نتائج البحث - "Gustav Collden"

1

دورية أكاديمية

Pirt deficiency has subtle female-specific effects on energy and glucose metabolism in mice

المؤلفون: Sigrid Jall, Brian Finan, Gustav Collden, Katrin Fischer, Xinzhong Dong, Matthias H. Tschöp, Timo D. Müller, Christoffer Clemmensen

المصدر: Molecular Metabolism, Vol 23, Iss , Pp 75-81 (2019)

مصطلحات موضوعية: Internal medicine, RC31-1245

وصف الملف: electronic resource

Relation: http://www.sciencedirect.com/science/article/pii/S2212877818312109; https://doaj.org/toc/2212-8778

URL الوصول: https://doaj.org/article/d018742e0c064bc5990c9502af8d06ee

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2

Inceptor counteracts insulin signalling in β-cells to control glycaemia

المصدر: Nature 590, 326–331 (2021)
Journal of Diabetes Investigation

مصطلحات موضوعية: Blood Glucose, Male, 0301 basic medicine, medicine.medical_treatment, Golgi Apparatus, Endoplasmic Reticulum, Insulin Antagonists, Mice, 0302 clinical medicine, Insulin-Secreting Cells, Insulin, Insulin-Like Growth Factor I, Receptor, Glucose tolerance test, Multidisciplinary, medicine.diagnostic_test, biology, Chemistry, Endocytosis, Neoplasm Proteins, Knockout mouse, hormones, hormone substitutes, and hormone antagonists, Signal Transduction, endocrine system, medicine.medical_specialty, 030209 endocrinology & metabolism, Cell Line, 03 medical and health sciences, Commentaries, Internal medicine, medicine, Animals, Humans, Cell Proliferation, Cell Size, Insulin-like growth factor 1 receptor, Growth factor, Insulin-like growth factor 2 receptor, Membrane Proteins, nutritional and metabolic diseases, Glucose Tolerance Test, Clathrin, Receptor, Insulin, Tamoxifen, Insulin receptor, 030104 developmental biology, Endocrinology, Commentary, biology.protein, Endocrine Cells, Lysosomes

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28cffb16a5b0db7181203ac08e24aad3
https://doi.org/10.1038/s41586-021-03225-8

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3

GLP-1-mediated delivery of the PPAR𝛼/𝛾 dual-agonist Tesaglitazar improves obesity and glucose metabolism in mice

مصطلحات موضوعية: Agonist, chemistry.chemical_classification, endocrine system, medicine.medical_specialty, Tesaglitazar, business.industry, medicine.drug_class, digestive, oral, and skin physiology, Peroxisome proliferator-activated receptor, Carbohydrate metabolism, medicine.disease, Obesity, chemistry.chemical_compound, Endocrinology, chemistry, Internal medicine, Medicine, business, hormones, hormone substitutes, and hormone antagonists

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::abbe6c86c35688a6dbf16211c6cfbeea
https://doi.org/10.21203/rs.3.rs-1081959/v1

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GLP-1-mediated delivery of tesaglitazar improves obesity and glucose metabolism in male mice

المصدر: Quarta, C, Stemmer, K, Novikoff, A, Yang, B, Klingelhuber, F, Harger, A, Bakhti, M, Bastidas-Ponce, A, Baugé, E, Campbell, J E, Capozzi, M, Clemmensen, C, Collden, G, Cota, P, Douros, J, Drucker, D J, DuBois, B, Feuchtinger, A, Garcia-Caceres, C, Grandl, G, Hennuyer, N, Herzig, S, Hofmann, S M, Knerr, P J, Kulaj, K, Lalloyer, F, Lickert, H, Liskiewicz, A, Liskiewicz, D, Maity, G, Perez-Tilve, D, Prakash, S, Sanchez-Garrido, M A, Zhang, Q, Staels, B, Krahmer, N, DiMarchi, R D, Tschöp, M H, Finan, B & Müller, T D 2022, ' GLP-1-mediated delivery of tesaglitazar improves obesity and glucose metabolism in male mice ', Nature Metabolism, vol. 4, no. 8, pp. 1071-1083 . https://doi.org/10.1038/s42255-022-00617-6
Nature Metabolism
Nat. Metab. 4, 1071-1083 (2022)

مصطلحات موضوعية: Alkanesulfonates, Male, Phenylpropionates, Endocrinology, Diabetes and Metabolism, Body Weight, Cell Biology, Glucagon-Like Peptide-1 Receptor, Mice, Glucose, Diabetes Mellitus, Type 2, Glucagon-Like Peptide 1, Physiology (medical), Internal Medicine, Animals, PPAR alpha, ddc:610, Obesity

