نتائج البحث - "Hans-Hermann Hoepken"

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دورية أكاديمية

Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

المؤلفون: Sylvia Torres-Odio, Jana Key, Hans-Hermann Hoepken, Júlia Canet-Pons, Lucie Valek, Bastian Roller, Michael Walter, Blas Morales-Gordo, David Meierhofer, Patrick N. Harter, Michel Mittelbronn, Irmgard Tegeder, Suzana Gispert, Georg Auburger

المصدر: Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-26 (2017)

مصطلحات موضوعية: Parkinson’s disease, Ubiquitin kinase PINK1, Mitochondrial dysfunction, Antiviral response, Neuroinflammation, Neurology. Diseases of the nervous system, RC346-429

وصف الملف: electronic resource

Relation: http://link.springer.com/article/10.1186/s12974-017-0928-0; https://doaj.org/toc/1742-2094

URL الوصول: https://doaj.org/article/37f1030eeb624662a92964439fd188fb

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دورية أكاديمية

Mitochondrial dysfunction, peroxidation damage and changes in glutathione metabolism in PARK6

المؤلفون: Hans-Hermann Hoepken, Suzana Gispert, Blas Morales, Oliver Wingerter, Domenico Del Turco, Alexander Mülsch, Robert L. Nussbaum, Klaus Müller, Stefan Dröse, Ulrich Brandt, Thomas Deller, Brunhilde Wirth, Alexei P. Kudin, Wolfram S. Kunz, Georg Auburger

المصدر: Neurobiology of Disease, Vol 25, Iss 2, Pp 401-411 (2007)

مصطلحات موضوعية: Malondialdehyde, MnSOD, Glutathione, Mitochondria, Oxidative stress, PINK1, Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571

وصف الملف: electronic resource

Relation: http://www.sciencedirect.com/science/article/pii/S0969996106002580; https://doaj.org/toc/1095-953X

URL الوصول: https://doaj.org/article/13394e1023644fa291cb79c68e130fa8

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دورية أكاديمية

Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration.

المصدر: PLoS ONE, Vol 4, Iss 6, p e5777 (2009)

مصطلحات موضوعية: Medicine, Science

وصف الملف: electronic resource

Relation: https://www.ncbi.nlm.nih.gov/pmc/articles/pmid/19492057/?tool=EBI; https://doaj.org/toc/1932-6203

URL الوصول: https://doaj.org/article/45f68ab49b2d403fa1646e411e62f433

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Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

المؤلفون: Suzana Gispert, Blas Morales-Gordo, Georg Auburger, Lucie Valek, Bastian Roller, David Meierhofer, Júlia Canet-Pons, Jana Key, Irmgard Tegeder, Patrick N. Harter, Hans-Hermann Hoepken, Michel Mittelbronn, Michael Walter, Sylvia Torres-Odio

المصدر: Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-26 (2017)

مصطلحات موضوعية: 0301 basic medicine, Pathology, Aging, Parkinson's disease, Parkin, lcsh:RC346-429, chemistry.chemical_compound, Mice, Neuroblastoma, Ubiquitin, Neuroinflammation, Mitophagy, Cells, Cultured, Cerebral Cortex, Neurons, biology, General Neuroscience, Microfilament Proteins, Age Factors, Parkinson Disease, Endoplasmic Reticulum Stress, Ubiquitin ligase, medicine.anatomical_structure, Neurology, Antiviral response, Disease Progression, alpha-Synuclein, medicine.medical_specialty, RNA Splicing, Immunology, Mice, Transgenic, 03 medical and health sciences, Cellular and Molecular Neuroscience, medicine, Animals, Humans, ddc:610, lcsh:Neurology. Diseases of the nervous system, Alpha-synuclein, Research, Gene Expression Profiling, Calcium-Binding Proteins, Ubiquitination, Lipid Metabolism, Disease Models, Animal, 030104 developmental biology, chemistry, biology.protein, Unfolded protein response, Parkinson’s disease, Neuron, Mitochondrial dysfunction, Protein Kinases, Ubiquitin kinase PINK1

وصف الملف: application/pdf; application/octet-stream

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5af4076a588b6aedcfb22b791953189b
https://hdl.handle.net/10668/11465

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Additional file 9: Table S7. of Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

