المؤلفون: Franck Lebrin, Simon Tual-Chalot, Helen M. Arthur, Steven Kroon, Frans Disch, Noël Lamandé, Christine L. Mummery, Damien Dos-Santos-Luis, Jérémy H. Thalgott, Johannes J. Mager, Ton J. Rabelink, Repke J. Snijder, Georgios Galaris, Karine Raymond, Anna E Hosman, Yihai Cao, Samly Srun, Sabrina Martin, Hetty C. de Boer, Diane Bracquart

المساهمون: Institut de Recherche Interdisciplinaire de Grenoble (IRIG), Direction de Recherche Fondamentale (CEA) (DRF (CEA)), Commissariat à l'énergie atomique et aux énergies alternatives (CEA)-Commissariat à l'énergie atomique et aux énergies alternatives (CEA)

المصدر: Circulation
Circulation, American Heart Association, 2018, 138 (23), pp.2698-2712. ⟨10.1161/CIRCULATIONAHA.117.033062⟩
Circulation, 138(23), 2698-2712
Circulation, 2018, 138 (23), pp.2698-2712. ⟨10.1161/CIRCULATIONAHA.117.033062⟩

مصطلحات موضوعية: Adult, Male, 0301 basic medicine, Heterozygote, Activin Receptors, Type II, [SDV]Life Sciences [q-bio], arteriovenous malformation, Neovascularization, Physiologic, Antibodies, Mycoplasma pulmonis, Arteriovenous Malformations, Pathogenesis, Mice, angiogenesis, 03 medical and health sciences, 0302 clinical medicine, Physiology (medical), Animals, Humans, hereditary hemorrhagic telangiectasia, Medicine, vascular endothelial growth factors, Telangiectasia, Skin, Vascular Endothelial Growth Factor Receptor-1, business.industry, Genetic disorder, Retinal Vessels, Mouse Embryonic Stem Cells, ACVRL1, Arteriovenous malformation, Middle Aged, medicine.disease, Mice, Inbred C57BL, Platelet Endothelial Cell Adhesion Molecule-1, Disease Models, Animal, 030104 developmental biology, 030220 oncology & carcinogenesis, cardiovascular system, Cancer research, Female, Telangiectasia, Hereditary Hemorrhagic, pathological, medicine.symptom, Cardiology and Cardiovascular Medicine, business, Haploinsufficiency, Activin Receptors, Type I, Signal Transduction

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c863f2798fab803c6c38e525cf96af34
https://doi.org/10.1161/circulationaha.117.033062

المؤلفون: Thomas Mathivet, Sabrina Martin, Johannes J. Mager, Samly Srun, Bruno Larrivée, Anne Eichmann, Frans Disch, Repke J. Snijder, Christine L. Mummery, Helen M. Arthur, Franck Lebrin, Catarina Freitas, Jean-Leon Thomas, Christiane Bréant, Stieneke van den Brink, Karine Raymond, Cornelis J.J. Westermann

المصدر: Nature Medicine, 16(4), 420

مصطلحات موضوعية: medicine.medical_treatment, Neovascularization, Physiologic, General Biochemistry, Genetics and Molecular Biology, Mural cell, Hemoglobins, Mice, Medicine, Animals, Humans, Telangiectasia, Aged, biology, business.industry, Growth factor, Endothelial Cells, General Medicine, Proto-Oncogene Proteins c-sis, Endoglin, Middle Aged, Null allele, Mice, Mutant Strains, Thalidomide, Disease Models, Animal, medicine.anatomical_structure, Epistaxis, smooth-muscle-cells tgf-beta receptor pdgf-b vascular development angiogenesis pericytes endoglin growth mouse mice, Immunology, Cancer research, biology.protein, Blood Vessels, Telangiectasia, Hereditary Hemorrhagic, medicine.symptom, business, Platelet-derived growth factor receptor, Blood vessel, medicine.drug

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0684a3efcb025ea7ed7e2c42a104cf97
https://pubmed.ncbi.nlm.nih.gov/20619985