المؤلفون: Ikuru Chiba, Hiroyuki Tsutsui, Hidehisa Takahashi, Keiichi I. Nakayama, Masaki Matsumoto, Shouji Matsushima, Shigetsugu Hatakeyama, Junichi Matsumoto, Masaya Tsuda, Shingo Takada, Masashi Watanabe, Shun Nagashima, Shigeru Yanagi, Wataru Mizushima, Takaaki Furihata, Takashi Yokota, Shintaro Kinugawa

المصدر: Journal of molecular and cellular cardiology. 100:43-53

مصطلحات موضوعية: 0301 basic medicine, medicine.medical_specialty, Voltage-dependent anion channel, Ubiquitin-Protein Ligases, Regulator, Gene Expression, RNF207, Heart failure, Plasma protein binding, Cardiomyocyte, Mitochondrion, Mitochondria, Heart, Cell Line, 03 medical and health sciences, chemistry.chemical_compound, Mice, Ubiquitin, Stress, Physiological, Internal medicine, Gene expression, medicine, Animals, Humans, Myocytes, Cardiac, Protein Interaction Domains and Motifs, Amino Acid Sequence, Molecular Biology, biology, VDAC, Voltage-Dependent Anion Channel 1, Ubiquitination, Energy metabolism, medicine.disease, Rats, 030104 developmental biology, Endocrinology, chemistry, Animals, Newborn, Organ Specificity, biology.protein, Cardiology and Cardiovascular Medicine, Adenosine triphosphate, Protein Binding

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::74a646a1979f6c8e3f5b8406cce9d102
http://hdl.handle.net/2115/67491