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1رسالة جامعية
المؤلفون: Hammarfjord, Oscar
مصطلحات موضوعية: Toll-Like Receptors -- biosynthesis, Toll-Like Receptors -- immunology, Toll-Like Receptors -- metabolism, Dendritic Cells -- immunology, Dendritic Cells -- metabolism, Cold Temperature, Gelsolin -- genetics, Gelsolin -- metabolism, Actins -- metabolism, Ligands, Phagocytosis
URL الوصول: http://hdl.handle.net/10616/40746
Degree: Diss. (sammanfattning) Stockholm : Karolinska institutet, 2011
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2AAV9 delivered bispecific nanobody attenuates amyloid burden in the gelsolin amyloidosis mouse model
المؤلفون: Adriaan Verhelle, Lynn Supply, Thierry Vandendriessche, Olivier Zwaenepoel, Marinee K Chuah, Cindy Peleman, Jan Gettemans, Tony Lahoutte, Nisha Nair, Nick Devoogdt, Wim Derave, Inge Everaert, Wouter Van Overbeke, Jo Van Dorpe
المساهمون: Cell Biology and Histology, Basic (bio-) Medical Sciences, Division of Gene Therapy & Regenerative Medicine, Supporting clinical sciences, Medical Imaging, Translational Imaging Research Alliance
المصدر: ResearcherID
مصطلحات موضوعية: 0301 basic medicine, Genetic enhancement, viruses, Mutant, 02 engineering and technology, 01 natural sciences, Amyloid disease, Antibodies, Bispecific, Furin, Genetics (clinical), Dependovirus/genetics, Medicine(all), Amyloidosis, Amyloidosis/genetics, General Medicine, Dependovirus, 021001 nanoscience & nanotechnology, musculoskeletal system, Single-Domain Antibodies/administration & dosage, Gelsolin/genetics, 0210 nano-technology, Furin/immunology, gelsolin gene, mice, Matrix Metalloproteinase 14/immunology, macromolecular substances, Biology, 010402 general chemistry, 03 medical and health sciences, Genetics, medicine, Matrix Metalloproteinase 14, Point Mutation, Humans, Animals, Amyloid burden, Point Mutation/genetics, Binding site, Molecular Biology, Antibodies, Bispecific/immunology, Actin, Gelsolin, 030102 biochemistry & molecular biology, Single-Domain Antibodies, medicine.disease, Molecular biology, 0104 chemical sciences, Disease Models, Animal, 030104 developmental biology, biology.protein, Cancer research, genetic therapy
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6dec09a43eab319b7c3e4e92b447c4c5
https://hdl.handle.net/20.500.14017/864eadef-40ed-4e81-aefe-4f9bebb9cd4a -
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المؤلفون: Barbara Yermen, Alejandra Tomas, Philippe A. Halban
المصدر: Diabetes, Vol. 56, No 1 (2007) pp. 80-87
مصطلحات موضوعية: medicine.medical_specialty, Cell type, Cell Survival, Ratón, Cell Survival/ physiology, Endocrinology, Diabetes and Metabolism, Apoptosis, Enzyme-Linked Immunosorbent Assay, macromolecular substances, Biology, Insulin-Secreting Cells/ cytology/ physiology, Mice, Insulin-Secreting Cells, Internal medicine, Internal Medicine, medicine, Animals, Gelsolin/genetics/ physiology, RNA, Small Interfering, Gelsolin, ddc:616, RNA, Small Interfering/genetics, Regulation of gene expression, Gene knockdown, TUNEL assay, musculoskeletal system, Molecular biology, B-1 cell, Endocrinology, Gene Expression Regulation, Apoptosis/ physiology, RNA Interference
