نتائج البحث - "Oncogene Proteins/genetics/*metabolism"

1

YY1 inhibits the activation of the p53 tumor suppressor in response to genotoxic stress

المؤلفون: Grönroos, Eva, Terentiev, Alexei A, Punga, Tanel, Ericsson, Johan

المصدر: Proceedings of the National Academy of Sciences of the United States of America. 101(33):12165-12170

مصطلحات موضوعية: Cell Line, DNA Damage, DNA-Binding Proteins/genetics/*metabolism, Humans, Nuclear Proteins/genetics/metabolism, Promoter Regions (Genetics), Protein Binding, Protein p53/genetics/*metabolism, Proto-Oncogene Proteins/genetics/metabolism, Recombinant Proteins/genetics/metabolism, Research Support, Non-U.S. Gov't, Sequence Deletion, Trans-Activators/genetics/metabolism, Transcription Factors/genetics/*metabolism, Transcription, Genetic, Ubiquitin/metabolism

وصف الملف: print

URL الوصول: https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-73040
https://doi.org/10.1073/pnas.0402283101
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed&cmd=Retrieve&list_uids=15295102&dopt=Citation

2

LYL1 activity is required for the maturation of newly formed blood vessels in adulthood

المؤلفون: Fred Sablitzky, Danièle Mathieu, Philippe Couttet, Christiane Dohet, Rawan El-Hajj, Valérie Pinet, Nelly Pirot, Virginie Deleuze

المساهمون: Centre National de la Recherche Scientifique (CNRS), Institut de Génétique Moléculaire de Montpellier (IGMM), Centre National de la Recherche Scientifique (CNRS)-Université de Montpellier (UM)

المصدر: Blood
Blood, American Society of Hematology, 2010, 115 (25), pp.5270-9. ⟨10.1182/blood-2010-03-275651⟩
Blood, American Society of Hematology, 2010, 115 (25), pp.5270--9. ⟨10.1182/blood-2010-03-275651⟩

مصطلحات موضوعية: MESH: Neoplasm Proteins, Pathology, MESH: Mice, Mutant Strains, Angiogenesis, [SDV]Life Sciences [q-bio], MESH: Hematopoiesis, Biochemistry, MESH: Cadherins, Neovascularization, Angiopoietin-2/biosynthesis/genetics Animals Antigens, Mice, 0302 clinical medicine, MESH: Basic Helix-Loop-Helix Transcription Factors, MESH: Up-Regulation, Basic Helix-Loop-Helix Transcription Factors, MESH: Animals, MESH: Antigens, CD, ComputingMilieux_MISCELLANEOUS, T-Cell Acute Lymphocytic Leukemia Protein 1, Regulation of gene expression, 0303 health sciences, Hematology, Neovascularization, Pathologic, MESH: Gene Expression Regulation, Neoplastic, Cadherins, Neoplasm Proteins, Up-Regulation, Cell biology, Gene Expression Regulation, Neoplastic, Haematopoiesis, MESH: Neoplasms, Experimental, medicine.anatomical_structure, Pathologic/genetics/*metabolism Proto-Oncogene Proteins/genetics/metabolism Up-Regulation/genetics, 030220 oncology & carcinogenesis, CD/biosynthesis/genetics Basic Helix-Loop-Helix Transcription Factors/genetics/*metabolism Cadherins/biosynthesis/genetics Gene Expression Regulation, medicine.symptom, Blood vessel, medicine.medical_specialty, Mutant Strains Neoplasm Proteins/genetics/*metabolism Neoplasm Transplantation Neoplasms, Immunology, Experimental/*blood supply/genetics/*metabolism Neovascularization, Biology, Angiopoietin-2, Neoplastic/genetics Hematopoiesis/genetics Humans Mice Mice, 03 medical and health sciences, MESH: Angiopoietin-2, Antigens, CD, Proto-Oncogene Proteins, Internal medicine, medicine, Animals, Humans, [SDV.BBM]Life Sciences [q-bio]/Biochemistry, Molecular Biology, MESH: Mice, 030304 developmental biology, Matrigel, MESH: Humans, Wild type, Neoplasms, Experimental, Cell Biology, Mice, Mutant Strains, Hematopoiesis, MESH: Proto-Oncogene Proteins, MESH: T-Cell Acute Lymphocytic Leukemia Protein 1, MESH: Neovascularization, Pathologic, MESH: Neoplasm Transplantation, Neoplasm Transplantation

