نتائج البحث - "Raymond W Zhou"

المصدر: Yu, Z; Li, CF; Mkhikian, H; Zhou, RW; Newton, BL; & Demetriou, M. (2014). Family studies of type 1 diabetes reveal additive and epistatic effects between MGAT1 and three other polymorphisms. Genes and Immunity, 15(4), 218-223. doi: 10.1038/gene.2014.7. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/39j0063f
Genes and immunity, vol 15, iss 4
Genes and immunity

مصطلحات موضوعية: Male, type 1 diabetes, Datasets as Topic, N-glycosylation, 0302 clinical medicine, Receptors, genetic interaction, MGAT1, 2.1 Biological and endogenous factors, Cytotoxic T cell, CTLA-4 Antigen, Aetiology, Genetics (clinical), Pediatric, Genetics, 0303 health sciences, Diabetes, Female, Type 1, Immunology, Biology, N-Acetylglucosaminyltransferases, Autoimmune Disease, Article, 03 medical and health sciences, Genetic, Antigen, Genetic variation, Diabetes Mellitus, medicine, Humans, Family, Gene, Metabolic and endocrine, 030304 developmental biology, Type 1 diabetes, Receptors, Interleukin-7, Interleukin-7, Multiple sclerosis, Haplotype, Interleukin-2 Receptor alpha Subunit, Genetic Variation, Epistasis, Genetic, medicine.disease, Brain Disorders, Diabetes Mellitus, Type 1, Epistasis, 030217 neurology & neurosurgery

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URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a10bb2f948c07b15c15b3f8b39f66b3
https://doi.org/10.1038/gene.2014.7

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5

Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants

المؤلفون: Barbara L. Newton, Raymond W. Zhou, Carey F. Li, Haik Mkhikian, Michael A. Demetriou, Zhaoxia Yu

المصدر: Li, CF; Zhou, RW; Mkhikian, H; Newton, BL; Yu, Z; & Demetriou, M. (2013). Hypomorphic MGAT5 polymorphisms promote multiple sclerosis cooperatively with MGAT1 and interleukin-2 and 7 receptor variants. Journal of Neuroimmunology, 256(1-2), 71-76. doi: 10.1016/j.jneuroim.2012.12.008. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/1b5170sc

مصطلحات موضوعية: Adult, Male, Interleukin 2, Multiple Sclerosis, T-Lymphocytes, T cell, Immunology, Down-Regulation, Biology, N-Acetylglucosaminyltransferases, medicine.disease_cause, Article, Autoimmunity, Cohort Studies, Young Adult, symbols.namesake, Downregulation and upregulation, Risk Factors, medicine, Humans, Immunology and Allergy, CTLA-4 Antigen, Receptor, Interleukin-7 receptor, Receptors, Interleukin-7, Multiple sclerosis, Genetic Variation, Receptors, Interleukin-2, Middle Aged, Golgi apparatus, Flow Cytometry, Galactosyltransferases, medicine.disease, Molecular biology, medicine.anatomical_structure, Neurology, Case-Control Studies, Cancer research, symbols, Female, Neurology (clinical), medicine.drug

وصف الملف: application/pdf

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca9087ce06d92631e4790d3625ec9c20
https://doi.org/10.1016/j.jneuroim.2012.12.008

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6

Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis

المؤلفون: Patil Kavarian, Stuart M. Haslam, Gang Wu, Christie-Lynn Mortales, Anne Dell, Haik Mkhikian, Michael A. Demetriou, Khachik Khachikyan, Raymond W. Zhou

المساهمون: Biotechnology and Biological Sciences Research Council (BBSRC), Wellcome Trust

المصدر: Mkhikian, Haik; Mortales, Christie-Lynn; Zhou, Raymond W; Khachikyan, Khachik; Wu, Gang; Haslam, Stuart M; et al.(2016). Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis. ELIFE, 5. doi: 10.7554/eLife.14814. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/0fb3v6d4
eLife
Mkhikian, H; Mortales, C-L; Zhou, RW; Khachikyan, K; Wu, G; Haslam, SM; et al.(2016). Golgi self-correction generates bioequivalent glycans to preserve cellular homeostasis.. eLife, 5. doi: 10.7554/eLife.14814. UC Irvine: Retrieved from: http://www.escholarship.org/uc/item/9dk0b4st
eLife, Vol 5 (2016)

مصطلحات موضوعية: 0301 basic medicine, Life Sciences & Biomedicine - Other Topics, Mouse, T-Lymphocytes, Cell, Cellular homeostasis, Golgi Apparatus, N-glycosylation, 0601 Biochemistry and Cell Biology, Biochemistry, O-GLYCOSYLATION, HIGH-AFFINITY, Mice, N-linked glycosylation, Golgi, Homeostasis, Biology (General), Receptor, Cells, Cultured, galectin, biology, General Neuroscience, General Medicine, MULTIPLE-SCLEROSIS, ASPARAGINE-LINKED OLIGOSACCHARIDES, Cell biology, medicine.anatomical_structure, symbols, Medicine, Life Sciences & Biomedicine, Protein Binding, Research Article, Human, Glycan, QH301-705.5, T cell, Science, Galectins, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, symbols.namesake, ADVANCED MALIGNANCIES, Polysaccharides, medicine, Animals, Biology, Galectin, POLY-N-ACETYLLACTOSAMINE, Glycoproteins, bioequivalence, Science & Technology, COMPLEX, General Immunology and Microbiology, RECEPTOR, Cell Biology, Golgi apparatus, carbohydrates (lipids), 030104 developmental biology, self-correction, biology.protein, SURFACE ORGANIZATION