يعرض 1 - 10 نتائج من 12 نتيجة بحث عن '"MESH : Mice, Inbred CFTR"', وقت الاستعلام: 1.28s تنقيح النتائج
  1. 1

    المساهمون: Défense innée et inflammation, Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut Pasteur [Paris], This work was supported by the French Association of Cystic Fibrosis 'Vaincre la Mucoviscidose', and the 'Legs Poix' Foundation., We are grateful to B. Polack and B. Toussaint (Université Joseph Fourier, Grenoble, France) for providing the CHA strain, and to G. Eberl (Institut Pasteur, Paris, France) for providing IL6−/− mice., Institut Pasteur [Paris] (IP)-Institut National de la Santé et de la Recherche Médicale (INSERM)

    المصدر: Biochimie
    Biochimie, Elsevier, 2014, 107, pp.95-104. ⟨10.1016/j.biochi.2014.08.018⟩
    Biochimie, 2014, 107, pp.95-104. ⟨10.1016/j.biochi.2014.08.018⟩

    مصطلحات موضوعية: Lung Diseases, Male, MAPK/ERK pathway, MESH: Macrophages, Alveolar, medicine.medical_treatment, p38 Mitogen-Activated Protein Kinases, MESH: Mice, Knockout, [SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunity, Biochemistry, MESH: Lung Diseases, [SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseases, Arachidonate 15-Lipoxygenase, MESH: Animals, Enzyme Inhibitors, Extracellular Signal-Regulated MAP Kinases, MESH: Extracellular Signal-Regulated MAP Kinases, Mice, Knockout, MESH: Immunoblotting, medicine.diagnostic_test, MESH: Pseudomonas Infections, General Medicine, respiratory system, 3. Good health, Survival Rate, medicine.anatomical_structure, Cytokine, MESH: Enzyme Inhibitors, Host-Pathogen Interactions, Pseudomonas aeruginosa, MESH: Pseudomonas aeruginosa, MESH: Mice, Inbred CFTR, Female, Tumor necrosis factor alpha, Acute lung infection, MESH: Cyclooxygenase 2, medicine.symptom, Bronchoalveolar Lavage Fluid, MESH: Survival Rate, MESH: Group IV Phospholipases A2, Immunoblotting, Inflammation, Arachidonic Acids, Lung injury, Biology, Cell Line, Proinflammatory cytokine, Species Specificity, MESH: Mice, Inbred C57BL, Macrophages, Alveolar, medicine, Animals, Mice, Inbred CFTR, MESH: Arachidonic Acids, MESH: Species Specificity, Pseudomonas Infections, Lung, Interleukin-6, MESH: Bronchoalveolar Lavage Fluid, Group IV Phospholipases A2, MESH: Host-Pathogen Interactions, Cytosolic phospholipase A2α (cPLA2α), MESH: Interleukin-6, [SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, Molecular biology, IL6, MESH: Male, MESH: Cell Line, Mice, Inbred C57BL, MESH: p38 Mitogen-Activated Protein Kinases, Bronchoalveolar lavage, Cyclooxygenase 2, Immunology, MESH: Arachidonate 15-Lipoxygenase, MESH: Female

