نتائج البحث - "Maj-Linda B. Selenica"

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دورية أكاديمية

TDP-43 mediated blood-brain barrier permeability and leukocyte infiltration promote neurodegeneration in a low-grade systemic inflammation mouse model

المؤلفون: Frank Zamudio, Anjanet R. Loon, Shayna Smeltzer, Khawla Benyamine, Nanda K. Navalpur Shanmugam, Nicholas J. F. Stewart, Daniel C. Lee, Kevin Nash, Maj-Linda B. Selenica

المصدر: Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-16 (2020)

مصطلحات موضوعية: TDP-43, Synaptic dysfunction, Microglial activation, Astrocytosis, Systemic inflammation, Neurovascular unit, Neurology. Diseases of the nervous system, RC346-429

وصف الملف: electronic resource

Relation: http://link.springer.com/article/10.1186/s12974-020-01952-9; https://doaj.org/toc/1742-2094

URL الوصول: https://doaj.org/article/e33a410a6c39471d91bd94eaabb92019

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2

دورية أكاديمية

Myeloid Arginase 1 Insufficiency Exacerbates Amyloid-β Associated Neurodegenerative Pathways and Glial Signatures in a Mouse Model of Alzheimer’s Disease: A Targeted Transcriptome Analysis

المؤلفون: Chao Ma, Jerry B. Hunt, Andrii Kovalenko, Huimin Liang, Maj-Linda B. Selenica, Michael B. Orr, Bei Zhang, John C. Gensel, David J. Feola, Marcia N. Gordon, Dave Morgan, Paula C. Bickford, Daniel C. Lee

المصدر: Frontiers in Immunology, Vol 12 (2021)

مصطلحات موضوعية: arginine metabolism, APP Tg2576, amyloidosis, neurodegeneration, neuroinflammation, infiltrating macrophage, Immunologic diseases. Allergy, RC581-607

وصف الملف: electronic resource

Relation: https://www.frontiersin.org/articles/10.3389/fimmu.2021.628156/full; https://doaj.org/toc/1664-3224

URL الوصول: https://doaj.org/article/c955b4c995af4d19a154a2fbbbf94d8f

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3

دورية أكاديمية

Spermidine/spermine-N 1-acetyltransferase ablation impacts tauopathy-induced polyamine stress response

المصدر: Alzheimer’s Research & Therapy, Vol 11, Iss 1, Pp 1-24 (2019)

مصطلحات موضوعية: Tau, Polyamine dysregulation, Hippocampus, Alzheimer’s disease, Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571, Neurology. Diseases of the nervous system, RC346-429

وصف الملف: electronic resource

Relation: http://link.springer.com/article/10.1186/s13195-019-0507-y; https://doaj.org/toc/1758-9193

URL الوصول: https://doaj.org/article/f6fdfce93d6549e184299e4d7d512e9b

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4

دورية أكاديمية

Arginase 1 Insufficiency Precipitates Amyloid-β Deposition and Hastens Behavioral Impairment in a Mouse Model of Amyloidosis

المصدر: Frontiers in Immunology, Vol 11 (2021)

مصطلحات موضوعية: Alzheimer’s disease, neuroinflammation, arginine metabolism, macrophage, microglia, phagocytosis, Immunologic diseases. Allergy, RC581-607

وصف الملف: electronic resource

Relation: https://www.frontiersin.org/articles/10.3389/fimmu.2020.582998/full; https://doaj.org/toc/1664-3224

URL الوصول: https://doaj.org/article/eb73ef7b7394472091f6f6e6c914d923

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5

دورية أكاديمية

CCL2 Overexpression in the Brain Promotes Glial Activation and Accelerates Tau Pathology in a Mouse Model of Tauopathy

المؤلفون: Aurelie Joly-Amado, Jordan Hunter, Zainuddin Quadri, Frank Zamudio, Patricia V. Rocha-Rangel, Deanna Chan, Anisha Kesarwani, Kevin Nash, Daniel C. Lee, Dave Morgan, Marcia N. Gordon, Maj-Linda B. Selenica

المصدر: Frontiers in Immunology, Vol 11 (2020)

مصطلحات موضوعية: monocyte chemoattractant protein-1 (MCP-1), neuroinflammation, Alzheimer's disease, tau, Aβ, gene therapy, Immunologic diseases. Allergy, RC581-607

وصف الملف: electronic resource

Relation: https://www.frontiersin.org/article/10.3389/fimmu.2020.00997/full; https://doaj.org/toc/1664-3224

URL الوصول: https://doaj.org/article/9b7b4496d3e9471cbe9ac95aad49ef5e

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TDP-43 mediated blood-brain barrier permeability and leukocyte infiltration promote neurodegeneration in a low-grade systemic inflammation mouse model

