نتائج البحث - "Elena Lasorsa"

المساهمون: Crescenzo, Ramona, Abate, Francesco, Lasorsa, Elena, Tabbo', Fabrizio, Gaudiano, Marcello, Chiesa, Nicoletta, Di Giacomo, Filomena, Spaccarotella, Elisa, Barbarossa, Luigi, Ercole, Elisabetta, Todaro, Maria, Boi, Michela, Acquaviva, Andrea, Ficarra, Elisa, Novero, Domenico, Rinaldi, Andrea, Tousseyn, Thoma, Rosenwald, Andrea, Kenner, Luka, Cerroni, Lorenzo, Tzankov, Alexander, Ponzoni, Maurilio, Paulli, Marco, Weisenburger, Denni, Chan, Wing C, Iqbal, Javeed, Piris, Miguel A, Zamo', Alberto, Ciardullo, Carmela, Rossi, Davide, Gaidano, Gianluca, Pileri, Stefano, Tiacci, Enrico, Falini, Brunangelo, Shultz, Leonard D, Mevellec, Laurence, Vialard, Jorge E, Piva, Roberto, Bertoni, Francesco, Rabadan, Raul, Inghirami, Giorgio, Ramona Crescenzo, Francesco Abate, Elena Lasorsa, Fabrizio Tabbo’, Marcello Gaudiano, Nicoletta Chiesa, Filomena Di Giacomo, Elisa Spaccarotella, Luigi Barbarossa, Elisabetta Ercole, Maria Todaro, Michela Boi, ACQUAVIVA, ANDREA, FICARRA, ELISA, Domenico Novero, Andrea Rinaldi, Thomas Tousseyn, Andreas Rosenwald, Lukas Kenner, Lorenzo Cerroni, Alexander Tzankov, Maurilio Ponzoni, Marco Paulli, Dennis Weisenburger, Wing C. Chan, Javeed Iqbal, Miguel A. Piri, Alberto Zamo’, Carmela Ciardullo, Davide Rossi, Gianluca Gaidano, Stefano Pileri, Enrico Tiacci, Brunangelo Falini, Leonard D. Shultz, Laurence Mevellec, Jorge E. Vialard, Roberto Piva, Francesco Bertoni, Raul Rabadan, Giorgio Inghirami

مصطلحات موضوعية: Cancer Research, Lymphoma, Somatic cell, massive sequencing, Activating Transcription Factor 3, Animals, Cell Line, Tumor, HEK293 Cells, Humans, Janus Kinase 1, Lymphoma, Large-Cell, Anaplastic, Mice, Mutant Chimeric Proteins, NF-kappa B, Phosphorylation, Proto-Oncogene Proteins, Receptor Protein-Tyrosine Kinases, STAT3 Transcription Factor, Signal Transduction, TYK2 Kinase, Gene Expression Regulation, Neoplastic, Cell Biology, Oncology, Medicine (all), Anaplastic Large Cell Lymphoma, JAK/STAT3 pathway, Oncogenic STAT3 Activation, gene fusions, 0302 clinical medicine, hemic and lymphatic diseases, RNA-Seq data, anaplastic large cell lymphoma (ALCL), driver genetic alterations, somatic point mutations, copy number alterations, Anaplastic, Anaplastic lymphoma kinase, Anaplastic large-cell lymphoma, 0303 health sciences, Tumor, Janus kinase 1, Kinase, Large-Cell, 3. Good health, driver genetic alteration, somatic point mutation, Tyrosine kinase 2, 030220 oncology & carcinogenesis, Tyrosine kinase, Biology, Article, Cell Line, 03 medical and health sciences, ROS1, medicine, 030304 developmental biology, Stat3 activation, Neoplastic, Point mutation, gene fusion, medicine.disease, Molecular biology, Gene Expression Regulation, Cancer cell, Cancer research

وصف الملف: STAMPA

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::87a9d987f67c88511b8a93dfda4325af
http://hdl.handle.net/11583/2598380

7

Involvement of Grb2 Adaptor Protein in Nucleophosmin-Anaplastic Lymphoma Kinase (NPM-ALK)-mediated Signaling and Anaplastic Large Cell Lymphoma Growth

المؤلفون: Chiara Ambrogio, Elena Lasorsa, Ludovica Riera, Roberto Chiarle, Nadia Surrenti, Claudia Voena

المصدر: Journal of Biological Chemistry. 285:26441-26450

مصطلحات موضوعية: Oncogene Proteins, Fusion, grb2, lymphoma, Protein Tyrosine Phosphatase, Non-Receptor Type 11, Biochemistry, Translocation, Genetic, 03 medical and health sciences, 0302 clinical medicine, ALK, hemic and lymphatic diseases, Tumor Cells, Cultured, medicine, Humans, Anaplastic lymphoma kinase, Anaplastic Lymphoma Kinase, Phosphorylation, Molecular Biology, Anaplastic large-cell lymphoma, Cell Proliferation, GRB2 Adaptor Protein, 030304 developmental biology, 0303 health sciences, Nucleophosmin, Binding Sites, integumentary system, biology, Nuclear Proteins, Receptor Protein-Tyrosine Kinases, Signal transducing adaptor protein, Molecular Bases of Disease, Cell Biology, Protein-Tyrosine Kinases, medicine.disease, Fusion protein, Cell biology, 030220 oncology & carcinogenesis, biology.protein, Cancer research, Lymphoma, Large-Cell, Anaplastic, GRB2, Signal transduction, Protein Binding, Signal Transduction

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c4387f28ca482b2fe202f19dba9ac52
https://doi.org/10.1074/jbc.m110.116327

