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    دورية أكاديمية

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    المؤلفون: Özduman, Gülseren

    المساهمون: Korkmaz, Kemal Sami, Fen Bilimleri Enstitüsü, Biyoteknoloji Anabilim Dalı

    وصف الملف: application/pdf

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    المساهمون: Council for Scientific and Industrial Research [Pretoria] (CSIR), AstraZeneca, Gothenburg, Sweden, University of Cape Town, Africa Health Research Institute [KwaZulu-Natal] (AHRI), University of KwaZulu-Natal (UKZN), Max Planck Institut für Infektionsbiologie (MPIIB), Max-Planck-Gesellschaft, International Centre for Genetic Engineering and Biotechnology [Cape Town] (ICGEB), Universidade de Lisboa (ULISBOA), This research is supported by grant PG V2KYP07 from the Council for Industrial and Scientific Research (CSIR, South Africa) and by a grant from the Emerging Research Area Program & the Centre of Competence Program of The Department of Science and Technology (DST, South Africa) and a grant from the Medical Research Council (South Africa) all to MMM.

    المصدر: Frontiers in Cellular and Infection Microbiology
    Frontiers in Cellular and Infection Microbiology, Frontiers, 2018, 8, pp.263. ⟨10.3389/fcimb.2018.00263⟩
    Frontiers in Cellular and Infection Microbiology, Vol 8 (2018)

    مصطلحات موضوعية: MESH: Signal Transduction, 0301 basic medicine, MAPK/ERK pathway, [SDV]Life Sciences [q-bio], MESH: DNA Breaks, Double-Stranded, lcsh:QR1-502, MAP Kinase Kinase 1, Plasma protein binding, HIV Envelope Protein gp120, lcsh:Microbiology, ERK2, Heterogeneous-Nuclear Ribonucleoprotein K, MESH: HIV-1, MESH: HIV Envelope Protein gp120, chemistry.chemical_compound, Cellular and Infection Microbiology, 0302 clinical medicine, Transcription (biology), MAP2K1, DNA Breaks, Double-Stranded, MESH: Immune Evasion, Cells, Cultured, Original Research, Mitogen-Activated Protein Kinase 1, 0303 health sciences, apoptosis, Long non-coding RNA, 3. Good health, Cell biology, MESH: RNA, Long Noncoding, Infectious Diseases, medicine.anatomical_structure, 030220 oncology & carcinogenesis, Host-Pathogen Interactions, HuR, RNA, Long Noncoding, MESH: MAP Kinase Kinase 1, Signal Transduction, MESH: Cells, Cultured, MESH: Mitogen-Activated Protein Kinase 1, Cyclin-Dependent Kinase Inhibitor p21, Nutlin3a, Microbiology (medical), DNA damage, T cell, Immunology, macrophage, Biology, Microbiology, Virus, MESH: Cyclin-Dependent Kinase Inhibitor p21, 03 medical and health sciences, hnRNP-K, MESH: Heterogeneous-Nuclear Ribonucleoprotein K, medicine, Humans, lincRNA-p21, Gene, Immune Evasion, 030304 developmental biology, MESH: Humans, Macrophages, MESH: Apoptosis, MESH: Host-Pathogen Interactions, MESH: Macrophages, 030104 developmental biology, chemistry, Apoptosis, HIV-1, Function (biology), DNA

    وصف الملف: application/pdf