المؤلفون: Nanda K. Navalpur Shanmugam, Frank Zamudio, Kevin Nash, Khawla Benyamine, Anjanet R. Loon, Shayna Smeltzer, Daniel C. Lee, Nicholas J. F. Stewart, Maj-Linda B. Selenica

المصدر: Journal of Neuroinflammation, Vol 17, Iss 1, Pp 1-16 (2020)
Journal of Neuroinflammation

مصطلحات موضوعية: Lipopolysaccharides, Male, Pathology, medicine.medical_specialty, Synaptic dysfunction, Lipopolysaccharide, TDP-43, CD3, Immunology, Blood–brain barrier, Systemic inflammation, lcsh:RC346-429, Capillary Permeability, Mice, Cellular and Molecular Neuroscience, chemistry.chemical_compound, Leukocytes, medicine, Animals, Humans, Microglial activation, Amyotrophic lateral sclerosis, lcsh:Neurology. Diseases of the nervous system, biology, business.industry, Research, General Neuroscience, Neurodegeneration, Neurodegenerative Diseases, medicine.disease, Systemic Inflammatory Response Syndrome, DNA-Binding Proteins, Disease Models, Animal, Astrocytosis, medicine.anatomical_structure, Neurology, chemistry, Blood-Brain Barrier, biology.protein, Neurovascular unit, Female, Pericyte, medicine.symptom, business, Infiltration (medical)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e1ba8bc0a0f870d9b891c5d971389ec7
http://link.springer.com/article/10.1186/s12974-020-01952-9

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7

CHCHD10‐regulated OPA1‐mitofilin complex mediates TDP‐43‐induced mitochondrial phenotypes associated with frontotemporal dementia

المؤلفون: Maj-Linda B. Selenica, Ann Chacko, David E. Kang, Tian Liu, Jung A. Woo, Patrick LePochat, Mohammed Zaheen Bukhari, Yan Yan, Xingyu Zhao, Sara Cazzaro Buosi, Peter Kotsiviras

المصدر: FASEB J

مصطلحات موضوعية: 0301 basic medicine, Genetically modified mouse, Muscle Proteins, Mice, Transgenic, Biology, medicine.disease_cause, Mitochondrial Dynamics, Biochemistry, Article, Cell Line, GTP Phosphohydrolases, Mitochondrial Proteins, Mice, 03 medical and health sciences, 0302 clinical medicine, Cell Line, Tumor, mental disorders, Genetics, medicine, Animals, Humans, Molecular Biology, Gene, Mutation, Gene knockdown, Amyotrophic Lateral Sclerosis, Wild type, nutritional and metabolic diseases, Transfection, Phenotype, Mitochondria, nervous system diseases, Cell biology, DNA-Binding Proteins, Mice, Inbred C57BL, HEK293 Cells, 030104 developmental biology, mitochondrial fusion, Frontotemporal Dementia, Mitochondrial Membranes, NIH 3T3 Cells, 030217 neurology & neurosurgery, HeLa Cells, Biotechnology

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67d85cb405696cfdaa178f065f65624c
https://doi.org/10.1096/fj.201903133rr

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8

Spermidine/spermine-N 1-acetyltransferase ablation impacts tauopathy-induced polyamine stress response

المصدر: Alzheimer’s Research & Therapy, Vol 11, Iss 1, Pp 1-24 (2019)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::66f7e770da9aa071b4410d08c755536a
http://link.springer.com/article/10.1186/s13195-019-0507-y

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9

Aberrant AZIN2 and polyamine metabolism precipitates tau neuropathology

المصدر: J Clin Invest

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c33b1bc68c081762982c122956567e7b
https://doi.org/10.1172/jci126299

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10

Leveraging amino acid sensors as therapeutic targets for tauopathies and related dementias

المؤلفون: Daniel Sejer Pedersen, Mani Kallupurackal, Andrii Kovalenko, Huimin Liang, Shalini Pandey, Hans Bräuner-Osborne, Dave Morgan, Chao Ma, Paula C. Bickford, John Ivan T. Calahatian, Maj-Linda B. Selenica, Daniel C. Lee, Danielle M. Blazier, Trond Ulven, Jerry B. Hunt, Camilla Michalski, Margaret Fahnestock, Leslie A. Sandusky-Beltran

المصدر: Alzheimer's & Dementia. 16

مصطلحات موضوعية: chemistry.chemical_classification, Psychiatry and Mental health, Cellular and Molecular Neuroscience, Developmental Neuroscience, chemistry, Epidemiology, Health Policy, Neurology (clinical), Computational biology, Geriatrics and Gerontology, Amino acid

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::efcd48e4273794fe6e0d58c321ff2ae7
https://doi.org/10.1002/alz.043859

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