  2. 2

    المساهمون: Service de pneumologie [CHU Cochin], Hôpital Cochin [AP-HP], Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP), Université Paris Descartes - Paris 5 (UPD5), Défense innée et inflammation, Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut Pasteur [Paris], Service d'ORL [Créteil], Centre Hospitalier Intercommunal de Créteil (CHIC), Institut Mondor de Recherche Biomédicale (IMRB), Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Paris-Est Créteil Val-de-Marne - Paris 12 (UPEC UP12)-IFR10, CHU Trousseau [APHP], Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Sorbonne Université (SU), Physiopathologie des maladies génétiques d'expression pédiatrique, Université Pierre et Marie Curie - Paris 6 (UPMC)-Institut National de la Santé et de la Recherche Médicale (INSERM), Service de Physiologie et Explorations Fonctionnelles [Cochin APHP], Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Hôpital Cochin [AP-HP], Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP), This work was funded by grants from Chancellerie des Universités de Paris (Legs Poix), Association Vaincre la Mucoviscidose and Association Cardif., The authors thank Sophie Danel-Beaucaire (Cochin Hospital, Paris, France) for help with immunohistochemical staining and Claire Danel (Bichat Hospital, Paris, France) for providing human CF tissues., Institut Pasteur [Paris] (IP)-Institut National de la Santé et de la Recherche Médicale (INSERM), Institut National de la Santé et de la Recherche Médicale (INSERM)-IFR10-Université Paris-Est Créteil Val-de-Marne - Paris 12 (UPEC UP12), Institut Pasteur [Paris]-Institut National de la Santé et de la Recherche Médicale (INSERM), Wu, Yongzheng

    المصدر: European Respiratory Journal
    European Respiratory Journal, European Respiratory Society, 2013, 42 (6), pp.1553-1562. ⟨10.1183/09031936.00164212⟩
    European Respiratory Journal, 2013, 42 (6), pp.1553-1562. ⟨10.1183/09031936.00164212⟩

    مصطلحات موضوعية: Vascular Endothelial Growth Factor A, Pathology, Cystic Fibrosis, Angiogenesis, medicine.medical_treatment, Cystic Fibrosis Transmembrane Conductance Regulator, MESH: Trachea / metabolism, Cystic fibrosis, Tyrosine-kinase inhibitor, Epithelium, chemistry.chemical_compound, Mice, 0302 clinical medicine, MESH: Cystic Fibrosis Transmembrane Conductance Regulator / physiology, [SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseases, MESH: Lung / blood supply, MESH: Animals, MESH: Cystic Fibrosis / physiopathology, Epidermal growth factor receptor, MESH: Cystic Fibrosis / genetics, Phosphorylation, RNA, Small Interfering, Lung, Aorta, Cells, Cultured, 0303 health sciences, biology, MESH: Gene Expression Regulation, MESH: Bronchi / metabolism, respiratory system, 3. Good health, Vascular endothelial growth factor, ErbB Receptors, Trachea, [SDV.MHEP.MI] Life Sciences [q-bio]/Human health and pathology/Infectious diseases, MESH: Mice, Inbred CFTR, MESH: Cells, Cultured, Pulmonary and Respiratory Medicine, congenital, hereditary, and neonatal diseases and abnormalities, medicine.medical_specialty, MESH: Mice, Transgenic, medicine.drug_class, MESH: Aorta / pathology, MESH: ErbB Receptors / metabolism, Bronchi, Mice, Transgenic, Cell Line, 03 medical and health sciences, medicine, Animals, Humans, Mice, Inbred CFTR, MESH: Mice, 030304 developmental biology, MESH: RNA, Small Interfering / metabolism, MESH: Humans, MESH: Phosphorylation, business.industry, Growth factor, MESH: Vascular Endothelial Growth Factor A / metabolism, medicine.disease, MESH: Cystic Fibrosis Transmembrane Conductance Regulator / genetics, [SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, MESH: Epithelium / metabolism, MESH: Cell Line, respiratory tract diseases, Transplantation, 030228 respiratory system, chemistry, Gene Expression Regulation, Cancer research, biology.protein, Respiratory epithelium, [SDV.MP.BAC] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, business

  3. 3

    المساهمون: Jacquot, Jacky, Biomatériaux et inflammation en site osseux - EA 4691 (BIOS), Université de Reims Champagne-Ardenne (URCA)-Institut National de la Santé et de la Recherche Médicale (INSERM)-SFR CAP Santé (Champagne-Ardenne Picardie Santé), Université de Reims Champagne-Ardenne (URCA)-Université de Picardie Jules Verne (UPJV)-Université de Reims Champagne-Ardenne (URCA)-Université de Picardie Jules Verne (UPJV), Os et articulations, Université Paris Diderot - Paris 7 (UPD7)-Institut National de la Santé et de la Recherche Médicale (INSERM)

