يعرض 1 - 9 نتائج من 9 نتيجة بحث عن '"Forkhead Transcription Factors physiology"', وقت الاستعلام: 0.96s تنقيح النتائج
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    المساهمون: University of Zurich

    المصدر: Evolution; international journal of organic evolution, vol. 73, no. 9, pp. 1774-1792
    Evolution; International Journal of Organic Evolution

    وصف الملف: EVO-73-1774.pdf - application/pdf; application/pdf

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    المصدر: PLoS Genetics
    PLoS Genetics, Vol 14, Iss 6, p e1007379 (2018)
    Aalto, A P, Nicastro, I A, Broughton, J P, Chipman, L B, Schreiner, W P, Chen, J S & Pasquinelli, A E 2018, ' Opposing roles of microRNA Argonautes during Caenorhabditis elegans aging ', PLoS Genetics, vol. 14, no. 6, e1007379 . https://doi.org/10.1371/journal.pgen.1007379

    مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Nematoda, Molecular biology, Biochemistry, RNA interference, Sequencing techniques, Gene expression, Medicine and Health Sciences, Developmental, Genetics (clinical), Caenorhabditis elegans, Genes, Helminth, Regulation of gene expression, Gene Expression Regulation, Developmental, Longevity/genetics, Insulin/genetics, Eukaryota, Forkhead Transcription Factors/physiology, RNA sequencing, Animal Models, Argonaute, Cell biology, Nucleic acids, Phenotypes, Insulin-Like Growth Factor I/metabolism, Genetic interference, Experimental Organism Systems, Caenorhabditis Elegans, Argonaute Proteins, Argonaute Proteins/physiology, Epigenetics, Anatomy, Receptor, Research Article, lcsh:QH426-470, Insulin/metabolism, RNA, Helminth/physiology, Longevity, Biology, Research and Analysis Methods, 03 medical and health sciences, Caenorhabditis elegans Proteins/genetics, Model Organisms, Receptor, Insulin/genetics, Helminth, Genetics, Animals, MicroRNAs/physiology, Non-coding RNA, Transcription factor, Gene, Ecology, Evolution, Behavior and Systematics, Biology and life sciences, Signal Transduction/physiology, Organisms, Helminth/physiology, biology.organism_classification, Invertebrates, Gene regulation, Gastrointestinal Tract, Insulin receptor, lcsh:Genetics, MicroRNAs, 030104 developmental biology, Molecular biology techniques, Gene Expression Regulation, Genes, Mutation, biology.protein, Caenorhabditis elegans/physiology, Caenorhabditis, RNA-Binding Proteins/physiology, RNA, Digestive System

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    المساهمون: Faculty of Behavioural and Movement Sciences, Neuromechanics, AMS - Ageing and Morbidity, Dulbecco Telethon Institute, Venetian Institute Molecular Medicine (VIMM), CNR Institute of Neuroscience, National Research Council [Italy] (CNR), Department of Biomedical Sciences, Universita degli Studi di Padova, Department of Anatomy, Histology, Forensic Medicine and Orthopedic [Roma] (DAHFMO), Institut Pasteur, Fondation Cenci Bolognetti - Istituto Pasteur Italia, Fondazione Cenci Bolognetti, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome], Department of internal Medicine, University of Amsterdam [Amsterdam] (UvA), Department of Gerontology and Geriatrics, Leiden University Medical Center (LUMC), Department of Rehabilitation, Centre de recherche Croissance et signalisation (UMR_S 845), Centre National de la Recherche Scientifique (CNRS)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Paris Descartes - Paris 5 (UPD5), ICREA Infection Biology Laboratory (Department of Experimental and Health Sciences), Universitat Pompeu Fabra [Barcelona] (UPF), Department of Biobehavioral Health, Pennsylvania State University (Penn State), Penn State System-Penn State System, Department of Neuroscience, Clinical Neurophysiology, Uppsala University, Supported by the EC FP7 Project MYOAGE (to LL, ABM, PM-C, AM, MP, CR, MS, and SS), Fondation Thierry Latran, MIUR and AFM (to AM), the Swedish Research Council (8651 to LL), MDA, Ministerio de Ciencia e Innovacion of Spain SAF2012-38547, FIS-PS09/01267, PLE2009-0124, Marató/TV3 and AFM (to PM-C and ALS), EU interregio project PANGEA (to CR), King Gustaf V and Queen Victoria’s Foundation, and the National Institute of Health Grants (AG-14731, AR-45627 and AR-47318 to LL)., European Project: 223576,EC:FP7:HEALTH,FP7-HEALTH-2007-B,MYOAGE(2009), Università degli Studi di Padova = University of Padua (Unipd), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Università degli Studi di Roma 'La Sapienza' = Sapienza University [Rome] (UNIROMA), Universiteit Leiden-Universiteit Leiden, Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), MOVE Research Institute, Internal medicine, Venetian Institute of Molecular Medicine, Padua, Department of Anatomy, Histology, Forensic Medicine and Orthopedic, Università degli Studi di Roma 'La Sapienza' [Rome]-Réseau International des Instituts Pasteur (RIIP)-Institut Pasteur, Fondation Cenci Bolognetti - Istituto Pasteur Italia, Fondazione Cenci Bolognetti, Réseau International des Instituts Pasteur (RIIP), Venetian Institute of Molecular Medicine, Padova, Italy, Universitat Pompeu Fabra [Barcelona]

