المؤلفون: Khaoula Ben-Farhat, Monia Khemiri, Zohra Fitouri, Sihem Barsaoui, Najla Mekki, Meriem Ben-Ali, Selim Abdelmoula, Mohamed-Neji Guediche, M. R. Barbouche, Samir Boukthir, Saber Hamami, Imen Ben-Mustapha, Amel Ben-Chehida, Jalel Chemli, Karen Rouault, Beya Larguèche

المساهمون: Université de Tunis El Manar (UTM), Laboratoire de Transmission, Contrôle et Immunobiologie des Infections - Laboratory of Transmission, Control and Immunobiology of Infection (LR11IPT02), Institut Pasteur de Tunis, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP), Faculté de Médecine de Tunis, Génétique, génomique fonctionnelle et biotechnologies (UMR 1078) (GGB), Institut Brestois Santé Agro Matière (IBSAM), Université de Brest (UBO)-Université de Brest (UBO)-EFS-Institut National de la Santé et de la Recherche Médicale (INSERM), Département pédiatrique [Hôpital Fattouma Bourguiba - Monastir], CHU Fattouma Bourguiba [Monastir] (HFB), Department of Pediatrics [Tunis], Hôpital La Rabta [Tunis], Béchir Hamza Children's Hospital, Hôpital Universitaire Sahloul (CHU Sahloul)

المصدر: Journal of Clinical Immunology
Journal of Clinical Immunology, Springer Verlag, 2016, 36 (6), pp.547-554. ⟨10.1007/s10875-016-0299-9⟩

مصطلحات موضوعية: Male, 0301 basic medicine, Neutrophils, [SDV]Life Sciences [q-bio], DNA Mutational Analysis, Granulomatous Disease, Chronic, medicine.disease_cause, Severity of Illness Index, Consanguinity, 0302 clinical medicine, Chronic granulomatous disease, MESH: Child, Immunology and Allergy, MESH: NADPH Oxidases/genetics, MESH: DNA Mutational Analysis, Child, MESH: Neutrophils/metabolism, MESH: Genetic Association Studies, Genetics, Mutation, MESH: Infant, Founder Effect, 3. Good health, Phenotype, Child, Preschool, 030220 oncology & carcinogenesis, Female, MESH: Tunisia, NCF2 gene, MESH: Alleles, MESH: Enzyme Activation, Tunisia, Immunology, Biology, MESH: Phenotype, 03 medical and health sciences, MESH: Genetic Predisposition to Disease, MESH: Severity of Illness Index, MESH: Founder Effect, medicine, Humans, Genetic Predisposition to Disease, Allele, Gene, Alleles, Genetic Association Studies, MESH: Consanguinity, MESH: Granulomatous Disease, Chronic/diagnosis, MESH: Humans, MESH: Granulomatous Disease, Chronic/genetics, MESH: Child, Preschool, Haplotype, Infant, NADPH Oxidases, MESH: Granulomatous Disease, Chronic/metabolism, MESH: Haplotypes, medicine.disease, MESH: Male, Enzyme Activation, 030104 developmental biology, Haplotypes, Genetic marker, MESH: Neutrophils/immunology, MESH: Mutation, MESH: NADPH Oxidases/metabolism, MESH: Female, Founder effect

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d93add6bc9843dc959ccce1265808deb
https://doi.org/10.1007/s10875-016-0299-9

المؤلفون: Carlsen, S., Nandakumar, Kutty Selva, 1965, Backlund, J., Holmberg, J., Hultqvist, M., Vestberg, M., Holmdahl, R.

المصدر: Arthritis and Rheumatism. 58(7):2000-2011

مصطلحات موضوعية: Animals, Antibodies/blood, Arthritis/blood/chemically induced/*immunology, Cartilage Oligomeric Matrix Protein, Disease Models, Animal, Extracellular Matrix Proteins/adverse effects/blood/*immunology, Female, Glycoproteins/adverse effects/blood/*immunology, Humans, Major Histocompatibility Complex/*genetics, Matrilin Proteins, Mice, NADPH Oxidases/*metabolism, Rats

وصف الملف: print

URL الوصول: https://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-48841

المؤلفون: Carine Duval, Johan Auwerx, Christophe Dardenne, Jean Loup Lemesre, Alexis Valentin, Hélène Authier, Bernard Pipy, Etienne Meunier, David Olagnier, Lise Lefèvre, Olivier Neyrolles, Geanncarlo Lugo-Villarino, Agnès Coste, José Bernad