المؤلفون: Torres-Odio, Sylvia, Key, Jana, Hans-Hermann Hoepken, Canet-Pons, Júlia, Valek, Lucie, Roller, Bastian, Walter, Michael, Morales-Gordo, Blas, Meierhofer, David, Harter, Patrick, Mittelbronn, Michel, Tegeder, Irmgard, Gispert, Suzana, Auburger, Georg

مصطلحات موضوعية: nervous system, sense organs

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::e7031b8998e490513e8ced26d7e761ea

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Additional file 7: Table S6. of Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

المؤلفون: Torres-Odio, Sylvia, Key, Jana, Hans-Hermann Hoepken, Canet-Pons, Júlia, Valek, Lucie, Roller, Bastian, Walter, Michael, Morales-Gordo, Blas, Meierhofer, David, Harter, Patrick, Mittelbronn, Michel, Tegeder, Irmgard, Gispert, Suzana, Auburger, Georg

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::41797d07c706945b80109b42e88ef4f1

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Additional file 4: Table S3. of Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

المؤلفون: Torres-Odio, Sylvia, Key, Jana, Hans-Hermann Hoepken, JĂşlia Canet-Pons, Valek, Lucie, Roller, Bastian, Walter, Michael, Morales-Gordo, Blas, Meierhofer, David, Harter, Patrick, Mittelbronn, Michel, Tegeder, Irmgard, Gispert, Suzana, Auburger, Georg

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::c58effa9cb5628ce3f466669219f0d2e

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Additional file 6: Table S5. of Progression of pathology in PINK1-deficient mouse brain from splicing via ubiquitination, ER stress, and mitophagy changes to neuroinflammation

المؤلفون: Torres-Odio, Sylvia, Key, Jana, Hans-Hermann Hoepken, Canet-Pons, Júlia, Valek, Lucie, Roller, Bastian, Walter, Michael, Morales-Gordo, Blas, Meierhofer, David, Harter, Patrick, Mittelbronn, Michel, Tegeder, Irmgard, Gispert, Suzana, Auburger, Georg

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::6c6fe65581117d61adaff5c4c9c2a457

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Parkinson phenotype in aged PINK1-deficient mice is accompanied by progressive mitochondrial dysfunction in absence of neurodegeneration

المساهمون: Okazawa, Hitoshi

المصدر: PloS one, vol 4, iss 6
PLoS ONE, Vol 4, Iss 6, p e5777 (2009)
PLoS ONE

مصطلحات موضوعية: Male, Pathology, Aging, Biochemistry/Membrane Proteins and Energy Transduction, Mitochondrion, Neurodegenerative, Transgenic, chemistry.chemical_compound, Mice, Adenosine Triphosphate, Biochemistry/Cell Signaling and Trafficking Structures, 2.1 Biological and endogenous factors, Aetiology, Neurological Disorders/Movement Disorders, Neurons, Multidisciplinary, Parkinson's Disease, biology, Kinase, Dopaminergic, Neurodegeneration, Parkinson Disease, Neurodegenerative Diseases, Cell Biology/Cellular Death and Stress Responses, Mitochondria, Genetics and Genomics/Gene Function, Phenotype, Neurological, alpha-Synuclein, Medicine, Female, Drosophila melanogaster, Neuroscience/Neurobiology of Disease and Regeneration, Research Article, medicine.drug, medicine.medical_specialty, General Science & Technology, Science, Mice, Transgenic, PINK1, Dopamine, Internal medicine, medicine, Animals, ddc:610, Alpha-synuclein, Animal, Neurosciences, biology.organism_classification, medicine.disease, Brain Disorders, Disease Models, Animal, Endocrinology, Genetics and Genomics/Disease Models, chemistry, Gene Expression Regulation, Cell Biology/Neuronal and Glial Cell Biology, Disease Models, Protein Kinases

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::611713730c4a02690e05bbad2d47f6d9
https://escholarship.org/uc/item/38t6k0ps

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Parkinson patient fibroblasts show increased alpha-synuclein expression

المؤلفون: Helmuth Steinmetz, Donat Kögel, Georg Auburger, Filomena Ricciardi, Michael Bonin, Olaf Riess, Alexander Kurz, Hans-Hermann Hoepken, Suzana Gispert, Michael Klinkenberg, Thomas Gasser, Mekhman Azizov, Blas Morales-Gordo

المصدر: Experimental neurology. 212(2)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::985246b805ac8b7c26f897428c076272
https://pubmed.ncbi.nlm.nih.gov/19166835

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