نتائج البحث - "G Del Poeta"

المساهمون: Pozzo, F., Bittolo, T., Vendramini, E., Bomben, R., Bulian, P., Rossi, F. M., Zucchetto, A., Tissino, E., Degan, M., D’Arena, G., Di Raimondo, F., Zaja, F., Pozzato, Gabriele, Rossi, D., Gaidano, G., Del Poeta, G., Gattei, V., Dal Bo, M.

المصدر: Leukemia. 31:2407-2415

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Small interfering RNA, Chronic lymphocytic leukemia, Genes, myc, Ribosome biogenesis, NOTCH1, hemic and lymphatic diseases, Tumor Cells, Cultured, Chronic, Receptor, Notch1, Leukemia, Cultured, Cell Proliferation, Coculture Techniques, Humans, Leukemia, Lymphocytic, Chronic, B-Cell, Nuclear Proteins, Ribosomes, Signal Transduction, Up-Regulation, Mutation, Hematology, Transfection, myc, Lymphocytic, Tumor Cells, Oncology, embryonic structures, cardiovascular system, NPM1, biological phenomena, cell phenomena, and immunity, Signal transduction, Nucleophosmin, Receptor, 03 medical and health sciences, medicine, business.industry, B-Cell, Settore MED/15, medicine.disease, Chronic lymphocytis leukemia, 030104 developmental biology, Genes, Cancer research, Oncogene MYC, sense organs, business, Chromatin immunoprecipitation

وصف الملف: ELETTRONICO

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9d5cf20bb6db264c861f747f8d703848
https://doi.org/10.1038/leu.2017.90

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7

NOTCH1 mutations are associated with high CD49d expression in chronic lymphocytic leukemia: Link between the NOTCH1 and the NF-Îº B pathways

المساهمون: Benedetti, D., Tissino, E., Pozzo, F., Bittolo, T., Caldana, C., Perini, C., Martorelli, D., Bravin, V., D'Agaro, T., Rossi, F. M., Bomben, R., Santinelli, E., Zaja, F., Pozzato, G., Chiarenza, A., Di Raimondo, F., Del Poeta, G., Rossi, D., Gaidano, G., Dal Bo, M., Gattei, V., Zucchetto, A.

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Chronic lymphocytic leukemia, Integrin alpha4, CD49d, medicine.disease_cause, chemistry.chemical_compound, 0302 clinical medicine, Hematology, Anesthesiology and Pain Medicine, hemic and lymphatic diseases, NOTCH1 mutation, Chronic, Receptor, Notch1, Leukemic, Mutation, Leukemia, Gene Expression Regulation, Leukemic, NF-kappa B, Transfection, Lymphocytic, Oncology, 030220 oncology & carcinogenesis, embryonic structures, cardiovascular system, biological phenomena, cell phenomena, and immunity, Signal transduction, Receptor, Signal Transduction, Biology, Humans, Leukemia, Lymphocytic, Chronic, B-Cell, 03 medical and health sciences, medicine, Notch1, CLL, B-Cell, NF-κB, medicine.disease, NFKB1, Settore MED/15, 030104 developmental biology, chemistry, Gene Expression Regulation, Immunology, sense organs, Trisomy

وصف الملف: STAMPA

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8eb29baf4ebc0704f62cde27dc64d60
http://hdl.handle.net/11368/2955777

8

PS1152 THE USE OF THE BCL-2 INHIBITOR IN CLL PATIENTS WHO PROGRESSED AFTER B-CELL-RECEPTOR INHIBITORS: A RETROSPECTIVE MULTICENTER ITALIAN EXPERIENCE

المصدر: HemaSphere. 3:522-523

مصطلحات موضوعية: Bcl-2 Inhibitor, business.industry, B-cell receptor, Cancer research, Medicine, Hematology, business

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::9d80b00b9c4dbfda24b81a15967410bc
https://doi.org/10.1097/01.hs9.0000562892.50367.9f

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9

PS1127 SF3B1 MUTATIONS ASSOCIATE WITH LOW CD20 EXPRESSION IN CLL: ANOTHER NOTCH1-DEPENDENT MECHANISM?

المؤلفون: Elena Vendramini, G Del Poeta, Gabriele Pozzato, Annalisa Chiarenza, L Laurenti, Tamara Bittolo, V. Gattei, Giovanni D'Arena, Federico Pozzo, F. Di Raimondo, Francesco Zaja, Riccardo Bomben, Antonella Zucchetto, M. Dal Bo

المصدر: HemaSphere. 3:510-511

مصطلحات موضوعية: CD20, biology, Expression (architecture), Chemistry, Mechanism (biology), biology.protein, Hematology, Cell biology

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::ce79540313b1825de2af79d856b884ae
https://doi.org/10.1097/01.hs9.0000562792.25366.c6

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10

NOTCH1 mutations associate with low CD20 level in chronic lymphocytic leukemia: evidence for a NOTCH1 mutation-driven epigenetic dysregulation

المساهمون: Pozzo, F., Bittolo, Tamara, Arruga, F., Bulian, P., Macor, Paolo, Tissino, E., Gizdic, B., Rossi, F. M., Bomben, Riccardo, Zucchetto, Antonella, Benedetti, D., Degan, MARIA CHIARA, D'Arena, G., Chiarenza, Arianna, Zaja, F., Pozzato, Gabriele, Rossi, D., Gaidano, G., Del Poeta, G., Deaglio, S., Gattei, V., Dal Bo, M.

المصدر: Leukemia. 30:182-189

مصطلحات موضوعية: 0301 basic medicine, Cancer Research, Small interfering RNA, Chronic lymphocytic leukemia, NOTCH1, CD20, B-CLL, RPBJ, HDAC, Histone Deacetylase 2, Histone Deacetylase 1, Epigenesis, Genetic, 0302 clinical medicine, Hematology, Anesthesiology and Pain Medicine, immune system diseases, hemic and lymphatic diseases, LYMPHOMA-CELLS, Histone Deacetylase Inhibitor, Chronic, Receptor, Notch1, Regulation of gene expression, Leukemia, CANCER, Lymphocytic, Gene Expression Regulation, Neoplastic, Oncology, Immunoglobulin J Recombination Signal Sequence-Binding Protein, 030220 oncology & carcinogenesis, embryonic structures, SURVIVAL, Receptor, CLINICAL-SIGNIFICANCE, CLL CELLS, EXPRESSION, RITUXIMAB, DIAGNOSIS, INHIBITORS, Human, Antigens, CD20, Histone Deacetylase Inhibitors, Humans, Leukemia, Lymphocytic, Chronic, B-Cell, Mutation, Repressor, Biology, 03 medical and health sciences, Genetic, medicine, Antigens, Transcription factor, Notch1, Neoplastic, RBPJ, B-Cell, medicine.disease, 030104 developmental biology, Gene Expression Regulation, biology.protein, Cancer research, Settore MED/15 - Malattie del Sangue, Epigenesis

وصف الملف: STAMPA

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::eee87be427067340069c81ea443e6f84
https://doi.org/10.1038/leu.2015.182

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