J & Petersen, S V 2013, ' Hydrogen peroxide induce modifications of human extracellular superoxide dismutase that results in enzyme inhibition ', Redox Biology, vol. 1, no. 1, pp. 24-31 . https://doi.org/10.1016/j.redox.2012.12.004
Redox Biologyمصطلحات موضوعية: Models, Molecular, EC-SOD, extracellular superoxide dismutase, Peroxidase activity, Short Communication, Clinical Biochemistry, Oxidative phosphorylation, Peptide Mapping, Biochemistry, Mass Spectrometry, DDC, diethyldithiocarbamate, TFA, trifluoroacetic acid, Superoxide dismutase, chemistry.chemical_compound, MALDI, matrix assisted laser desorption/ionization, Catalytic Domain, Oxidation, Humans, DMPO, 5,5-dimethyl-pyrroline N-oxide, Histidine, EC-SOD, Hydrogen peroxide, lcsh:QH301-705.5, Bond cleavage, Peroxidase, FA, formic acid, Inhibition, chemistry.chemical_classification, Reactive oxygen species, lcsh:R5-920, biology, Superoxide Dismutase, Superoxide, Organic Chemistry, Active site, Hydrogen Peroxide, Zinc, chemistry, lcsh:Biology (General), biology.protein, lcsh:Medicine (General), Oxidation-Reduction, Copper
وصف الملف: application/pdf
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4دورية أكاديمية
المؤلفون: Gottfredsen RH; Department of Biomedicine, Aarhus University, DK-8000 Aarhus, Denmark., Larsen UG, Enghild JJ, Petersen SV
المصدر: Redox biology [Redox Biol] 2013 Jan 11; Vol. 1, pp. 24-31. Date of Electronic Publication: 2013 Jan 11 (Print Publication: 2013).
نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't
بيانات الدورية: Publisher: Elsevier, B.V Country of Publication: Netherlands NLM ID: 101605639 Publication Model: eCollection Cited Medium: Internet ISSN: 2213-2317 (Electronic) Linking ISSN: 22132317 NLM ISO Abbreviation: Redox Biol Subsets: MEDLINE
مواضيع طبية MeSH: Hydrogen Peroxide/*metabolism , Peroxidase/*metabolism , Superoxide Dismutase/*antagonists & inhibitors , Superoxide Dismutase/*metabolism, Catalytic Domain ; Copper/metabolism ; Histidine ; Humans ; Mass Spectrometry ; Models, Molecular ; Oxidation-Reduction ; Peptide Mapping ; Superoxide Dismutase/chemistry ; Zinc/metabolism
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