8

A novel patient-derived tumorgraft model with TRAF1-ALK anaplastic large-cell lymphoma translocation

المساهمون: Abate, F., Todaro, M., Van Der Krogt, J.-A., Boi, M., Landra, I., Machiorlatti, R., Tabbò, F., Messana, K., Abele, C., Barreca, A., Novero, D., Gaudiano, M., Aliberti, S., Di Giacomo, F., Tousseyn, T., Lasorsa, E., Crescenzo, R., Bessone, L., Ficarra, E., Acquaviva, A., Rinaldi, A., Ponzoni, M., Longo, D.L., Aime, S., Cheng, M., Ruggeri, B., Piccaluga, P.P., Pileri, S., Tiacci, E., Falini, B., Pera-Gresely, B., Cerchietti, L., Iqbal, J., Chan, W.C., Shultz, L.D., Kwee, I., Piva, R., Wlodarska, I., Rabadan, R., Bertoni, F., Inghirami, G., The European T-cell Lymphoma Study Group [.., Agostinelli, C., ], European T-cell Lymphoma Study Group, Cavallo, F., Chiesa, N., Fienga, A., di Giacomo, F., Marchiorlatti, R., Martinoglio, B., Medico, E., Ferrero, GB., Mereu, E., Pellegrino, E., Scafò, I., Spaccarotella, E., Ubezzi, I., Urigu, S., Chiapella, A., Vitolo, U., Agnelli, L., Neri, A., Chilosi£££Anna Caliò Marco£££ AC., Zamó, A., Facchetti, F., Lonardi, S., De Chiara, A., Fulciniti, F., Ferreri, A., Piccaluga, PP., Van Loo, P., De Wolf-Peeters, C., Geissinger, E., Muller-Hermelink, HK., Rosenwald, A., Piris, MA., Rodriguez, ME., Chiattone, C., Paes, RA., Abate, F, Todaro, M, van der Krogt, Ja, Boi, M, Landra, I, Machiorlatti, R, Tabbò, F, Messana, K, Abele, C, Barreca, A, Novero, D, Gaudiano, M, Aliberti, S, Di Giacomo, F, Tousseyn, T, Lasorsa, E, Crescenzo, R, Bessone, L, Ficarra, E, Acquaviva, A, Rinaldi, A, Ponzoni, M, Longo, Dl, Aime, S, Cheng, M, Ruggeri, B, Piccaluga, Pp, Pileri, S, Tiacci, E, Falini, B, Pera-Gresely, B, Cerchietti, L, Iqbal, J, Chan, Wc, Shultz, Ld, Kwee, I, Piva, R, Wlodarska, I, Rabadan, R, Bertoni, F, Inghirami, G, andThe European T-cell Lymphoma Study, Group

المصدر: Leukemia, vol. 29, no. 6, pp. 1390-1401

مصطلحات موضوعية: Pathology, Cancer Research, Lymphoma, TRAF1, Messenger, Drug Resistance, Translocation, Genetic, Fusion gene, Mice, Mice, Inbred NOD, hemic and lymphatic diseases, Tumor Cells, Cultured, Anaplastic lymphoma kinase, Anaplastic, Anaplastic Lymphoma Kinase, Anaplastic large-cell lymphoma, Animals, Blotting, Western, Flow Cytometry, Gene Expression Profiling, High-Throughput Nucleotide Sequencing, Humans, Immunoprecipitation, In Situ Hybridization, Fluorescence, Lymphoma, Large-Cell, Anaplastic, NF-kappa B, Proteasome Inhibitors, Proto-Oncogene Proteins c-myc, RNA, Messenger, Real-Time Polymerase Chain Reaction, Receptor Protein-Tyrosine Kinases, Repressor Proteins, Reverse Transcriptase Polymerase Chain Reaction, Signal Transduction, TNF Receptor-Associated Factor 1, Tumor Suppressor Protein p53, Xenograft Model Antitumor Assays, Drug Resistance, Neoplasm, In Situ Hybridization, Hematology, Cultured, Blotting, Medicine (all), Large-Cell, Tumor Cells, Proteasome Inhibitor, Receptor Protein-Tyrosine Kinase, Oncology, Western, Human, medicine.medical_specialty, fusion detection tool, Xenograft Model Antitumor Assay, medicine.drug_class, Translocation, Anesthesiology and Pain Medicine, Biology, anaplastic large-cell lymphomas (ALCL), RNA-Seq data, Fluorescence, Article, Genetic, Internal medicine, PRDM1, medicine, traslocation, Animal, Repressor Protein, medicine.disease, ALK inhibitor, anaplastic lymphoma kinase (ALK), Cancer research, Inbred NOD, RNA, Neoplasm, Positive Regulatory Domain I-Binding Factor 1, Lymphoma, Large-Cell, Anaplastic/drug therapy, Lymphoma, Large-Cell, Anaplastic/genetics, NF-kappa B/genetics, NF-kappa B/metabolism, Proteasome Inhibitors/pharmacology, Proto-Oncogene Proteins c-myc/genetics, Proto-Oncogene Proteins c-myc/metabolism, RNA, Messenger/genetics, Receptor Protein-Tyrosine Kinases/genetics, Receptor Protein-Tyrosine Kinases/metabolism, Repressor Proteins/genetics, Repressor Proteins/metabolism, TNF Receptor-Associated Factor 1/genetics, TNF Receptor-Associated Factor 1/metabolism, Translocation, Genetic/genetics, Tumor Suppressor Protein p53/genetics, Tumor Suppressor Protein p53/metabolism