    المصدر: Biogerontology, 14(3), 303-23. Springer Netherlands
    Biogerontology
    Biogerontology, Springer Verlag, 2013, 14 (3), pp.303-23. ⟨10.1007/s10522-013-9432-9⟩
    Biogerontology; Vol 14
    Sandri, M, Barberi, L, Bijlsma, A Y, Blaauw, B, Dyar, K A, Milan, G, Mammucari, C, Meskers, C G M, Pallafacchina, G, Paoli, A, Pion, D, Roceri, M, Romanello, V, Serrano, A L, Toniolo, L, Larsson, L, Maier, A B, Munoz-Canoves, P, Musaro, A, Pende, M, Reggiani, C, Rizzuto, R & Schiaffino, S 2013, ' Signalling pathways regulating muscle mass in ageing skeletal muscle. The role of the IGF1-Akt-mTOR-FoxO pathway ', Biogerontology, vol. 14, no. 3, pp. 303-323 . https://doi.org/10.1007/s10522-013-9432-9
    Biogerontology, 2013, 14 (3), pp.303-23. ⟨10.1007/s10522-013-9432-9⟩
    Biogerontology, 14(3), 303-323. Springer Netherlands
    Biogerontology, 14(3), 303-323
    Sandri, M, Barberi, L, Bijlsma, A Y, Blaauw, B, Dyar, K A, Milan, G, Mammucari, C, Meskers, C G M, Pallafacchina, G, Paoli, A, Pion, D, Roceri, M, Romanello, V, Serrano, A L, Toniolo, L, Larsson, L, Maier, A B, Muñoz-Cánoves, P, Musarò, A, Pende, M, Reggiani, C, Rizzuto, R & Schiaffino, S 2013, ' Signalling pathways regulating muscle mass in ageing skeletal muscle : the role of the IGF1-Akt-mTOR-FoxO pathway ', Biogerontology, vol. 14, no. 3, pp. 303-23 . https://doi.org/10.1007/s10522-013-9432-9

    مصطلحات موضوعية: Male, Sarcopenia, FOXO1, mTORC1, Transgenic, Muscle hypertrophy, Sarcopenia/physiopathology, MESH: Serpin E2, Mice, SKP Cullin F-Box Protein Ligases/genetics, 0302 clinical medicine, Models, Serpin E2, 80 and over, MESH: Aging, MESH: Animals, MESH: Sarcopenia, Aged, 80 and over, Muscle Proteins/genetics, 0303 health sciences, Insulin-Like Growth Factor I/physiology, TOR Serine-Threonine Kinases, IGF1, Forkhead Transcription Factors/physiology, MESH: Young Adult, Mice, Inbred DBA, MESH: SKP Cullin F-Box Protein Ligases, medicine.medical_specialty, Knockout, muscle atrophy, signaling, aging, IGF-1, 03 medical and health sciences, MESH: Muscle Proteins, SDG 3 - Good Health and Well-being, MESH: Forkhead Transcription Factors, Skeletal/physiology, Serpin E2/genetics, Humans, Inbred DBA, Ubiquitin-Protein Ligases/genetics, Protein kinase B, Aged, MESH: Adolescent, MESH: Humans, MESH: Mice, Inbred DBA, MESH: Proto-Oncogene Proteins c-akt, Animal, MESH: Adult, medicine.disease, MESH: Ubiquitin-Protein Ligases, Mice, Inbred C57BL, Muscle, Skeletal/physiology, Ageing, MESH: Microtubule-Associated Proteins, Endocrinology, TOR Serine-Threonine Kinases/physiology, FoxO, Geriatrics and Gerontology, Gerontology, MESH: Female, 030217 neurology & neurosurgery, MESH: Signal Transduction, MESH: Insulin-Like Growth Factor I, Muscle Proteins, Inbred C57BL, MESH: Mice, Knockout, Autophagy-Related Protein 7, Tripartite Motif Proteins, MESH: Aged, 80 and over, Insulin-Like Growth Factor I, MESH: Aged, Mice, Knockout, MESH: Muscle, Skeletal, Forkhead Box Protein O1, Forkhead Transcription Factors, medicine.anatomical_structure, MESH: Models, Animal, Models, Animal, mTOR, Muscle, Female, Microtubule-Associated Proteins, Signal Transduction, Adult, Adolescent, MESH: Mice, Transgenic, Ubiquitin-Protein Ligases, Mice, Transgenic, Protein degradation, Biology, Young Adult, MESH: Mice, Inbred C57BL, Internal medicine, medicine, Animals, [SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology, Muscle, Skeletal, MESH: Mice, PI3K/AKT/mTOR pathway, MESH: TOR Serine-Threonine Kinases, 030304 developmental biology, SKP Cullin F-Box Protein Ligases, Signal Transduction/physiology, Akt, Skeletal muscle, Proto-Oncogene Proteins c-akt/physiology, Microtubule-Associated Proteins/genetics, MESH: Male, Aging/physiology, Proto-Oncogene Proteins c-akt

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    المصدر: Developmental cell
    Developmental Cell, Vol. 22, No 2 (2012) pp. 430-45

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    المصدر: Ormestad, M, Astorga (Johansson), J, Landgren, H, Wang, T, Johansson, B R, Miura, N & Carlsson, P 2006, ' Foxf1 and Foxf2 control murine gut development by limiting mesenchymal Wnt signaling and promoting extracellular matrix production ', Development, vol. 133, no. 5, pp. 833-43 . https://doi.org/10.1242/dev.02252

    وصف الملف: application/pdf

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