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0c6b15f3f46b37bd3b4784267d61ee12
https://doi.org/10.1182/blood-2010-03-275651

3

Cooperation between PU.1 and CAAT/Enhancer-binding Protein β Is Necessary to Induce the Expression of the MD-2 Gene

المؤلفون: Agnieszka Ochoda, Irène Dunn-Siegrist, Geneviève Drifte, Jérôme Pugin, Pierre Tissières, Tanguy Araud

المصدر: Journal of Biological Chemistry, Vol. 284, No 39 (2009) pp. 26261-72
The Journal of biological chemistry

مصطلحات موضوعية: Myeloid, Interleukin-6/pharmacology, Binding Sites/genetics, Biochemistry, Monocytes, 0302 clinical medicine, Enhancer binding, Myeloid Cells, Luciferases, Promoter Regions, Genetic, Receptor, ddc:616, Regulation of gene expression, 0303 health sciences, Reverse Transcriptase Polymerase Chain Reaction, Lymphocyte Antigen 96/genetics/metabolism, CCAAT-Enhancer-Binding Protein-beta/genetics/metabolism, Protein Binding/drug effects, Luciferases/genetics/metabolism, 3. Good health, medicine.anatomical_structure, 030220 oncology & carcinogenesis, RNA Interference, Signal transduction, Protein Binding, Chromatin Immunoprecipitation, Blotting, Western, Molecular Sequence Data, Lymphocyte Antigen 96, Trans-Activators/genetics/metabolism, HL-60 Cells, Biology, Transfection, Cell Line, 03 medical and health sciences, Cell Line, Tumor, Proto-Oncogene Proteins, medicine, Humans, Transcription, Chromatin, and Epigenetics, Binding site, Hepatocytes/drug effects/metabolism/pathology, Molecular Biology, Gene, Transcription factor, 030304 developmental biology, Myeloid Cells/drug effects/metabolism/pathology, Binding Sites, Base Sequence, Interleukin-6, CCAAT-Enhancer-Binding Protein-beta, Proto-Oncogene Proteins/genetics/metabolism, Cell Biology, Promoter Regions, Genetic/genetics, Molecular biology, Alternative Splicing, Gene Expression Regulation, Mutation, Monocytes/cytology/drug effects/metabolism, Hepatocytes, Trans-Activators

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3e4e90364c8c0f1c09418b7dbd591ef3
https://doi.org/10.1074/jbc.m109.042580

4

C. elegans ced-13 can promote apoptosis and is induced in response to DNA damage.

المؤلفون: Schumacher, B, Schertel, C, Wittenburg, N, Tuck, S, Mitani, S, Gartner, A, Conradt, B, Shaham, S

المصدر: Cell Death Differ. 12(2):153-61

وصف الملف: print

URL الوصول: https://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-17950
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed&cmd=Retrieve&list_uids=15605074&dopt=Citation

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5

Evidence of impaired adipogenesis in insulin resistance

المؤلفون: Yang, Xiao Lin, 1955, Jansson, Per-Anders, 1961, Nagaev, Ivan, 1962, Jack, M. M., Carvalho, Eugénia, 1967, Stibrant Sunnerhagen, Katharina, 1957, Cam, M. C., Cushman, S. W., Smith, Ulf, 1943

