نتائج البحث - "Fibrosis chemically induced"

المصدر: Graversen, M, Detlefsen, S, Pfeiffer, P, Lundell, L & Mortensen, M B 2018, ' Severe peritoneal sclerosis after repeated pressurized intraperitoneal aerosol chemotherapy with oxaliplatin (PIPAC OX) : report of two cases and literature survey ', Clinical and Experimental Metastasis, vol. 35, no. 3, pp. 103–108 . https://doi.org/10.1007/s10585-018-9895-9

مصطلحات موضوعية: Male, Cancer Research, Complications, medicine.medical_treatment, Carcinoid Tumor/drug therapy, Gastroenterology, Intraperitoneal chemotherapy, Severity of Illness Index, 0302 clinical medicine, Infusions, Parenteral, Prospective Studies, Prospective cohort study, Laparoscopy, Goblet cell carcinoid, Peritoneal Neoplasms, medicine.diagnostic_test, Adenocarcinoma, Mucinous/drug therapy, Peritoneal Fibrosis, General Medicine, Encapsulated peritoneal sclerosis, Adenocarcinoma, Mucinous, Antineoplastic Agents/administration & dosage, Oxaliplatin, Oncology, Appendiceal Neoplasms, 030220 oncology & carcinogenesis, Adenocarcinoma, 030211 gastroenterology & hepatology, medicine.drug, Insufflation, Adult, medicine.medical_specialty, PIPAC, Antineoplastic Agents, Carcinoid Tumor, 03 medical and health sciences, Oxaliplatin/administration & dosage, Internal medicine, medicine, Humans, Aged, Aerosols, Chemotherapy, business.industry, Peritoneal Fibrosis/chemically induced, Peritoneal Neoplasms/drug therapy, medicine.disease, Peritoneal sclerosis, Peritoneal metastasis, Appendiceal Neoplasms/drug therapy, Literature survey, business

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0ce535ed01e2c7778d03053c864c1777
https://pubmed.ncbi.nlm.nih.gov/29705882

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4

Nano-risk Science: application of toxicogenomics in an adverse outcome pathway framework for risk assessment of multi-walled carbon nanotubes

المؤلفون: Sarah Labib, Andrew Williams, Håkan Wallin, Sabina Halappanavar, Carole L. Yauk, Jake K. Nikota, Ulla Vogel

المصدر: Particle and Fibre Toxicology
Labib, S, Williams, A, Yauk, C L, Nikota, J K, Wallin, H, Vogel, U & Halappanavar, S 2016, ' Nano-risk Science : application of toxicogenomics in an adverse outcome pathway framework for risk assessment of multi-walled carbon nanotubes ', Particle and Fibre Toxicology, vol. 13, 15 . https://doi.org/10.1186/s12989-016-0125-9
Labib, S, Williams, A, Yauk, C L, Nikota, J K, Wallin, H, Vogel, U B & Halappanavar, S 2016, ' Nano-risk Science: application of toxicogenomics in an adverse outcome pathway framework for risk assessment of multi-walled carbon nanotubes ', Particle and Fibre Toxicology, vol. 13, no. 1 . https://doi.org/10.1186/s12989-016-0125-9

مصطلحات موضوعية: 0301 basic medicine, Adverse outcome pathways, Male, Time Factors, Transcription, Genetic, Health, Toxicology and Mutagenesis, Pulmonary Fibrosis, Engineered nanomaterials, Gene regulatory network, Gene Expression Regulation/drug effects, 02 engineering and technology, Pharmacology, Toxicology, Toxicogenetics, Transcription, Genetic/drug effects, Mice, Adverse Outcome Pathway, Databases, Genetic, Nanotechnology, Gene Regulatory Networks, Lung, Oligonucleotide Array Sequence Analysis, Lung fibrosis, General Medicine, Nano, 021001 nanoscience & nanotechnology, Toxicogenomics, Benchmarking, Nanotubes, Carbon/toxicity, 0210 nano-technology, Risk assessment, Case study, Computational biology, Biology, Risk Assessment, 03 medical and health sciences, SDG 3 - Good Health and Well-being, Gene Regulatory Networks/drug effects, Animals, Humans, Benchmark dose, Lung/drug effects, Dose-Response Relationship, Drug, Nanotubes, Carbon, Research, Gene Expression Profiling, Pulmonary Fibrosis/chemically induced, Computational Biology, Gene expression profiling, 030104 developmental biology, Gene Expression Regulation, Gene Expression Profiling/methods

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5108492ee9d8627a7ae2cccc99d3235e
http://europepmc.org/articles/PMC4792104