    المصدر: American Journal of Pathology
    American Journal of Pathology, 2012, 180 (5), pp.2068-75. ⟨10.1016/j.ajpath.2012.01.039⟩
    American Journal of Pathology, American Society for Investigative Pathology, 2012, 180 (5), pp.2068-75. ⟨10.1016/j.ajpath.2012.01.039⟩

  4. 4

    المساهمون: Lille Inflammation Research International Center - U 995 [LIRIC], Lille Inflammation Research International Center - U 995 (LIRIC), Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), This study was supported by the French Cystic Fibrosis Association Vaincre la Mucoviscidose (JLD, FC0203, and IC0806)., We thank the microbiology department of the Centre de Biologie-Pathologie Pierre-Marie Degand (Lille Hospital) for access to the ABI Prism 7700, MH Gevaert and RM Siminski (Service Commun-Morphologie cellulaire, Université Lille 2) for slides, J Bara (Inserm UMR S938, Paris, France) for the 45M1 antibody, J Ryerse (Department of Pathology, Saint Louis, MO) for the anti-CCSP (R42AP) antibody, and MR Parsek and BR Borlee (Seattle, WA) for the PAO1 strain.

    المصدر: Histochemistry and Cell Biology
    Histochemistry and Cell Biology, Springer Verlag, 2011, 136 (6), pp.699-708. ⟨10.1007/s00418-011-0872-5⟩
    Histochemistry and Cell Biology, 2011, 136 (6), pp.699-708. ⟨10.1007/s00418-011-0872-5⟩

    مصطلحات موضوعية: Pathology, Mucus plug, [SDV]Life Sciences [q-bio], Peptide, medicine.disease_cause, MESH: Mice, Knockout, Cystic fibrosis, Animals, Antibodies, Monoclonal, Antibody Specificity, Female, Gene Expression Regulation, Immunohistochemistry, Mammary Neoplasms, Animal, Mammary Tumor Virus, Mouse, Mice, Inbred C57BL, Inbred CFTR, Knockout, Mucin-5B, Real-Time Polymerase Chain Reaction, ras Proteins, MESH: Antibodies, Monoclonal/genetics, Gel-forming mucin, Breast cancer, 0302 clinical medicine, MESH: Animals, MESH: Mucin-5B/metabolism, Muc5b, Mice, Knockout, chemistry.chemical_classification, 0303 health sciences, biology, MESH: Real-Time Polymerase Chain Reaction, MESH: Gene Expression Regulation, Antibodies, Monoclonal, respiratory system, 3. Good health, MESH: Mammary Neoplasms, Animal/metabolism, Medical Laboratory Technology, 030220 oncology & carcinogenesis, MESH: Mice, Inbred CFTR, MESH: Mammary Tumor Virus, Mouse, Antibody, medicine.medical_specialty, Histology, Mammary Neoplasms, Animal, 03 medical and health sciences, MESH: Mice, Inbred C57BL, MESH: Antibody Specificity/genetics, medicine, Mice, Inbred CFTR, MESH: Mucin-5B/genetics, MESH: Mice, Molecular Biology, 030304 developmental biology, MESH: Antibodies, Monoclonal/metabolism, Pseudomonas aeruginosa, MESH: ras Proteins/metabolism, Mucin, Cancer, MESH: Immunohistochemistry, Cell Biology, MESH: Mammary Neoplasms, Animal/physiopathology, medicine.disease, Mucus, Molecular biology, Mice, Inbred C57BL, Mammary Tumor Virus, Mouse, chemistry, Polyclonal antibodies, biology.protein, MESH: Female

