يعرض 1 - 8 نتائج من 8 نتيجة بحث عن '"Hyperbilirubinemia etiology"', وقت الاستعلام: 2.20s تنقيح النتائج
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    المساهمون: Nutrition and Movement Sciences, RS: NUTRIM - R1 - Obesity, diabetes and cardiovascular health

    المصدر: American Journal of Physiology-Gastrointestinal and Liver Physiology, 318(1), G41-G52. American Physiological Society
    Guthrie, G, Stoll, B, Chacko, S, Lauridsen, C, Plat, J & Burrin, D G 2020, ' Rifampicin, not vitamin E, suppresses parenteral nutrition-associated liver disease development through the pregnane X receptor pathway in piglets ', American Journal of Physiology: Gastrointestinal and Liver Physiology, vol. 318, no. 1, pp. G41-G52 . https://doi.org/10.1152/ajpgi.00193.2019
    Am J Physiol Gastrointest Liver Physiol

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    المساهمون: González, M, Iglesias, J, Tiribelli, Claudio, Ribalta, J, Reyes, H, Hernández, I, Bianchi, M, Andrighetti, F, Molina, C.

    وصف الملف: STAMPA

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    مورد إلكتروني

    عناروين إضافية: Antioxidative Vitamine und Glucose-6-Phosphat-Dehydrogenase-Mangel bei ausgereiften Neugeborenen