المصدر: Biochem Biophys Res Commun. 317(4):1045-51

مصطلحات موضوعية: Endocrinology and Diabetes, Endokrinologi och diabetes, Adipocytes/*metabolism/physiology, Adipose Tissue/cytology/*metabolism, Adult, Case-Control Studies, Cell Size, Cytoskeletal Proteins/biosynthesis, DNA-Binding Proteins/metabolism, Diabetes Mellitus, Type 2/genetics/metabolism, Family Health, Gene Expression Profiling, Glycogen Synthase Kinase 3/metabolism, Humans, Insulin Resistance/*physiology, Lymphoid Enhancer-Binding Factor 1, Male, Middle Aged, Muscle, Skeletal/metabolism/physiology, Oligonucleotide Array Sequence Analysis, Proto-Oncogene Proteins/genetics/metabolism, Regression Analysis, Signal Transduction, T Cell Transcription Factor 1, Trans-Activators/biosynthesis, Transcription Factors/metabolism, Wnt Proteins, Wnt1 Protein, *Zebrafish Proteins, beta Catenin

URL الوصول: https://gup.ub.gu.se/publication/50385

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6

Fibrosis and stenosis of the long penetrating cerebral arteries: the cause

المؤلفون: Miao, Qing, Paloneva, Timo, Tuominen, Susanna, Pöyhönen, Minna, Tuisku, Seppo, Viitanen, Matti, Kalimo, Hannu

المصدر: Brain Pathol. 14(4):358-64

مصطلحات موضوعية: Actins/metabolism, Aged, Brain/metabolism/pathology/ultrastructure, CADASIL/complications/genetics/metabolism/*pathology, Case-Control Studies, Cerebral Arteries/*pathology/ultrastructure, Cerebral Infarction/complications/genetics/metabolism/*pathology, Collagen Type I/metabolism, Comparative Study, Constriction, Pathologic/physiopathology, Dementia, Vascular/complications/genetics/metabolism/*pathology, Female, Fibrosis/physiopathology, Humans, Immunohistochemistry/methods, Male, Microscopy, Electron, Transmission/methods, Middle Aged, Mutation, Myocytes, Smooth Muscle/metabolism/pathology, Proto-Oncogene Proteins/genetics/metabolism, Receptors, Cell Surface/genetics/metabolism, Research Support, Non-U.S. Gov't, Risk Factors, Staining and Labeling/methods

وصف الملف: print

URL الوصول: https://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-73774
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=PubMed&cmd=Retrieve&list_uids=15605982&dopt=Citation

7

An integrated genomic-transcriptomic approach supports a role for the proto-oncogene BCL3 in atherosclerosis

المؤلفون: Domenico Girelli, Francesco Mascoli, Oliviero Olivieri, Nicola Martinelli, Silvia Meneghetti, Manuela Ferracin, Francesco Bernardi, Francesca Pizzolo, Giovanni Malerba, Marie-Luce Bochaton-Piallat, Carlotta Zerbinati, Giuseppe Guastella, Simonetta Friso, Teresa Gagliano, Matteo Coen, Giovanna Marchetti, Barbara Lunghi, Matteo Bovolenta

المساهمون: Marchetti G, Girelli D, Zerbinati C, Lunghi B, Friso S, Meneghetti S, Coen M, Gagliano T, Guastella G, Bochaton-Piallat ML, Pizzolo F, Mascoli F, Malerba G, Bovolenta M, Ferracin M, Olivieri O, Bernardi F, Martinelli N