    وصف الملف: application/octet-stream

  5. 5

    المساهمون: Défense innée et inflammation, Institut Pasteur [Paris] (IP)-Institut National de la Santé et de la Recherche Médicale (INSERM), Mucoviscidose et bronchopathies chroniques : biopathologie et phénotypes cliniques (EA 2511), Hôpital Cochin [AP-HP], Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Université Paris Descartes - Paris 5 (UPD5), Centre de recherche Croissance et signalisation (UMR_S 845), Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Department of Cell Biology [Rotterdam, Pays-Bas], Erasmus University Medical Center [Rotterdam] (Erasmus MC), Service de Pneumologie et soins intensifs respiratoires [Hôpital Européen Georges Pompidou, Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Hôpital Européen Georges Pompidou [APHP] (HEGP), Assistance publique - Hôpitaux de Paris (AP-HP) (AP-HP)-Hôpitaux Universitaires Paris Ouest - Hôpitaux Universitaires Île de France Ouest (HUPO)-Hôpitaux Universitaires Paris Ouest - Hôpitaux Universitaires Île de France Ouest (HUPO), Department of Biochemistry and Molecular Biology [Tokyo], Graduate School of Science [Tokyo], The University of Tokyo (UTokyo)-The University of Tokyo (UTokyo), This work was supported in part by a Contrat de Recherche with the French Associations 'Vaincre la Mucoviscidose' and DARRI, Institut Pasteur. F. Dif was supported by 'Vaincre la Mucoviscidose'. J. Aarbiou and R. Buijs-Offerman (Erasmus MC, Cell Biology Dept, Rotterdam, The Netherlands) were supported by grants STW 6565 and EU FP6IMPROVED PRECISION LSHB-CT-2004-005213., The authors would like to thank V. Balloy (Unite de Défense Innée et Inflammation/Unité INSERM U. 874, Institut Pasteur, Paris, France) for her helpful assistance for animal instillations. We are grateful to M. Huerre (Unité de Recherche et d'Expertise Histotechnologie et Pathologie, Institut Pasteur) for expert advice and criticisms and P. Ave and S. Bergère (Unité de Recherche et d'Expertise Histotechnologie et Pathologie, Institut Pasteur) for help on histological analyses. M. Wilke (Erasmus MC, Biochemistry Dept, Rotterdam, The Netherlands) and R. Buijs-Offerman (Erasmus MC, Cell Biology Dept) are acknowledged for assistance in preparing the lung tissues and BALF samples., Wu, Yongzheng, Cell biology, Institut National de la Santé et de la Recherche Médicale (INSERM)-Institut Pasteur [Paris], Centre National de la Recherche Scientifique (CNRS)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Paris Descartes - Paris 5 (UPD5), Institut Pasteur [Paris]-Institut National de la Santé et de la Recherche Médicale (INSERM)

    المصدر: European Respiratory Journal
    European Respiratory Journal, 2010, 36 (5), pp.1120-1130. ⟨10.1183/09031936.00183409⟩
    European Respiratory Journal, 36(5), 1120-1130. European Respiratory Society
    European Respiratory Journal, European Respiratory Society, 2010, 36 (5), pp.1120-1130. ⟨10.1183/09031936.00183409⟩

