المؤلفون: R. Nuboer, Andrew T. Hattersley, Mijna Hadders-Algra, Annabelle S. Slingerland, G. J. Bruining

المساهمون: Faculteit Medische Wetenschappen/UMCG, Extremities Pain and Disability (EXPAND)

المصدر: Diabetologia, 49(11), 2559-2563. SPRINGER

مصطلحات موضوعية: Blood Glucose, Male, Kir6.2, Developmental Disabilities, Endocrinology, Diabetes and Metabolism, medicine.medical_treatment, DEND syndrome, THERAPY, Glibenclamide, MELLITUS, Epilepsy, Homeostasis, KCNJ11, ACTIVATING MUTATIONS, sulphonylurea, developmental delay, glibenclamide, glyburide, neonatal diabetes, KIR6.2 MUTATION, hormones, hormone substitutes, and hormone antagonists, medicine.drug, endocrine system, medicine.medical_specialty, medicine.drug_class, sulfonylurea, Motor Activity, neurological features, INSULIN-SECRETION, HYPOGLYCEMIA, Internal medicine, Diabetes mellitus, Diabetes Mellitus, ATP CHANNEL, Internal Medicine, medicine, Humans, Hypoglycemic Agents, Potassium Channels, Inwardly Rectifying, business.industry, V59M, Insulin, Infant, Newborn, nutritional and metabolic diseases, COGNITIVE FUNCTION, medicine.disease, Sulfonylurea, Endocrinology, Amino Acid Substitution, Mutation, Sulfonylurea receptor, SENSITIVE K+ CHANNELS, business, Follow-Up Studies

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36e092201de4e1369e740270abef545b
https://doi.org/10.1007/s00125-006-0407-0

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