المؤلفون: Albert Lehr, Niklas Henneberg, Tarana Nigam, Walter Paulus, Andrea Antal

المصدر: Neural Plasticity, Vol 2019 (2019)

مصطلحات موضوعية: Neurosciences. Biological psychiatry. Neuropsychiatry, RC321-571

وصف الملف: electronic resource

Relation: https://doaj.org/toc/2090-5904; https://doaj.org/toc/1687-5443

URL الوصول: https://doaj.org/article/51f7d98b090440bd8831c766191aa1c1

المؤلفون: A. Claudio Cuello, Igor Klyubin, Niklas Henneberg, Hans-Rüdiger Geis, Falko Fuhrmann, Kevin Keppler, Stefan Remy, Kerstin Hoffmann, Detlef Friedrichs, Daniel Justus, Julia Steffen, Yingjie Qi, Martin Fuhrmann, Liudmila Sosulina, Manuel Mittag, Michael J. Rowan

المصدر: Journal of neurochemistry 157(6), 2128-2144 (2021). doi:10.1111/jnc.15323
Journal of neurochemistry, 157(6):2128-2144
Journal of Neurochemistry

مصطلحات موضوعية: Male, physiology [Excitatory Postsynaptic Potentials], Amyloid beta, Transgene, Hippocampus, hyperexcitability, Alzheimer's disease, disease model, β-amyloidosis, metabolism [Hippocampus], Biology, Hippocampal formation, Inhibitory postsynaptic potential, Biochemistry, 03 medical and health sciences, Cellular and Molecular Neuroscience, Amyloid beta-Protein Precursor, pathology [Alzheimer Disease], 0302 clinical medicine, Organ Culture Techniques, In vivo, Alzheimer Disease, metabolism [Amyloid beta-Protein Precursor], medicine, Extracellular, Biological neural network, Animals, ddc:610, 030304 developmental biology, 0303 health sciences, Chemistry, Excitatory Postsynaptic Potentials, medicine.disease, Rats, Disease Models, Animal, pathology [Hippocampus], nervous system, genetics [Amyloid beta-Protein Precursor], biology.protein, Female, Rats, Transgenic, Neuroscience, 030217 neurology & neurosurgery, metabolism [Alzheimer Disease]

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::48dbc18f3fbed18754361ce42e178282

المؤلفون: Julika Pitsch, Susanne Schoch, Daniel Justus, Tatjana Beutel, Stefan Remy, Zuzana Šišková, Albert J. Becker, Detlef Friedrichs, Heinz Von Der Kammer, Niklas Henneberg, Hiroshi Kaneko

المصدر: Neuron 84(5), 1023-1033 (2014). doi:10.1016/j.neuron.2014.10.024

مصطلحات موضوعية: Male, Neuroscience(all), Transgene, Models, Neurological, genetics [Alzheimer Disease], Mice, Transgenic, genetics [Mutation], Disease, Degeneration (medical), genetics [Action Potentials], Biology, In Vitro Techniques, biocytin, pathology [Alzheimer Disease], Mice, Amyloid beta-Protein Precursor, PSEN1 protein, human, Presenilin-1, Animals, Humans, pathology [Neurons], In patient, Computer Simulation, ddc:610, etiology [Nerve Degeneration], Electric stimulation, metabolism [Presenilin-1], General Neuroscience, drug effects [Action Potentials], Lysine, Lysine metabolism, pathology [Dendrites], pathology [Nerve Degeneration], complications [Alzheimer Disease], genetics [Presenilin-1], analogs & derivatives [Lysine], Electric Stimulation, metabolism [Lysine], Disease Models, Animal, pathology [Hippocampus], genetics [Amyloid beta-Protein Precursor], Neuronal Hyperexcitability, Female, Neuroscience, Function (biology)

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::17530b342671df57123d2a7398edea3c