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1دورية أكاديمية
المؤلفون: Veltrop RJA; Department of Biochemistry, CARIM, Maastricht University, Maastricht, The Netherlands. r.veltrop@maastrichtuniversity.nl.; Department of Cardiology, Division Heart and Lungs, University Medical Center Utrecht, Utrecht, the Netherlands. r.veltrop@maastrichtuniversity.nl.; Regenerative Medicine Utrecht, University Medical Center Utrecht, Utrecht, The Netherlands. r.veltrop@maastrichtuniversity.nl.; LMNAcardiac.org, LMNA Patient Organization, Soest, The Netherlands. r.veltrop@maastrichtuniversity.nl.; Striated Muscle Laminopathy Consortium, ENMC, Baarn, The Netherlands. r.veltrop@maastrichtuniversity.nl., Kukk MM; Department of Biochemistry, CARIM, Maastricht University, Maastricht, The Netherlands., Topouzidou K; Department of Biochemistry, CARIM, Maastricht University, Maastricht, The Netherlands., Didden L; Department of Biochemistry, CARIM, Maastricht University, Maastricht, The Netherlands., Muchir A; Centre de Recherche en Myologie, U974 SU-INSERM, 75013, Paris, France., van Steenbeek FG; Department of Clinical Sciences of Companion Animals, Faculty of Veterinary Medicine, Utrecht University, Utrecht, The Netherlands., Schurgers LJ; Department of Biochemistry, CARIM, Maastricht University, Maastricht, The Netherlands., Harakalova M; Department of Cardiology, Division Heart and Lungs, University Medical Center Utrecht, Utrecht, the Netherlands.; Regenerative Medicine Utrecht, University Medical Center Utrecht, Utrecht, The Netherlands.
المصدر: Cell communication and signaling : CCS [Cell Commun Signal] 2024 Mar 27; Vol. 22 (1), pp. 197. Date of Electronic Publication: 2024 Mar 27.
نوع المنشور: Journal Article; Review
بيانات الدورية: Publisher: BioMed Central Country of Publication: England NLM ID: 101170464 Publication Model: Electronic Cited Medium: Internet ISSN: 1478-811X (Electronic) Linking ISSN: 1478811X NLM ISO Abbreviation: Cell Commun Signal Subsets: MEDLINE
مواضيع طبية MeSH: Cardiomyopathies*/genetics , Cardiomyopathies*/metabolism , Heart Failure*/genetics, Humans ; Mechanotransduction, Cellular ; Lamin Type A/genetics ; Lamin Type A/metabolism ; Mutation/genetics ; Biophysics