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5

Selective activation of angiotensin AT2 receptors attenuates progression of pulmonary hypertension and inhibits cardiopulmonary fibrosis

المؤلفون: Bruce, E, Shenoy, V, Rathinasabapathy, A, Espejo, A, Horowitz, A, Oswalt, A, Francis, J, Nair, A, Unger, T, Raizada, M K, Steckelings, Ulrike Muscha, Sumners, C, Katovich, M J

المصدر: Bruce, E, Shenoy, V, Rathinasabapathy, A, Espejo, A, Horowitz, A, Oswalt, A, Francis, J, Nair, A, Unger, T, Raizada, M K, Steckelings, U M, Sumners, C & Katovich, M J 2015, ' Selective activation of angiotensin AT 2 receptors attenuates progression of pulmonary hypertension and inhibits cardiopulmonary fibrosis ', British Journal of Pharmacology, vol. 172, no. 9, pp. 2219-2231 . https://doi.org/10.1111/bph.13044

مصطلحات موضوعية: Male, Myocardium/metabolism, Pyridines, Angiotensin II Type 2 Receptor Blockers/pharmacology, Hypertension, Pulmonary, Pulmonary Fibrosis, Ventricular Dysfunction, Right, Proto-Oncogene Proteins/antagonists & inhibitors, Ventricular Remodeling/drug effects, Angiotensin II Type 2 Receptor Blockers, Vascular Remodeling, Proto-Oncogene Mas, Receptor, Angiotensin, Type 2, Receptors, G-Protein-Coupled, Rats, Sprague-Dawley, Signal Transduction/drug effects, Proto-Oncogene Proteins, Animals, Angiotensin II/analogs & derivatives, Ventricular Dysfunction, Right/chemically induced, Lung, Imidazoles/pharmacology, Pyridines/pharmacology, Receptors, G-Protein-Coupled/antagonists & inhibitors, Peptide Fragments/pharmacology, Monocrotaline, Lung/drug effects, Hypertrophy, Right Ventricular, Ventricular Remodeling, Angiotensin II, Myocardium, Ventricular Function, Right/drug effects, Hemodynamics, Imidazoles, Pulmonary Fibrosis/chemically induced, Hypertrophy, Right Ventricular/chemically induced, Cardiovascular Agents, Receptor, Angiotensin, Type 2/agonists, Research Papers, Fibrosis, Peptide Fragments, Disease Models, Animal, Hemodynamics/drug effects, Cardiovascular Agents/pharmacology, Hypertension, Pulmonary/chemically induced, Ventricular Function, Right, Vascular Remodeling/drug effects, Signal Transduction

URL الوصول: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::b3e044c48f01f109844d8f467705bbdd
https://europepmc.org/articles/PMC4403089/

6

The mineralocorticoid receptor antagonist eplerenone reduces renal interstitial fibrosis after long-term cyclosporine treatment in rat: antagonizing cyclosporine nephrotoxicity

المؤلفون: Boye L. Jensen, Pernille Lund Hansen, Niels Marcussen, Peter Bie, Finn Thomsen Nielsen

المصدر: Nielsen, F T, Jensen, B L, Hansen, P B, Marcussen, N & Bie, P 2013, ' The mineralocorticoid receptor antagonist eplerenone reduces renal interstitial fibrosis after long-term cyclosporine treatment in rat : antagonizing cyclosporine nephrotoxicity ', B M C Nephrology, vol. 14, no. 1, 42 . https://doi.org/10.1186/1471-2369-14-42
BMC Nephrology

مصطلحات موضوعية: Male, medicine.medical_specialty, Immunosuppressive Agents/adverse effects, Renal function, Spironolactone, Kidney/drug effects, Kidney, Nephropathy, Nephrotoxicity, Rats, Sprague-Dawley, chemistry.chemical_compound, Mineralocorticoid receptor, Nephritis, Interstitial/chemically induced, Internal medicine, Renin, medicine, Animals, Drug Interactions, Cyclosporine/adverse effects, Longitudinal Studies, Aldosterone, Mineralocorticoid Receptor Antagonists, Fibrosis/chemically induced, business.industry, Calcineurin, Spironolactone/administration & dosage, Mineralocorticoid Receptor Antagonists/administration & dosage, medicine.disease, Fibrosis, Rats, Eplerenone, Treatment Outcome, Endocrinology, medicine.anatomical_structure, chemistry, Nephrology, Hypertension, Cyclosporine, Nephritis, Interstitial, business, Immunosuppressive Agents, Research Article, medicine.drug