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المؤلفون: Minh Tran, Natalie Castillo, Michael B. Jordan, Nora Lakes, Vandana Chaturvedi
المصدر: Blood. 138:1034-1039
مصطلحات موضوعية: Ruxolitinib, medicine.medical_treatment, Immunology, Pharmacology, Biochemistry, Lymphohistiocytosis, Hemophagocytic, Interferon-gamma, Mice, Pharmacokinetics, Interferon, hemic and lymphatic diseases, Nitriles, medicine, Animals, Hemophagocytic lymphohistiocytosis, business.industry, Cell Biology, Hematology, Janus Kinase 2, medicine.disease, Blockade, Disease Models, Animal, Pyrimidines, Cytokine, Toxicity, Pyrazoles, Janus kinase, business, Signal Transduction, medicine.drug
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::75d0e4393ed6a5538826244736147d57
https://doi.org/10.1182/blood.2020007930 -
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المؤلفون: Nora Lakes, Laura M. Canaday, Dan H. Barouch, Durga Krishnamurthy, Jasmine A. Tuazon, Hilal Cevik, Eryn Blass, Sanjeeth Rajaram, Ayad Ali, H. Alex Feldman, Harsha Seelamneni, Michael T. Moran, Stephen N. Waggoner
مصطلحات موضوعية: medicine.anatomical_structure, Immune system, Immunization, Interferon, Cell, medicine, Spleen, Biology, CXCR3, Natural killer cell, medicine.drug, Cell biology, Viral vector
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::04defe3fb9013721355c758e628db61c
https://doi.org/10.1101/2021.05.11.443590 -
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المؤلفون: Nora Lakes, Elisa Boscolo, Timothy Le Cras, Patricia Pastura, Jillian Goines
المصدر: The FASEB Journal. 33
مصطلحات موضوعية: Pathology, medicine.medical_specialty, Lymphatic system, Gain of function, PIK3CA gene, Capillary action, business.industry, Genetics, Medicine, business, Molecular Biology, Biochemistry, Biotechnology
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المؤلفون: Jay Greenberg, Simon P. Hogan, Jonathan D. Katz, David A. Hildeman, Carl E. Allen, Michael M. Henry, Scott H. Millen, Catherine E. Terrell, Vandana Chaturvedi, Kaitlin R. Carroll, Michelle L. Hermiston, Nora Lakes, Eileen E. Elfers, Michael B. Jordan, Julie Kanter, Stephan Ladisch, Michael Joyce, Jonathan P. McNally, Paul R. Andreassen
المصدر: Proceedings of the National Academy of Sciences of the United States of America, vol 114, iss 24
McNally, JP; Millen, SH; Chaturvedi, V; Lakes, N; Terrell, CE; Elfers, EE; et al.(2017). Manipulating DNA damage-response signaling for the treatment of immune-mediated diseases. PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 114(24), E4782-E4791. doi: 10.1073/pnas.1703683114. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/7056s1hrمصطلحات موضوعية: 0301 basic medicine, Cell cycle checkpoint, Cell division, T-Lymphocytes, Hemophagocytic, Drug Evaluation, Preclinical, 129 Strain, Inbred C57BL, Lymphocyte Activation, DNA damage response, medicine.disease_cause, Transgenic, Autoimmunity, Mice, 0302 clinical medicine, therapeutics, Encephalomyelitis, Inbred BALB C, Etoposide, Mice, Knockout, Lymphohistiocytosis, Mice, Inbred BALB C, Multidisciplinary, autoimmunity, Proto-Oncogene Proteins c-mdm2, Preclinical, PNAS Plus, Immune System Diseases, 030220 oncology & carcinogenesis, Signal transduction, Signal Transduction, Encephalomyelitis, Autoimmune, Experimental, Mice, 129 Strain, Multiple Sclerosis, DNA damage, Knockout, Mice, Transgenic, Biology, Lymphohistiocytosis, Hemophagocytic, Experimental, 03 medical and health sciences, Immune system, Antigen, medicine, Animals, Humans, Animal, immune regulation, Cell Cycle Checkpoints, biochemical phenomena, metabolism, and nutrition, In vitro, Mice, Inbred C57BL, Disease Models, Animal, 030104 developmental biology, Disease Models, Immunology, Cancer research, bacteria, Drug Evaluation, Tumor Suppressor Protein p53, Autoimmune, DNA Damage
وصف الملف: application/pdf
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c2722170f54800d62e18982b8ef9724
https://doi.org/10.1073/pnas.1703683114
