نتائج البحث - "Jorge J. Palop"

1

دورية أكاديمية

Identifying behavioral structure from deep variational embeddings of animal motion

المؤلفون: Kevin Luxem, Petra Mocellin, Falko Fuhrmann, Johannes Kürsch, Stephanie R. Miller, Jorge J. Palop, Stefan Remy, Pavol Bauer

المصدر: Communications Biology, Vol 5, Iss 1, Pp 1-15 (2022)

مصطلحات موضوعية: Biology (General), QH301-705.5

وصف الملف: electronic resource

Relation: https://doaj.org/toc/2399-3642

URL الوصول: https://doaj.org/article/c4892a9a2ca94f1b999f09244ffe23aa

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دورية أكاديمية

Novel App knock-in mouse model shows key features of amyloid pathology and reveals profound metabolic dysregulation of microglia

المؤلفون: Dan Xia, Steve Lianoglou, Thomas Sandmann, Meredith Calvert, Jung H. Suh, Elliot Thomsen, Jason Dugas, Michelle E. Pizzo, Sarah L. DeVos, Timothy K. Earr, Chia-Ching Lin, Sonnet Davis, Connie Ha, Amy Wing-Sze Leung, Hoang Nguyen, Roni Chau, Ernie Yulyaningsih, Isabel Lopez, Hilda Solanoy, Shababa T. Masoud, Chun-chi Liang, Karin Lin, Giuseppe Astarita, Nathalie Khoury, Joy Yu Zuchero, Robert G. Thorne, Kevin Shen, Stephanie Miller, Jorge J. Palop, Dylan Garceau, Michael Sasner, Jennifer D. Whitesell, Julie A. Harris, Selina Hummel, Johannes Gnörich, Karin Wind, Lea Kunze, Artem Zatcepin, Matthias Brendel, Michael Willem, Christian Haass, Daniel Barnett, Till S. Zimmer, Anna G. Orr, Kimberly Scearce-Levie, Joseph W. Lewcock, Gilbert Di Paolo, Pascal E. Sanchez

المصدر: Molecular Neurodegeneration, Vol 17, Iss 1, Pp 1-29 (2022)

مصطلحات موضوعية: Neuritic plaques, Vascular amyloid, Neurodegeneration, Astrogliosis, Phagocytic microglia, Lipid dyshomeostasis, Neurology. Diseases of the nervous system, RC346-429, Geriatrics, RC952-954.6

وصف الملف: electronic resource

Relation: https://doaj.org/toc/1750-1326

URL الوصول: https://doaj.org/article/9eb417ef38964cb082e476c72c827f5f

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3

دورية أكاديمية

Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer’s disease mutations but not by inhibition of BACE1

المؤلفون: Erik C. B. Johnson, Kaitlyn Ho, Gui-Qiu Yu, Melanie Das, Pascal E. Sanchez, Biljana Djukic, Isabel Lopez, Xinxing Yu, Michael Gill, Weiping Zhang, Jeanne T. Paz, Jorge J. Palop, Lennart Mucke

المصدر: Molecular Neurodegeneration, Vol 15, Iss 1, Pp 1-26 (2020)

مصطلحات موضوعية: Alzheimer’s disease, Amyloid, APP, APP-KI, App NL-G-F, BACE, Neurology. Diseases of the nervous system, RC346-429, Geriatrics, RC952-954.6

وصف الملف: electronic resource

Relation: http://link.springer.com/article/10.1186/s13024-020-00393-5; https://doaj.org/toc/1750-1326

URL الوصول: https://doaj.org/article/241e604b9e154d01b93f232dcc4d0aa4

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4

دورية أكاديمية

GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer’s Disease Models

المصدر: Cell Reports, Vol 30, Iss 2, Pp 381-396.e4 (2020)

مصطلحات موضوعية: Biology (General), QH301-705.5

وصف الملف: electronic resource

Relation: http://www.sciencedirect.com/science/article/pii/S2211124719316808; https://doaj.org/toc/2211-1247

URL الوصول: https://doaj.org/article/086ee7e911424901b05bcdfb74e615c9

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5

Swollen axons impair neuronal circuits in Alzheimer’s disease

المؤلفون: Inma Cobos, Jorge J. Palop

المصدر: Nature. 612:218-220

مصطلحات موضوعية: Multidisciplinary

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::60cffa18cecd8bded319f72786c4a69a
https://doi.org/10.1038/d41586-022-03800-7

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6

Small-molecule ketone esters treat brain network abnormalities in an Alzheimer’s disease mouse model

المؤلفون: John C. Newman, Keran Ma, François Kroll, Erin Higgins, Scott Ulrich, Jorge J. Palop, Eric Verdin

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::ef8d64bc1020e6fff6d0ef9f89273d1e
https://doi.org/10.1101/2022.09.24.509337

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Microglial Gi-dependent dynamics regulate brain network hyperexcitability

المصدر: Nature Neuroscience. 24:19-23

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::d558dcdacc49cb06330157f8ecf84e06
https://doi.org/10.1038/s41593-020-00756-7