المصدر: Thrombosis and Haemostasis, Vol. 113, No 3 (2015) pp. 655-663

مصطلحات موضوعية: 0301 basic medicine, metabolic disorder, Carotid Artery Diseases, Male, Microarray, Messenger, Myocardial Infarction, Coronary Artery Disease, ddc:616.07, 030204 cardiovascular system & hematology, Transcription Factors/genetics/metabolism, Bioinformatics, Proto-Oncogene Mas, Muscle, Smooth, Vascular, Coronary artery disease, genomic, transcriptomics, Muscle, Smooth, Vascular/metabolism, 0302 clinical medicine, Gene Frequency, Smooth Muscle, B-Cell Lymphoma 3 Protein, Risk Factors, Odds Ratio, Cells, Cultured, Oligonucleotide Array Sequence Analysis, Genetics, education.field_of_study, Cultured, RNA, Messenger/analysis, Single Nucleotide, Hematology, Genomics, Middle Aged, BCL3, genomics, coronary artery disease, metabolic disorders, Phenotype, Carotid Artery Diseases/diagnosis/genetics/metabolism, Muscle, Female, Smooth, Coronary Artery Disease/diagnosis/genetics/metabolism, Cells, Population, Myocytes, Smooth Muscle, Socio-culturale, Single-nucleotide polymorphism, Biology, Polymorphism, Single Nucleotide, 03 medical and health sciences, Vascular, Chromosome 19, Proto-Oncogene Proteins, medicine, Humans, Genetic Predisposition to Disease, RNA, Messenger, Polymorphism, Allele, education, Metabolic disorders, Transcriptomics, Aged, Case-Control Studies, Chi-Square Distribution, Gene Expression Profiling, Gene Expression Regulation, Genome-Wide Association Study, Linear Models, Multivariate Analysis, Transcription Factors, Transcriptome, Genetic association, Myocytes, Myocytes, Smooth Muscle/metabolism, BCL3, coronary artery disease, genomics, metabolic disorders, transcriptomics, Proto-Oncogene Proteins/genetics/metabolism, Odds ratio, medicine.disease, Myocardial Infarction/diagnosis/genetics/metabolism, Genomics/methods, 030104 developmental biology, RNA, Gene Expression Profiling/methods

وصف الملف: STAMPA

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7caae23ab9057b521b08bcc05c2c1b35
https://pubmed.ncbi.nlm.nih.gov/25374339

8

The Liver Receptor Homolog-1 (LRH-1) is Expressed in Human Islets and Protects {beta}-cells against Stress-Induced Apoptosis

المؤلفون: Sophie Borot, Nadia Cobo-Vuilleumier, Benoit R. Gauthier, Julie Kerr-Conte, Mathurin Baquié, Claes B. Wollheim, François Pattou, Petra I. Lorenzo, Haiyan Wang, Domenico Bosco, Carmen M. Jimenez Moreno, Christopher R. Cederroth, Serge Nef, Luc St-Onge, Piero Marchetti

المساهمون: Swiss National Science Foundation, Juvenile Diabetes Research Foundation International, Junta de Andalucía, Fundación Progreso y Salud

المصدر: Human molecular genetics
Human Molecular Genetics, Vol. 20, No 14 (2011) pp. 2823-33
Digital.CSIC. Repositorio Institucional del CSIC
instname

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::39d1c6daba9f7307efadf39d9c7cab77
http://hdl.handle.net/11568/151212

9

Reduced basal autophagy and impaired mitochondrial dynamics due to loss of Parkinson's disease-associated protein DJ-1

المؤلفون: Zemfira Gizatullina, Sabine Ruckerbauer, Philipp J. Kahle, Brigitte Maurer, Frank N. Gellerich, Nicole Kieper, Jens Waak, Dirk Woitalla, Olaf Riess, Guido Krebiehl, Hartwig Wolburg, Tassula Proikas-Cezanne, Rejko Krüger, Lena F. Burbulla

المساهمون: Luxembourg Centre for Systems Biomedicine (LCSB): Clinical & Experimental Neuroscience (Krüger Group) [research center]

المصدر: PloS one, 5(2), 9367. United States (2010).
PLoS ONE, Vol 5, Iss 2, p e9367 (2010)
PLoS ONE