    مصطلحات موضوعية: Lipopolysaccharides, Cystic Fibrosis Transmembrane Conductance Regulator, Phospholipase, Mucin 5AC, MESH: Lipopolysaccharides / pharmacology, Cystic fibrosis, [SDV.IMM.II]Life Sciences [q-bio]/Immunology/Innate immunity, cystic fibrosis, chemistry.chemical_compound, Mice, 0302 clinical medicine, Cytosol, [SDV.MHEP.MI]Life Sciences [q-bio]/Human health and pathology/Infectious diseases, MESH: RNA, Small Interfering, MESH: Bronchi / metabolism, MESH: Animals, MESH: Respiratory Mucosa / metabolism, RNA, Small Interfering, 0303 health sciences, MESH: Chlorides / metabolism, lipopolysaccharide, respiratory system, mucusphospholipase A2, Cystic fibrosis transmembrane conductance regulator, MESH: Group IV Phospholipases A2 / genetics, 3. Good health, Arachidonic acid, MESH: Cystic Fibrosis Transmembrane Conductance Regulator / metabolism, [SDV.MHEP.MI] Life Sciences [q-bio]/Human health and pathology/Infectious diseases, MESH: Mice, Inbred CFTR, medicine.symptom, Pulmonary and Respiratory Medicine, medicine.medical_specialty, MESH: Bronchi / cytology, Bronchial mucus, Inflammation, Bronchi, MESH: Group IV Phospholipases A2 / metabolism, MESH: Mucin 5AC / genetics, Respiratory Mucosa, Biology, Cell Line, 03 medical and health sciences, Phospholipase A2, Chlorides, MESH: Mice, Inbred C57BL, Internal medicine, medicine, MESH: Arachidonic Acid / metabolism, MESH: Mucus / metabolism, Animals, Humans, Mice, Inbred CFTR, [SDV.IMM.II] Life Sciences [q-bio]/Immunology/Innate immunity, MESH: Mice, 030304 developmental biology, MESH: Humans, Group IV Phospholipases A2, MESH: Respiratory Mucosa / cytology, MESH: Mucin 5AC / metabolism, medicine.disease, MUC5AC, Mucus, [SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, respiratory tract diseases, MESH: Cell Line, Mice, Inbred C57BL, Disease Models, Animal, Endocrinology, 030228 respiratory system, chemistry, biology.protein, MESH: Cystic Fibrosis Transmembrane Conductance Regulator / genetics, MESH: Cytosol / metabolism, [SDV.MP.BAC] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology, MESH: Disease Models, Animal

  6. 6

    المساهمون: Evolution des régulations endocriniennes (ERE), Muséum national d'Histoire naturelle (MNHN)-Centre National de la Recherche Scientifique (CNRS), Laboratoire de chimie et biochimie des substances naturelles, Centre National de la Recherche Scientifique (CNRS)-Muséum national d'Histoire naturelle (MNHN)

    المصدر: The Journal of Gene Medicine
    The Journal of Gene Medicine, Wiley, 2006, 8 (1), pp.82-9. ⟨10.1002/jgm.831⟩
    HAL
    The Journal of Gene Medicine, Wiley, 2006, 8(1), pp.82-89

  7. 7

    المساهمون: Evolution des régulations endocriniennes (ERE), Muséum national d'Histoire naturelle (MNHN)-Centre National de la Recherche Scientifique (CNRS), Adaptations et évolution des systèmes ostéomusculaires (AESO), Muséum national d'Histoire naturelle (MNHN)-Université Pierre et Marie Curie - Paris 6 (UPMC)-Centre National de la Recherche Scientifique (CNRS), Laboratoire d'Ethologie Expérimentale et Comparée (LEEC), Université Sorbonne Paris Cité (USPC)-Université Paris 13 (UP13), Os et articulations, Université Paris Diderot - Paris 7 (UPD7)-Institut National de la Santé et de la Recherche Médicale (INSERM), Université Paris 13 (UP13)-Université Sorbonne Paris Cité (USPC)

    المصدر: BONE
    BONE, Elsevier, 2004, 35 (3), pp.595-603. ⟨10.1016/j.bone.2004.05.021⟩
    BONE, 2004, 35 (3), pp.595-603. ⟨10.1016/j.bone.2004.05.021⟩