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GluN2A NMDA Receptor Enhancement Improves Brain Oscillations, Synchrony, and Cognitive Functions in Dravet Syndrome and Alzheimer’s Disease Models

المصدر: Cell Reports, Vol 30, Iss 2, Pp 381-396.e4 (2020)
Elsevier
Cell reports, vol 30, iss 2
Cell reports

مصطلحات موضوعية: 0301 basic medicine, Cyclopropanes, Male, Aging, Medical Physiology, Epilepsies, Myoclonic, Electroencephalography, Epilepsies, Neurodegenerative, Inbred C57BL, Alzheimer's Disease, memory, Epilepsy, Mice, 0302 clinical medicine, Cognition, Receptors, 2.1 Biological and endogenous factors, EEG, SCN1A, Aetiology, lcsh:QH301-705.5, MK-801, learning, Behavior, Animal, medicine.diagnostic_test, musculoskeletal, neural, and ocular physiology, Brain, medicine.anatomical_structure, Neurological, NMDA receptor, Mental health, N-Methyl-D-Aspartate, Allosteric modulator, Interneuron, Intellectual and Developmental Disabilities (IDD), Allosteric regulation, CHO Cells, interneuron, Receptors, N-Methyl-D-Aspartate, Article, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, Cricetulus, Dravet syndrome, Allosteric Regulation, Alzheimer Disease, Nitriles, Behavioral and Social Science, medicine, Acquired Cognitive Impairment, Animals, Humans, Behavior, business.industry, Animal, Nav1.1, Neurosciences, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), medicine.disease, Brain Disorders, Mice, Inbred C57BL, Disease Models, Animal, Thiazoles, 030104 developmental biology, nervous system, lcsh:Biology (General), Disease Models, Pyrazoles, epilepsy, Dementia, Biochemistry and Cell Biology, J20, business, Myoclonic, APP, Neuroscience, 030217 neurology & neurosurgery

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::97b0ddfb3fdc3d2e4aa7d6b4342a59d2
http://www.sciencedirect.com/science/article/pii/S2211124719316808

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9

Network abnormalities and interneuron dysfunction in Alzheimer’s disease

المؤلفون: Jorge J. Palop

المصدر: Alzheimer's & Dementia. 16

مصطلحات موضوعية: Interneuron, Epidemiology, business.industry, Health Policy, Perspective (graphical), Disease, Psychiatry and Mental health, Cellular and Molecular Neuroscience, medicine.anatomical_structure, Developmental Neuroscience, Medicine, Premovement neuronal activity, Neurology (clinical), Geriatrics and Gerontology, business, Neuroscience

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::280fe4442d6057b21a1475f645e16d13
https://doi.org/10.1002/alz.040396

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10

Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer’s disease mutations but not by inhibition of BACE1

المؤلفون: Jorge J. Palop, Biljana Djukic, Pascal E. Sanchez, Jeanne T. Paz, Kaitlyn Ho, Xinxing Yu, Lennart Mucke, Michael Gill, Weiping Zhang, Isabel Lopez, Erik C. B. Johnson, Gui-Qiu Yu, Melanie Das

المصدر: Molecular Neurodegeneration
Molecular neurodegeneration, vol 15, iss 1
Molecular Neurodegeneration, Vol 15, Iss 1, Pp 1-26 (2020)

مصطلحات موضوعية: 0301 basic medicine, Aging, App NL-G-F, Knock-in, Neurodegenerative, lcsh:Geriatrics, Hippocampal formation, Inbred C57BL, Transgenic, lcsh:RC346-429, Pathogenesis, I5, Mice, Amyloid beta-Protein Precursor, 0302 clinical medicine, Amyloid precursor protein, Aspartic Acid Endopeptidases, 2.1 Biological and endogenous factors, Gene Knock-In Techniques, Aetiology, Behavior, Animal, biology, Brain, Alzheimer's disease, Cell biology, Neurological, Alzheimer’s disease, APP-KI, Research Article, Genetically modified mouse, Amyloid, Inhibitor, C-Fos, Transgene, Clinical Sciences, Mice, Transgenic, Learning and memory, 03 medical and health sciences, Cellular and Molecular Neuroscience, SWD, Alzheimer Disease, Gene knockin, mental disorders, Acquired Cognitive Impairment, Genetics, Animals, Humans, BACE, Molecular Biology, lcsh:Neurology. Diseases of the nervous system, Epileptiform, Behavior, Neurology & Neurosurgery, Calbindin, Epilepsy, Animal, AppNL-G-F, Wild type, Neurosciences, Alzheimer's Disease including Alzheimer's Disease Related Dementias (AD/ADRD), Molecular medicine, Brain Disorders, Mice, Inbred C57BL, Disease Models, Animal, lcsh:RC952-954.6, 030104 developmental biology, App(NL-G-F), Oligomers, Disease Models, Mutation, biology.protein, Dementia, Neurology (clinical), Nerve Net, Amyloid Precursor Protein Secretases, J20, APP, 030217 neurology & neurosurgery

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0789322d9d12b4d8e8bef6dc52aae963
http://europepmc.org/articles/PMC7489007

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