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7d8cb25c43d6de39a162496e77b8cbe0
https://pubmed.ncbi.nlm.nih.gov/35995998

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The glucose-dependent insulinotropic polypeptide (GIP) regulates body weight and food intake via CNS-GIPR signaling

المصدر: Cell Metabolism, 33 (4)
Cell Metabolism
Cell Metab. 33, 833-844.e5 (2021)

مصطلحات موضوعية: 0301 basic medicine, Central Nervous System, Male, Food intake, food intake, Physiology, Eating, Mice, 0302 clinical medicine, Glucagon-Like Peptide 1, Premovement neuronal activity, 2. Zero hunger, GIP, GIPR CNS KO, incretin, 3. Good health, Peripheral, ddc, Cns, Gip, Gipr Cns Ko, Body Weight, Diet-induced Obesity, Food Intake, Glucose Metabolism, Incretin, Type 2 Diabetes, Metabolic effects, Glucose-dependent insulinotropic polypeptide, type 2 diabetes, CNS, Proto-Oncogene Proteins c-fos, hormones, hormone substitutes, and hormone antagonists, Signal Transduction, medicine.medical_specialty, diet-induced obesity, glucose metabolism, Hypothalamus, Mice, Transgenic, Gastric Inhibitory Polypeptide, Biology, Carbohydrate metabolism, Body weight, Diet, High-Fat, Article, Receptors, Gastrointestinal Hormone, 03 medical and health sciences, body weight, Internal medicine, medicine, Animals, Humans, ddc:610, Obesity, Molecular Biology, Cell Biology, medicine.disease, Mice, Inbred C57BL, 030104 developmental biology, Endocrinology, 030217 neurology & neurosurgery

وصف الملف: application/application/pdf; application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4ad196864aba83940728afa229ef093f
https://hdl.handle.net/20.500.11850/478121

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The Glucose-Dependent Insulinotropic Polypeptide (GIP) Regulates Body Weight and Food Intake Via CNS-GIPR Signaling

المصدر: SSRN Electronic Journal.

مصطلحات موضوعية: endocrine system, medicine.medical_specialty, Food intake, Lateral hypothalamus, digestive, oral, and skin physiology, Wild type, Type 2 diabetes, Biology, Body weight, medicine.disease, Obesity, Endocrinology, Internal medicine, medicine, Glucose-dependent insulinotropic polypeptide, Premovement neuronal activity, hormones, hormone substitutes, and hormone antagonists

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::f79aeb9542d6bf63e1a383daa8faa6b4
https://doi.org/10.2139/ssrn.3667144

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Pirt deficiency has subtle female-specific effects on energy and glucose metabolism in mice

المؤلفون: Timo D. Müller, Brian Finan, Xinzhong Dong, Katrin Fischer, Christoffer Clemmensen, Matthias H. Tschöp, Sigrid Jall, Gustav Collden

المصدر: Jall, S, Finan, B, Collden, G, Fischer, K, Dong, X, Tschoep, M H, Mueller, T D & Clemmensen, C 2019, ' Pirt deficiency has subtle female-specific effects on energy and glucose metabolism in mice ', Molecular Metabolism, vol. 23, pp. 75-81 . https://doi.org/10.1016/j.molmet.2019.02.011
Molecular Metabolism
Mol. Metab. 23, 75-81 (2019)
Molecular Metabolism, Vol 23, Iss, Pp 75-81 (2019)

مصطلحات موضوعية: 0301 basic medicine, Male, Regulator, Brown adipose tissue, Energy homeostasis, Gene Knockout Techniques, Mice, 0302 clinical medicine, Adipose Tissue, Brown, Diet, Fat-Restricted, Mice, Knockout, Thermogenesis, Corrigenda, ddc, medicine.anatomical_structure, Female, TRPM8, lcsh:Internal medicine, medicine.medical_specialty, TRPM Cation Channels, 030209 endocrinology & metabolism, Context (language use), Pyrimidinones, Biology, Carbohydrate metabolism, Diet, High-Fat, Brief Communication, 03 medical and health sciences, Sex Factors, Internal medicine, Glucose Intolerance, Sex differences, medicine, Animals, Obesity, lcsh:RC31-1245, Molecular Biology, Signaling Molecule, Sex Differences, Body Weight, Energy Metabolism, Trpm8, Brown Adipose Tissue, Signaling molecule, Membrane Proteins, Cell Biology, Energy metabolism, Glucose Tolerance Test, Body weight, Mice, Inbred C57BL, Calcium Channel Agonists, 030104 developmental biology, Endocrinology, Glucose, Metabolic control analysis, Carrier Proteins, Transcriptome