المؤلفون: David Novo, Nikki Heath, Louise Mitchell, Giuseppina Caligiuri, Amanda MacFarlane, Dide Reijmer, Laura Charlton, John Knight, Monika Calka, Ewan McGhee, Emmanuel Dornier, David Sumpton, Susan Mason, Arnaud Echard, Kerstin Klinkert, Judith Secklehner, Flore Kruiswijk, Karen Vousden, Iain R. Macpherson, Karen Blyth, Peter Bailey, Huabing Yin, Leo M. Carlin, Jennifer Morton, Sara Zanivan, Jim C. Norman

المصدر: Nature Communications, Vol 9, Iss 1, Pp 1-17 (2018)

مصطلحات موضوعية: Science

وصف الملف: electronic resource

Relation: https://doaj.org/toc/2041-1723

URL الوصول: https://doaj.org/article/5887f05591104f9e9068f2413b7885fd

المؤلفون: Susan M. Mason, John R. P. Knight, Arnaud Echard, Giuseppina Caligiuri, Dide Reijmer, Monika Calka, Flore Kruiswijk, David Sumpton, David Novo, Karen Blyth, Iain R. Macpherson, Karen H. Vousden, Peter Bailey, Sara Zanivan, Kerstin Klinkert, Jim C. Norman, Judith Secklehner, Amanda MacFarlane, Nikki Heath, Huabing Yin, Jennifer P. Morton, Leo M. Carlin, Louise Mitchell, Ewan J. McGhee, Laura Charlton, Emmanuel Dornier

المساهمون: The Beatson Institute for Cancer Research, University of Glasgow, Institute of Cancer Sciences [Glasgow, UK] (CR-UK Beatson Institute), James Watt School of Engineering [Univ Glasgow], Trafic membranaire et Division cellulaire - Membrane Traffic and Cell Division, Institut Pasteur [Paris]-Centre National de la Recherche Scientifique (CNRS), Inflammation, Repair and Development, National Heart & Lung Institute, Imperial College London, Wolfson Wohl Cancer Research Centre [Glasgow, UK], University of Glasgow-Institute of Cancer Sciences [Glasgow, UK], This work was funded by Cancer Research UK and Breast Cancer Now. We acknowledge the Cancer Research UK Glasgow Centre (C596/A18076) and the BSU facilities at the Cancer Research UK Beatson Institute (C596/A17196)., Institut Pasteur [Paris] (IP)-Centre National de la Recherche Scientifique (CNRS)

المصدر: Nature Communications, Vol 9, Iss 1, Pp 1-17 (2018)
Nature Communications
Nature Communications, Nature Publishing Group, 2018, 9 (1), pp.5069. ⟨10.1038/s41467-018-07339-y⟩
Nature Communications, 2018, 9 (1), pp.5069. ⟨10.1038/s41467-018-07339-y⟩
Novo, D, Heath, N, Mitchell, L, Caligiuri, G, MacFarlane, A, Reijmer, D, Charlton, L, Knight, J, Calka, M, McGhee, E, Dornier, E, Sumpton, D, Mason, S, Echard, A, Klinkert, K, Secklehner, J, Kruiswijk, F, Vousden, K, Macpherson, I R, Blyth, K, Bailey, P, Yin, H, Carlin, L M, Morton, J, Zanivan, S & Norman, J C 2018, ' Mutant p53s generate pro-invasive niches by influencing exosome podocalyxin levels ', Nature Communications, vol. 9, no. 1, 5069 . https://doi.org/10.1038/s41467-018-07339-y

مصطلحات موضوعية: 0301 basic medicine, [SDV]Life Sciences [q-bio], Mutant, General Physics and Astronomy, Exosomes, Microscopy, Atomic Force, Extracellular matrix, Mice, chemistry.chemical_compound, MESH: Animals, MESH: Sialoglycoproteins, MESH: Tumor Suppressor Protein p53, lcsh:Science, MESH: Microscopy, Atomic Force, Multidisciplinary, Manchester Cancer Research Centre, biology, Chemistry, Cell biology, Podocalyxin, Female, MESH: Sialomucins, MESH: Mutation, Sialomucins, Endosome, Sialoglycoproteins, Science, Integrin, Mice, Nude, [SDV.CAN]Life Sciences [q-bio]/Cancer, [SDV.BC]Life Sciences [q-bio]/Cellular Biology, Exosome, Article, General Biochemistry, Genetics and Molecular Biology, Cell Line, 03 medical and health sciences, MESH: Mice, Nude, Animals, Humans, MESH: Exosomes, MESH: Mice, Diacylglycerol kinase, MESH: Humans, ResearchInstitutes_Networks_Beacons/mcrc, General Chemistry, Microvesicles, MESH: Cell Line, MESH: rab GTP-Binding Proteins, 030104 developmental biology, rab GTP-Binding Proteins, Mutation, biology.protein, lcsh:Q, Tumor Suppressor Protein p53, MESH: Female

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4508e22176087bbcbdf320820985074e
http://link.springer.com/article/10.1038/s41467-018-07339-y