    المصدر: GMS German Medical Science; VOL: 6; DOC10 /20080924/

    URL: http://www.egms.de/en/journals/gms/2008-6/000055.shtml
    Abdul-Razzak KK, Nusier MK, Obediat AD, Salim AM. Antioxidant vitamins and hyperbilirubinemia in neonates. GMS Ger Med Sci. 2007;5:Doc03. Available from: http://www.egms.de/en/gms/2007-5/000039.shtml
    Baydas G, Karatas F, Gursu MF, Bozkurt HA, Ilhan N, Yasar A, Canatan H. Antioxidant vitamin levels in term and preterm infants and their relation to maternal vitamin status. Arch Med Res. 2002;33(3):276-80. DOI: 10.1016/S0188-4409(02)00356-9
    Beutler E. Glucose-6-phosphate dehydrogenase deficiency: a historical perspective. Blood. 2008;111(1):16-24. DOI: 10.1182/blood-2007-04-077412
    Bizzarro MJ, Colson E, Ehrenkranz RA. Differential diagnosis and management of anemia in the newborn. Pediatr Clin North Am. 2004;51(4):1087-107, xi. DOI: 10.1016/j.pcl.2004.03.006
    Brouillard RP. Measurement of red blood cell life-span. JAMA. 1974;230(9):1304-5. DOI: 10.1001/jama.230.9.1304
    Catignani GL, Bieri JG. Simultaneous determination of retinol and alpha-tocopherol in serum or plasma by liquid chromatography. Clin Chem. 1983;29(4):708-12.
    Chan AC, Chow CK, Chiu D. Interaction of antioxidants and their implication in genetic anemia. Proc Soc Exp Biol Med. 1999;222(3):274-82. DOI: 10.1046/j.1525-1373.1999.d01-144.x
    Connolly AM, Volpe JJ. Clinical features of bilirubin encephalopathy. Clin Perinatol. 1990;17(2):371-9.
    Frank JE. Diagnosis and Management of G6PD Deficiency. Am Fam Physician. 2005;72(7):1277-82
    Gaetani GF, Rolfo M, Arena S, Mangerini R, Meloni GF, Ferraris AM. Active Involvement of Catalase During Hemolytic Crises of Favism. Blood. 1996;88(3):1084-8.
    Gordon-Smith EC. Investigation of the hereditary haemolytic anemia. In: Dacie JV, Lewis SM, editors. Practical haematology. Edinburgh: Churchill Livingstone, 1984. p. 152-78.
    Gross SJ. Vitamin E and neonatal bilirubinemia. Pediatrics. 1979;64(3):321-3.
    Jablonska-Skwiecinska E, Lewandowska I, Plochocka D, Topczewski J, Zimowski JG, Klopocka J, Burzynska B. Several mutations including two novel mutations of the glucose-6-phosphate dehydrogenase gene in Polish G6PD deficient subjects with chronic nonspherocytic hemolytic anemia, acute hemolytic anemia, and favism. Hum Mutat. 1999;14(6):477-84. DOI: 10.1002/(SICI)1098-1004(199912)14:6<477::AID-HUMU6>3.0.CO;2-X
    Jalloh S, Van Rostenberghe H, Yusoff NM, Ghazali S, Nik Ismail NZ, Matsuo M, Wahab NA, Nishio H. Poor correlation between hemolysis and jaundice in glucose 6-phosphate dehydrogenase-deficient babies. Pediatr Int. 2005;47(3):258-61. DOI: 10.1111/j.1442-200x.2005.02052.x
    Kaplan M, Vreman HJ, Hammerman C, Leiter C, Abramov A, Stevenson DK. Contribution of haemolysis to jaundice in Sephardic Jewish glucose-6-phosphate dehydrogenase deficient neonates. Br J Haematol. 1996;93(4):822-7.
    Kilic M, Turgut M, Taskin E, Cekmen M, Aygun AD. Nitric oxide levels and antioxidant enzyme activities in jaundices of premature infants. Cell Biochem Funct. 2004;22(5):339-42. DOI: 10.1002/cbf.1112
    Kondo H, Takahashi M, Niki E. Peroxynitrite-induced hemolysis of human erythrocytes and its inhibition by antioxidants. FEBS Lett. 1997;413(2):236-8. DOI: 10.1016/S0014-5793(97)00922-8
    Kwok CJ, Martin AC, Au SW, Lam VM. G6PDdb, an integrated database of glucose-6-phosphate dehydrogenase (G6PD) mutations. Hum Mutat. 2002;19(3):217-24. DOI: 10.1002/humu.10036
    Laosombat V, Sattayasevana B, Chotsampancharoen T, Wongchanchailert M. Glucose-6-Phosphate Dehydrogenase Variants Associated with Favism in Thai Children. Int J Hematol. 2006;83(2):139-43. DOI: 10.1532/IJH97.A20513
    Lau HK, Li CH, Lee AC. Acute massive haemolysis in children with glucose-6-phosphate dehydrogenase deficiency. Hong Kong Med J. 2006;12(2):149-51.
    Lim F, Vulliamy T, Abdalla SH. An Ashkenazi Jewish woman presenting with favism. J Clin Pathol. 2005;58(3):317-9. DOI: 10.1136/jcp.2004.017426
    Manco L, Gonçalves P, Macedo-Ribeiro S, Seabra C, Melo P, Ribeiro ML. Two new glucose-6-phosphate dehydrogenase mutations causing chronic hemolysis. Haematologica. 2005;90(8):1135-6.
    McCormick DB, Green HL. The vitamins. In: Burtis CA, Ashwood ER, editors. Tietz text book of clinical chemistry. Philadelphia: Saunders; 1999. p. 999-1028.
    Mehta A, Mason PJ, Vulliamy TJ. Glucose-6-phosphate dehydrogenase deficiency. Baillieres Best Pract Res Clin Haematol. 2000;13(1):21-38. DOI: 10.1053/beha.1999.0055
    Niki E, Yamamoto Y, Komuro E, Sato K. Membrane damage due to lipid oxidation. Am J Clin Nutr. 1991;53(1 Suppl):201S-205S.
    Niki E, Komuro E, Takahashi M, Urano S, Ito E, Terao K. Oxidative hemolysis of erythrocytes and its inhibition by free radical scavengers. J Biol Chem. 1988;263(36):19809-14.
    Ojo CO, Dawodu AH, Osifo BO. Vitamin E deficiency in the pathogenesis of haemolysis and hyperbilirubinaemia of neonatal jaundice. J Trop Pediatr. 1986;32(5):251-4.
    Pearson HA. Life-span of the fetal red blood cell. J Pediatr. 1967;70(2):166-71. DOI: 10.1016/S0022-3476(67)80410-4
    Strain JJ, Mulholland CW. Vitamin C and vitamin E - synergistic interaction in vivo? EXS. 1992;62:419-22.
    Turan Y. Prevalence of erythrocyte glucose-6-phosphate dehydrogenase (G6PD) deficiency in the population of western Turkey. Arch Med Res. 2006;37(7):880-2. DOI: 10.1016/j.arcmed.2006.03.005
    Turgut M, Basaran O, Cekmen M, Karatas F, Kurt A, Aygün AD. Oxidant and antioxidant levels in preterm newborns with idiopathic hyperbilirubinemia. J Paediatr Child Health. 2004;40(11):633-7. DOI: 10.1111/j.1440-1754.2004.00489.x
    Usanga EA, Ameen R. Glucose-6-Phosphate Dehydrogenase Deficiency in Kuwait, Syria, Egypt, Iran, Jordan and Lebanon. Hum Hered. 2000;50(3):158-61. DOI: 10.1159/000022906
    Valaes T. Severe neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency: pathogenesis and global epidemiology. Acta Paediatr Suppl. 1994;394:58-76. DOI: 10.1111/j.1651-2227.1994.tb13216.x
    Vanderpas J, Vertongen F. Erythrocyte vitamin E is oxidized at a lower peroxide concentration in neonates than in adults. Blood. 1985;66(6):1272-7.
    Weng YH, Chou YH, Lien RI. Hyperbilirubinemia in healthy neonates with glucose-6-phosphate dehydrogenase deficiency. Early Hum Dev. 2003;71(2):129-36. DOI: 10.1016/S0378-3782(03)00002-1
    Yeruchimovich M, Shapira B, Mimouni FB, Dollberg S. Neonatal Nucleated Red Blood Cells in G6PD Deficiency. Am J Perinatol. 2002;19(4):215-9. DOI: 10.1055/s-2002-28485

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