المساهمون: Infections Parasitaires : Transmission, Physiopathologie et Thérapeutiques (IP-TPT), Service de Santé des Armées-Assistance Publique - Hôpitaux de Marseille (APHM)-Aix Marseille Université (AMU)-Institut de Recherche pour le Développement (IRD), Institut de pharmacologie et de biologie structurale (IPBS), Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Centre National de la Recherche Scientifique (CNRS), Focal Area Infection Biology, Biozentrum, University of Basel (Unibas), Division of Infectious Diseases, University of South Florida (USF), Université Paris Descartes - Faculté de Médecine (UPD5 Médecine), Université Paris Descartes - Paris 5 (UPD5), Departament de Bioquímica i Biologia Molecular, Universitat de Barcelona (UB), Pharmacochimie et Biologie pour le Développement (PHARMA-DEV), Institut de Recherche pour le Développement (IRD)-Institut de Chimie de Toulouse (ICT-FR 2599), Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut de Recherche pour le Développement (IRD)-Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Centre National de la Recherche Scientifique (CNRS)-Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Centre National de la Recherche Scientifique (CNRS), Laboratoire de la Signalisation et de la Différenciation des Macrophages, Institution Louis Bugnard, Institut de génétique et biologie moléculaire et cellulaire (IGBMC), Université Louis Pasteur - Strasbourg I-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Institut de Chimie de Toulouse (ICT-FR 2599), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées-Centre National de la Recherche Scientifique (CNRS)-Institut de Recherche pour le Développement (IRD)-Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Institut de Chimie du CNRS (INC)-Institut National Polytechnique (Toulouse) (Toulouse INP), Université Fédérale Toulouse Midi-Pyrénées-Centre National de la Recherche Scientifique (CNRS)-Institut de Recherche pour le Développement (IRD)-Institut de Chimie du CNRS (INC)-Institut de Recherche pour le Développement (IRD), Centre National de la Recherche Scientifique (CNRS)-Université Toulouse III - Paul Sabatier (UT3), Université Fédérale Toulouse Midi-Pyrénées-Université Fédérale Toulouse Midi-Pyrénées, Institut de Recherche pour le Développement (IRD)-Aix Marseille Université (AMU)-Assistance Publique - Hôpitaux de Marseille (APHM)-Service de Santé des Armées, Institut de Recherche pour le Développement (IRD)-Institut de Chimie de Toulouse (ICT), Institut de Recherche pour le Développement (IRD)-Université Toulouse III - Paul Sabatier (UT3), Université de Toulouse (UT)-Université de Toulouse (UT)-Institut de Chimie du CNRS (INC)-Centre National de la Recherche Scientifique (CNRS)-Institut National Polytechnique (Toulouse) (Toulouse INP), Université de Toulouse (UT)-Institut de Recherche pour le Développement (IRD)-Université Toulouse III - Paul Sabatier (UT3), Université de Toulouse (UT), Université de Toulouse (UT)-Université de Toulouse (UT)-Centre National de la Recherche Scientifique (CNRS)

المصدر: Immunity
Immunity, Elsevier, 2013, 38 (5), pp.1038-1049. ⟨10.1016/j.immuni.2013.04.010⟩
Lefèvre, L, Lugo-Villarino, G, Meunier, E, Valentin, A, Olagnier, D, Authier, H, Duval, C, Dardenne, C, Bernad, J, Lemesre, J L, Auwerx, J, Neyrolles, O, Pipy, B & Coste, A 2013, ' The C-type lectin receptors dectin-1, MR, and SIGNR3 contribute both positively and negatively to the macrophage response to Leishmania infantum ' Immunity, vol. 38, no. 5, pp. 1038-49 . https://doi.org/10.1016/j.immuni.2013.04.010
Immunity, 2013, 38 (5), pp.1038-1049. ⟨10.1016/j.immuni.2013.04.010⟩