    مصطلحات موضوعية: Pathology, Bone density, Cystic Fibrosis, Physiology, [SDV.NEU.NB]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC]/Neurobiology, Endocrinology, Diabetes and Metabolism, Osteoporosis, Cystic Fibrosis Transmembrane Conductance Regulator, MESH: Mice, Knockout, Cystic fibrosis, Mice, 0302 clinical medicine, Bone Density, MESH: Animals, MESH: Gene Silencing, MESH: Bone Density, [SDV.BDD]Life Sciences [q-bio]/Development Biology, ComputingMilieux_MISCELLANEOUS, MESH: Cystic Fibrosis Transmembrane Conductance Regulator, Bone mineral, Mice, Knockout, 0303 health sciences, biology, Cystic fibrosis transmembrane conductance regulator, medicine.anatomical_structure, MESH: Mice, Inbred CFTR, [SDV.NEU]Life Sciences [q-bio]/Neurons and Cognition [q-bio.NC], MESH: Osteoporosis, congenital, hereditary, and neonatal diseases and abnormalities, medicine.medical_specialty, Histology, MESH: Cystic Fibrosis, Bone resorption, [SDV.BDLR.RS]Life Sciences [q-bio]/Reproductive Biology/Sexual reproduction, 03 medical and health sciences, Internal medicine, medicine, Animals, Mice, Inbred CFTR, Gene Silencing, MESH: Mice, 030304 developmental biology, business.industry, [SDV.BDLR]Life Sciences [q-bio]/Reproductive Biology, [SDV.BDD.MOR]Life Sciences [q-bio]/Development Biology/Morphogenesis, medicine.disease, Osteopenia, Bone Diseases, Metabolic, [SDV.BDD.EO]Life Sciences [q-bio]/Development Biology/Embryology and Organogenesis, Endocrinology, 030228 respiratory system, biology.protein, Cortical bone, business, MESH: Bone Diseases, Metabolic

  8. 8

    المساهمون: Physiologie cellulaire et moléculaire des systèmes intégrés (PCMSI), Université Nice Sophia Antipolis (... - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-Centre National de la Recherche Scientifique (CNRS)

    المصدر: Journal of Membrane Biology
    Journal of Membrane Biology, Springer Verlag (Germany), 2004, 198 (3), pp.113-24. ⟨10.1007/s00232-004-0665-z⟩

  9. 9

    المساهمون: Physiologie cellulaire et moléculaire des systèmes intégrés (PCMSI), Université Nice Sophia Antipolis (... - 2019) (UNS), COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-COMUE Université Côte d'Azur (2015-2019) (COMUE UCA)-Centre National de la Recherche Scientifique (CNRS), Institut des Sciences sociales du Politique (ISP), École normale supérieure - Cachan (ENS Cachan)-Université Paris Nanterre (UPN)-Centre National de la Recherche Scientifique (CNRS), Université Nice Sophia Antipolis (1965 - 2019) (UNS)

    المصدر: AJP Renal Physiology
    AJP Renal Physiology, American Physiological Society, 2003, 284 (4), pp.F812-28. ⟨10.1152/ajprenal.00238.2002⟩
    AJP Renal Physiology, 2003, 284 (4), pp.F812-28. ⟨10.1152/ajprenal.00238.2002⟩
    Scopus-Elsevier

  10. 10

    المساهمون: Défense innée et inflammation, Institut Pasteur [Paris]-Institut National de la Santé et de la Recherche Médicale ( INSERM ), Immunothérapie, Institut Pasteur [Paris], Centre de recherche Croissance et signalisation ( UMR_S 845 ), Université Paris Descartes - Paris 5 ( UPD5 ) -Institut National de la Santé et de la Recherche Médicale ( INSERM ) -Centre National de la Recherche Scientifique ( CNRS ), Department of Biochemistry and Molecular Biology, The University of Tokyo, this work was supported by the French association 'Vaincre la Mucoviscidose' and the Foundation Legs Poix, Paris., Institut Pasteur [Paris]-Institut National de la Santé et de la Recherche Médicale (INSERM), Centre de recherche Croissance et signalisation (UMR_S 845), Centre National de la Recherche Scientifique (CNRS)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Paris Descartes - Paris 5 (UPD5), Department of Biochemistry and Molecular Biology [Tokyo], Graduate School of Science [Tokyo], The University of Tokyo (UTokyo)-The University of Tokyo (UTokyo), Institut Pasteur [Paris] (IP)-Institut National de la Santé et de la Recherche Médicale (INSERM), Institut Pasteur [Paris] (IP), Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), BMC, Ed.

