نتائج البحث - "Pahini Pandya"

المصدر: Nature cell biology
Recercat. Dipósit de la Recerca de Catalunya
instname
Nature Cell Biology
Dipòsit Digital de la UB
Universidad de Barcelona
Torrano, V, Valcarcel-Jimenez, L, Cortazar, A R, Liu, X, Urosevic, J, Castillo-Martin, M, Fernández-Ruiz, S, Morciano, G, Caro-Maldonado, A, Guiu, M, Zúñiga-García, P, Graupera, M, Bellmunt, A, Pandya, P, Lorente, M, Martín-Martín, N, David Sutherland, J, Sanchez-Mosquera, P, Bozal-Basterra, L, Zabala-Letona, A, Arruabarrena-Aristorena, A, Berenguer, A, Embade, N, Ugalde-Olano, A, Lacasa-Viscasillas, I, Loizaga-Iriarte, A, Unda-Urzaiz, M, Schultz, N, Aransay, A M, Sanz-Moreno, V, Barrio, R, Velasco, G, Pinton, P, Cordon-Cardo, C, Locasale, J W, Gomis, R R & Carracedo, A 2016, ' The metabolic co-regulator PGC1α suppresses prostate cancer metastasis ', Nature Cell Biology, vol. 18, no. 6, pp. 645-656 . https://doi.org/10.1038/ncb3357
Repositorio Institucional de la Consejería de Sanidad de la Comunidad de Madrid
Consejería de Sanidad de la Comunidad de Madrid

مصطلحات موضوعية: 0301 basic medicine, peroxisome proliferator-activated receptor gamma co-activator 1α (PGC1α), Oncology, Male, medicine.medical_specialty, Urology, 030232 urology & nephrology, Regulator, Biology, Article, NO, Metastasis, Transcriptome, Metastasis Suppression, Mice, 03 medical and health sciences, Prostate cancer, 0302 clinical medicine, Metàstasi, PGC1α, Internal medicine, medicine, peroxisome proliferator-activated receptor gamma co-activator 1α (PGC1α), prostate cancer,cancer progression, metastasis, metastasis, Animals, Humans, Neoplasm Metastasis, Receptor, Càncer, Heat-Shock Proteins, Cancer, business.industry, Prostatic Neoplasms, Cell Biology, medicine.disease, cancer progression, Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha, Cell biology, Mitochondria, Metabolic pathway, Disease Models, Animal, 030104 developmental biology, Receptors, Estrogen, Energy Metabolism, business

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URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e34f0e9d7610f954a76c2ba87f82ad29
http://europepmc.org/articles/PMC4884178

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Myosin II Reactivation and Cytoskeletal Remodeling as a Hallmark and a Vulnerability in Melanoma Therapy Resistance

المؤلفون: Silvia Mele, Jo Monger, Sophia N. Karagiannis, Panagiotis Karagiannis, Mirella Georgouli, Lena Boehme, Oscar Maiques, Anna Perdrix-Rosell, Victoria L. Bridgeman, Amine Sadok, Victoria Sanz-Moreno, Rebecca Lee, Irene Rodriguez-Hernandez, Ilaria Malanchi, Fredrik Wallberg, Jose L. Orgaz, Pahini Pandya, Eva Crosas-Molist, Christopher J. Tape

المصدر: Cancer Cell
Orgaz, J L, Crosas-Molist, E, Sadok, A, Perdrix-Rosell, A, Maiques, O, Rodriguez-Hernandez, I, Monger, J, Mele, S, Georgouli, M, Bridgeman, V, Karagiannis, P, Lee, R, Pandya, P, Boehme, L, Wallberg, F, Tape, C, Karagiannis, S N, Malanchi, I & Sanz-Moreno, V 2020, ' Myosin II Reactivation and Cytoskeletal Remodeling as a Hallmark and a Vulnerability in Melanoma Therapy Resistance ', Cancer Cell, vol. 37, no. 1, pp. 85-103.e9 . https://doi.org/10.1016/j.ccell.2019.12.003
CANCER CELL
Orgaz, J L, Crosas-Molist, E, Sadok, A, Perdrix-Rosell, A, Maiques, O, Rodriguez-Hernandez, I, Monger, J, Mele, S, Georgouli, M, Bridgeman, V, Karagiannis, P, Lee, R, Pandya, P, Boehme, L, Wallberg, F, Tape, C, Karagiannis, S N, Malanchi, I & Sanz-Moreno, V 2020, ' Myosin II Reactivation and Cytoskeletal Remodeling as a Hallmark and a Vulnerability in Melanoma Therapy Resistance ', CANCER CELL, vol. 37, no. 1, pp. 85-103.e9 . https://doi.org/10.1016/j.ccell.2019.12.003

مصطلحات موضوعية: 0301 basic medicine, MAPK/ERK pathway, Male, Cancer Research, medicine.medical_treatment, Mice, SCID, VEMURAFENIB, T-Lymphocytes, Regulatory, B7-H1 Antigen, regulatory T cells, Targeted therapy, ACTIVATION, Mice, 0302 clinical medicine, Mice, Inbred NOD, Myosin, Medicine and Health Sciences, Tumor Microenvironment, Medicine, cytoskeletal remodeling, Rho-associated protein kinase, Melanoma, Cytoskeleton, rho-Associated Kinases, melanoma therapy resistance, Manchester Cancer Research Centre, transcriptional rewiring, phosphoproteomics and transcriptomics, Stem Cells, Cell Cycle, 3. Good health, tumor-promoting macrophages, Treatment Outcome, Oncology, BRAF INHIBITOR RESISTANCE, INDUCED TRANSCRIPTION, 030220 oncology & carcinogenesis, Female, immunotherapy, Genetics & Genomics, EXPRESSION, Model organisms, Proto-Oncogene Proteins B-raf, DNA damage, MAP Kinase Signaling System, Mice, Nude, RHO-GTPASES, Article, MECHANISMS, 03 medical and health sciences, myosin II, Cell Line, Tumor, KINASE, Animals, Humans, MEK INHIBITION, Rho-kinase, Protein Kinase Inhibitors, Myosin Type II, business.industry, ResearchInstitutes_Networks_Beacons/mcrc, Cell Biology, Immunotherapy, Tumour Biology, medicine.disease, MAPK, Immune checkpoint, Mice, Inbred C57BL, Oxidative Stress, 030104 developmental biology, Drug Resistance, Neoplasm, Cancer research, business, Reactive Oxygen Species, ACQUIRED-RESISTANCE, DNA Damage

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f80ff2407510ff4659e97caceb2b1d67
https://pubmed.ncbi.nlm.nih.gov/31935375

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