مصطلحات موضوعية: Parkinson's disease, Oncogene Proteins/genetics/metabolism, Protein Deglycase DJ-1, lcsh:Medicine, Mitochondrion, medicine.disease_cause, Oxidative Phosphorylation, Mice, Intracellular Signaling Peptides and Proteins/genetics/metabolism, Phosphorylation, lcsh:Science, Mitogen-Activated Protein Kinase 1, Oncogene Proteins, chemistry.chemical_classification, Mice, Knockout, Multidisciplinary, Neurodegeneration, Intracellular Signaling Peptides and Proteins, Parkinson Disease, Mitochondria, Cell biology, Genetics & genetic processes [F10] [Life sciences], Génétique & processus génétiques [F10] [Sciences du vivant], Research Article, Lysosomes/metabolism/ultrastructure, Blotting, Western, Oxidative phosphorylation, Biology, Neurological Disorders, Mitogen-Activated Protein Kinase 1/metabolism, Reactive Oxygen Species/metabolism, Mitochondria/metabolism, medicine, Autophagy, Animals, Humans, Loss function, Reactive oxygen species, Parkinson Disease/genetics/pathology, lcsh:R, Fibroblasts/cytology/metabolism/ultrastructure, Peroxiredoxins, Cell Biology, Fibroblasts, medicine.disease, Microscopy, Electron, chemistry, Mutation, lcsh:Q, Lysosomes, Reactive Oxygen Species, Oxidative stress, Neuroscience

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::28e923b3a28eb42762b7868e602b3939
http://orbilu.uni.lu/handle/10993/17172

10

Tumour angiogenesis is reduced in the Tc1 mouse model of Down's syndrome

المصدر: Nature, Vol. 465, No 7299 (2010) pp. 813-817

مصطلحات موضوعية: Vascular Endothelial Growth Factor A, Male, Candidate gene, Angiogenesis, Oncogene Proteins/genetics/metabolism, ETS2, Gene Dosage, Melanoma, Experimental, CADHERIN, Trisomy, ddc:616.07, Neovascularization, Neovascularization, Pathologic/ genetics/pathology, chemistry.chemical_compound, Carcinoma, Lewis Lung, Mice, 0302 clinical medicine, inhibitors/metabolism/pharmacology, Oncogene Proteins, 0303 health sciences, Multidisciplinary, Neovascularization, Pathologic, Intracellular Signaling Peptides and Proteins, ENDOTHELIAL-CELL PROLIFERATION, VEGF, Carcinoma, Lewis Lung/ blood supply/complications/genetics/pathology, Trisomy/genetics, APOPTOSIS, ADAMTS-1, 3. Good health, Multidisciplinary Sciences, Vascular endothelial growth factor, Vascular endothelial growth factor A, Carrier Proteins/genetics/metabolism, 030220 oncology & carcinogenesis, Science & Technology - Other Topics, GROWTH, Female, medicine.symptom, General Science & Technology, Immunoglobulins, Melanoma, Experimental/ blood supply/complications/genetics/pathology, Biology, Cell Adhesion Molecules/antagonists & inhibitors/genetics/metabolism, Proto-Oncogene Protein c-ets-2, 03 medical and health sciences, Down Syndrome/complications/ genetics/physiopathology, Transcriptional Regulator ERG, ADAMTS1 Protein, Vascular Endothelial Growth Factor Receptor-2/metabolism, MD Multidisciplinary, Immunoglobulins/genetics/metabolism, medicine, Chromosomes, Mammalian/genetics, Animals, Humans, DSCR1, Vascular Endothelial Growth Factor A/antagonists &, 030304 developmental biology, Science & Technology, Lewis lung carcinoma, Kinase insert domain receptor, GENE, Chromosomes, Mammalian, Vascular Endothelial Growth Factor Receptor-2, ADAM Proteins, Disease Models, Animal, chemistry, Immunology, Cancer research, ADAM Proteins/genetics/metabolism, Proto-Oncogene Protein c-ets-2/genetics/metabolism, Down Syndrome, Chromosome 21, Carrier Proteins, Cell Adhesion Molecules, Neoplasm Transplantation, Transcription Factors, Gene Dosage/ genetics

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c763c0fe2256ff15e5cc52b2b740dba
https://archive-ouverte.unige.ch/unige:11549

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