    المصدر: Respiratory Research
    Respiratory Research, BioMed Central, 2010, 11 (1), pp.49. 〈10.1186/1465-9921-11-49〉
    Respiratory Research, BioMed Central, 2010, 11 (1), pp.49. ⟨10.1186/1465-9921-11-49⟩
    Respiratory Research, 2010, 11 (1), pp.49. ⟨10.1186/1465-9921-11-49⟩
    Respiratory Research, Vol 11, Iss 1, p 49 (2010)

    مصطلحات موضوعية: Chemokine CXCL2, Pharmacology, Cystic fibrosis, Mice, 0302 clinical medicine, MESH : Arachidonic Acids, MESH: Animals, Enzyme Inhibitors, Lung, 0303 health sciences, Arachidonic Acid, MESH: Dinoprostone, MESH : Pseudomonas aeruginosa, MESH: Bronchoconstriction, Neutrophil Infiltration, MESH: Mice, Inbred CFTR, Arachidonic acid, Bronchoalveolar Lavage Fluid, Pulmonary and Respiratory Medicine, MESH : Leukotrienes, MESH: Group IV Phospholipases A2, Dinoprostone, 03 medical and health sciences, MESH: Cyclooxygenase Inhibitors, MESH : Cystic Fibrosis, MESH: Arachidonic Acids, Cyclooxygenase Inhibitors, Aspirin, Group IV Phospholipases A2, MESH : Pneumonia, MESH : Administration, Intranasal, lcsh:Diseases of the respiratory system, Pneumonia, medicine.disease, MESH : Disease Models, Animal, respiratory tract diseases, 030228 respiratory system, chemistry, Immunology, MESH : Bronchoconstriction, MESH : Aspirin, MESH: Disease Models, Animal, MESH: Lipopolysaccharides, [ SDV.MHEP.PSR ] Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract, Lipopolysaccharides, Time Factors, Cystic Fibrosis, MESH : Lipopolysaccharides, Phospholipase, medicine.disease_cause, MESH: Mice, Knockout, [SDV.MHEP.PSR]Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract, MESH : Mice, Inbred CFTR, MESH : Group IV Phospholipases A2, chemistry.chemical_compound, MESH: Administration, Intranasal, Prostaglandin E2, Mice, Knockout, MESH : Bronchoalveolar Lavage Fluid, MESH : Cyclooxygenase Inhibitors, respiratory system, MESH : Chemokine CXCL2, MESH : Neutrophil Infiltration, MESH: Enzyme Inhibitors, Pseudomonas aeruginosa, MESH: Pseudomonas aeruginosa, Bronchoconstriction, medicine.symptom, MESH : Time Factors, medicine.drug, Leukotrienes, MESH: Pneumonia, MESH : Dinoprostone, MESH: Cystic Fibrosis, MESH : Lung, Arachidonic Acids, Biology, MESH : Arachidonic Acid, MESH : Mice, medicine, Animals, Mice, Inbred CFTR, MESH: Chemokine CXCL2, Plethysmograph, MESH: Lung, Pediatrics, Perinatology, and Child Health, MESH: Aspirin, MESH: Mice, Administration, Intranasal, 030304 developmental biology, lcsh:RC705-779, MESH : Enzyme Inhibitors, MESH: Leukotrienes, MESH: Bronchoalveolar Lavage Fluid, business.industry, Research, MESH: Time Factors, MESH: Arachidonic Acid, Disease Models, Animal, MESH: Neutrophil Infiltration, Pediatrics, Perinatology and Child Health, [SDV.MHEP.PSR] Life Sciences [q-bio]/Human health and pathology/Pulmonology and respiratory tract, MESH : Mice, Knockout, MESH : Animals, Airway, business

    وصف الملف: application/pdf