مصطلحات موضوعية: MESH: Signal Transduction, MESH: Mannose-Binding Lectins / metabolism, [SDV]Life Sciences [q-bio], Interleukin-1beta, MESH: Leishmaniasis, Visceral / immunology, Antigens, CD/metabolism, Macrophages/immunology, MESH: Mice, Knockout, MESH: Antigens, CD / metabolism, MESH: Protein-Tyrosine Kinases / metabolism, Caspase 1/metabolism, Mice, 0302 clinical medicine, MESH: Leishmania infantum / immunology, MESH: Caspase 1 / metabolism, C-type lectin, MESH: RNA, Small Interfering, MESH: Interleukin-1beta / antagonists & inhibitors, Macrophage, Immunology and Allergy, MESH: Animals, MESH: Leishmaniasis, Visceral / parasitology, MESH: Receptors, Cell Surface / metabolism, Leishmania infantum, RNA, Small Interfering, Receptor, MESH: NADPH Oxidases / metabolism, Cells, Cultured, ComputingMilieux_MISCELLANEOUS, Mice, Knockout, 0303 health sciences, Arachidonic Acid, biology, Effector, Caspase 1, Intracellular Signaling Peptides and Proteins, Protein-Tyrosine Kinases, MESH: Macrophages / immunology, 3. Good health, Cell biology, MESH: Leukotriene B4 / antagonists & inhibitors, Infectious Diseases, Intracellular Signaling Peptides and Proteins/metabolism, NADPH Oxidases/metabolism, Leishmaniasis, Visceral, [SDV.IMM]Life Sciences [q-bio]/Immunology, RNA Interference, Signal transduction, Interleukin-1beta/antagonists & inhibitors, Mannose receptor, Mannose Receptor, Signal Transduction, MESH: Cells, Cultured, MESH: Reactive Oxygen Species / metabolism, Immunology, MESH: RNA Interference, Receptors, Cell Surface, Leukotriene B4, Arachidonic Acid/metabolism, MESH: Syk Kinase, 03 medical and health sciences, Antigens, CD, Reactive Oxygen Species/metabolism, MESH: Mice, Inbred C57BL, parasitic diseases, MESH: Arachidonic Acid / metabolism, Animals, Humans, Syk Kinase, Lectins, C-Type, Lectins, C-Type/immunology, MESH: Interleukin-1beta / metabolism, Receptors, Cell Surface/metabolism, Mannose-Binding Lectins/metabolism, MESH: Mice, 030304 developmental biology, MESH: Humans, Leishmania infantum/immunology, Macrophages, NADPH Oxidases, biology.organism_classification, Leishmania, MESH: Mannose Receptor, Leishmaniasis, Visceral/immunology, Protein-Tyrosine Kinases/metabolism, MESH: Lectins, C-Type / metabolism, Mice, Inbred C57BL, MESH: Lectins, C-Type / immunology, Mannose-Binding Lectins, Leukotriene B4/antagonists & inhibitors, Reactive Oxygen Species, MESH: Intracellular Signaling Peptides and Proteins / metabolism, 030215 immunology

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6effd582598ceebe3d4eb3a68796326c
https://hal.archives-ouvertes.fr/hal-02348593

المؤلفون: Sumitra Ramachandran, Angela A. Dunne, Neil Derek Miller, Laiq Chaudry, Vincent Jaquet, Craig Allan, Aleksandra Filippova, Jean Lim, Ceri H. Davies, Agnes B. Ong, May Thu Ma, Shabina Nasir, Woei Shin Chen, Nicole Y. Ye, Edward R. Browne, Kiew Ching K Lee, Shihua Liao, Douglas G. Johns, Angela Bridges, Yasuji Matsuoka, Chiu Cheong Aw, A. Richard Rutter, Adeline L.W. Chueng, Dizheng Li, Deborah W. Chen, Sri Mulyanidewi, Sarah Duncan, Tamara Seredenina, Gary Pettman, Kazufumi Hirano, Pamela P. Oon

المصدر: Antioxidants & redox signaling, Vol. 23, No 5 (2015) pp. 358-374

مصطلحات موضوعية: Male, Physiology, Clinical Biochemistry, Aminopyridines, ddc:616.07, medicine.disease_cause, Biochemistry, Rats, Sprague-Dawley, Enzyme Inhibitors / therapeutic use, Membrane Glycoproteins / metabolism, Drug Discovery, Enzyme Inhibitors, Pancreatitis / drug therapy, Cells, Cultured, General Environmental Science, chemistry.chemical_classification, Sulfonamides, Membrane Glycoproteins, NADPH oxidase, biology, Chemistry, Enzyme Inhibitors / pharmacology, respiratory system, Small molecule, 3. Good health, Aminopyridines / chemistry, NADPH Oxidase 2, cardiovascular system, Sulfonamides / chemistry, circulatory and respiratory physiology, inorganic chemicals, In vivo, Forum Original Research Communication, medicine, Animals, NADPH Oxidases / metabolism, Molecular Biology, NOx, Sulfonamides / pharmacology, Reactive oxygen species, Aminopyridines / pharmacology, NADPH Oxidases / antagonists & inhibitors, Reactive Oxygen Species / metabolism, NADPH Oxidases, Cell Biology, In vitro, Mice, Inbred C57BL, Enzyme, Pancreatitis, Membrane Glycoproteins / antagonists & inhibitors, biology.protein, General Earth and Planetary Sciences, Reactive Oxygen Species, Oxidative stress

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2f7ea3e136424a97ddab898f120e2665
https://europepmc.org/articles/PMC4545375/

مصطلحات الفهرس: Animals, Antibodies/blood, Arthritis/blood/chemically induced/*immunology, Cartilage Oligomeric Matrix Protein, Disease Models, Animal, Extracellular Matrix Proteins/adverse effects/blood/*immunology, Female, Glycoproteins/adverse effects/blood/*immunology, Humans, Major Histocompatibility Complex/*genetics, Matrilin Proteins, Mice, NADPH Oxidases/*metabolism, Rats, Rheumatology and Autoimmunity, Reumatologi och inflammation, Article in journal, info:eu-repo/semantics/article, text

URL: http://urn.kb.se/resolve?urn=urn:nbn:se:hh:diva-48841
Arthritis and Rheumatism, 0004-3591, 2008, 58:7, s. 2000-2011