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1
المؤلفون: Swarmistha Devi Aribam, Momoko Nakayama, Satoki Ichimura, Kyori Tokuyama, Yuka Hara, Yohsuke Ogawa, Yoshihiro Shimoji, Masahiro Eguchi
المصدر: Journal of Microbiological Methods. 209:106729
مصطلحات موضوعية: Microbiology (medical), Molecular Biology, Microbiology
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2
المؤلفون: Shiro Ikegawa, Tatsuya Furuichi, Satoki Ichimura, Ryutaro Fukumura, Yoichi Gondo, Takuya Murata, Shun Sasaki
المصدر: Experimental Animals. 66:137-144
مصطلحات موضوعية: 0301 basic medicine, Reporter gene, Mutation, General Veterinary, fungi, Mutant, General Medicine, Biology, medicine.disease_cause, Phenotype, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, 030104 developmental biology, 0302 clinical medicine, Mutant protein, 030220 oncology & carcinogenesis, Cancer research, medicine, Missense mutation, Animal Science and Zoology, Gene, Transforming growth factor
URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_________::28d1c8c26f026aa530822a57b6a46deb
https://doi.org/10.1538/expanim.16-0085 -
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المؤلفون: Shigeharu Wakana, Makoto Kimura, Kuniaki Sasaki, Satoki Ichimura, Tomohiro Suzuki, Hiroshi Masuya, Tatsuya Furuichi, Shiro Ikegawa
المصدر: Biochemical and Biophysical Research Communications. 468:86-91
مصطلحات موضوعية: Male, Thanatophoric Dysplasia, Thanatophoric dysplasia, Mutant, Mutation, Missense, Biophysics, Apoptosis, Biology, medicine.disease_cause, Biochemistry, Mice, Chondrocytes, medicine, Animals, Humans, Missense mutation, Growth Plate, Collagen Type II, Molecular Biology, Skeleton, Genetics, Mutation, Endoplasmic reticulum, Platyspondylic lethal skeletal dysplasia, Torrance type, Cell Biology, Endoplasmic Reticulum Stress, medicine.disease, Molecular biology, Mice, Inbred C57BL, Dysplasia, Unfolded Protein Response, Unfolded protein response, Female
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المؤلفون: Satoki, Ichimura, Shun, Sasaki, Takuya, Murata, Ryutaro, Fukumura, Yoichi, Gondo, Shiro, Ikegawa, Tatsuya, Furuichi
المصدر: Experimental Animals
مصطلحات موضوعية: Male, Original, fungi, Mutation, Missense, Camurati-Engelmann Syndrome, Camurati-Engelmann disease, Mice, Inbred C57BL, Transforming Growth Factor beta1, Mice, ENU-mutagenized mice, HEK293 Cells, Phenotype, Amino Acid Substitution, Mice, Inbred DBA, Ethylnitrosourea, TGF-β1, Serine, Animals, Humans, Female, Cysteine, Molecular Targeted Therapy, Genetic Association Studies, Gene Library, Signal Transduction
URL الوصول: https://explore.openaire.eu/search/publication?articleId=pmid________::5c2d7db011f5156def3f5ed54343c5c6
https://pubmed.ncbi.nlm.nih.gov/27928112