المؤلفون: Julia Nedelcu, Christin Reinbach, Philipp Riedler, Matthias Brendel, Axel Rominger, Joel Kaye, Newshan Behrangi, Zhan Jiangshan, Christoph Schmitz, Markus Kipp

المصدر: Neurobiology of Disease, Vol 134, Iss , Pp - (2020)

مصطلحات موضوعية: Cuprizone, Laquinimod, Demyelination, Multiple sclerosis, Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571

وصف الملف: electronic resource

Relation: http://www.sciencedirect.com/science/article/pii/S096999611930350X; https://doaj.org/toc/1095-953X

URL الوصول: https://doaj.org/article/f5504117da3e49749314902ce6884ca3

المؤلفون: Anne Nack, Matthias Brendel, Julia Nedelcu, Markus Daerr, Stella Nyamoya, Cordian Beyer, Carola Focke, Maximilian Deussing, Chloé Hoornaert, Peter Ponsaerts, Christoph Schmitz, Peter Bartenstein, Axel Rominger, Markus Kipp

المصدر: Cells, Vol 8, Iss 2, p 94 (2019)

مصطلحات موضوعية: multiple sclerosis, cuprizone, TSPO, PET, neurodegeneration, Cytology, QH573-671

وصف الملف: electronic resource

Relation: https://www.mdpi.com/2073-4409/8/2/94; https://doaj.org/toc/2073-4409

URL الوصول: https://doaj.org/article/6fa24f04718143e2adb687355decacb4

المؤلفون: Paul van der Valk, Jiangshan Zhan, Stefan Gingele, Emily Santrau, Sandra Amor, Julia Frenz, Christoph Schmitz, Hannes Kaddatz, Markus Kipp, Julia Nedelcu, Michael Müller, Viktoria Gudi, Martin Stangel, Uta Chrzanowski, Sarah Joost, Brigitte Müller-Hilke, Theresa Greiner

المساهمون: AII - Inflammatory diseases, Amsterdam Neuroscience - Neuroinfection & -inflammation, Pathology

المصدر: GLIA, 69(4), 925-942. John Wiley and Sons Inc.
Kaddatz, H, Joost, S, Nedelcu, J, Chrzanowski, U, Schmitz, C, Gingele, S, Gudi, V, Stangel, M, Zhan, J, Santrau, E, Greiner, T, Frenz, J, Müller-Hilke, B, Müller, M, Amor, S, van der Valk, P & Kipp, M 2021, ' Cuprizone-induced demyelination triggers a CD8-pronounced T cell recruitment ', GLIA, vol. 69, no. 4, pp. 925-942 . https://doi.org/10.1002/glia.23937

مصطلحات موضوعية: 0301 basic medicine, T cell, CD8-Positive T-Lymphocytes, Biology, Cuprizone, Mice, 03 medical and health sciences, Cellular and Molecular Neuroscience, 0302 clinical medicine, Immune system, Antigen, medicine, Animals, Cytotoxic T cell, Neuroinflammation, Multiple sclerosis, medicine.disease, Oligodendrocyte, Mice, Inbred C57BL, Disease Models, Animal, Oligodendroglia, 030104 developmental biology, medicine.anatomical_structure, Neurology, Immunology, 030217 neurology & neurosurgery, CD8, Demyelinating Diseases

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cfbb8ce8f3cfd0f6fcf3bd7a2eac8c0e
https://doi.org/10.1002/glia.23937

المؤلفون: Joel Kaye, Julia Nedelcu, Zhan Jiangshan, Christoph Schmitz, Axel Rominger, Philipp Riedler, Christin Reinbach, Matthias Brendel, Markus Kipp, Newshan Behrangi

المصدر: Neurobiology of Disease, Vol 134, Iss, Pp-(2020)
Nedelcu, Julia; Reinbach, Christin; Riedler, Philipp; Brendel, Matthias; Rominger, Axel; Kaye, Joel; Behrangi, Newshan; Jiangshan, Zhan; Schmitz, Christoph; Kipp, Markus (2020). Laquinimod ameliorates secondary brain inflammation. Neurobiology of disease, 134, p. 104675. Elsevier 10.1016/j.nbd.2019.104675 <http://dx.doi.org/10.1016/j.nbd.2019.104675>

مصطلحات موضوعية: 0301 basic medicine, Pathology, medicine.medical_specialty, T cell, Inflammation, Neural degeneration, 610 Medicine & health, Quinolones, Myelin oligodendrocyte glycoprotein, lcsh:RC321-571, Multiple sclerosis, 03 medical and health sciences, chemistry.chemical_compound, Cuprizone, 0302 clinical medicine, Immune system, medicine, Animals, Gliosis, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, Laquinimod, biology, business.industry, Brain, medicine.disease, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, medicine.anatomical_structure, Neurology, chemistry, Forebrain, biology.protein, Encephalitis, Female, medicine.symptom, Demyelination, business, 030217 neurology & neurosurgery

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ad17cfbe433cd34d307102053d38271

المؤلفون: Tim Clarner, Christian Beißel, Anand Goswami, Felix Fischbach, Christoph Schmitz, Patrizia Leopold, Marion Victor, Cordian Beyer, Tanja Hochstrasser, Yasemin van Heuvel, Lara Nellessen, Joachim Weis, Jiangshan Zhan, Julia Nedelcu, Stella Nyamoya, Bernd Denecke, Markus Kipp

المصدر: Glia. 67(2)

مصطلحات موضوعية: 0301 basic medicine, Male, Monoamine Oxidase Inhibitors, Mice, Transgenic, Nerve Tissue Proteins, DNA damage-inducible transcript 3, Biology, Microgliosis, Corpus Callosum, 03 medical and health sciences, Cellular and Molecular Neuroscience, Cuprizone, Mice, 0302 clinical medicine, medicine, Animals, Demyelinating Disorder, Transcription factor, Cells, Cultured, Multiple sclerosis, Calcium-Binding Proteins, Microfilament Proteins, medicine.disease, Endoplasmic Reticulum Stress, Oligodendrocyte, Cell biology, Mice, Inbred C57BL, Disease Models, Animal, Oligodendroglia, 030104 developmental biology, medicine.anatomical_structure, Neurology, Animals, Newborn, Gene Expression Regulation, Astrocytes, Unfolded protein response, Microscopy, Electron, Scanning, Astrocytosis, 030217 neurology & neurosurgery, Transcription Factor CHOP, Demyelinating Diseases

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::273660b586018e2bcfd61acd05f8aaad
https://pubmed.ncbi.nlm.nih.gov/30511355