المؤلفون: Sünderhauf, Annika, Hicken, Maren, Schlichting, Heidi, Skibbe, Kerstin, Ragab, Mohab, Raschdorf, Annika, Hirose, Misa, Schäffler, Holger, Bokemeyer, Arne, Bettenworth, Dominik, Savitt, Anne G., Perner, Sven, Ibrahim, Saleh, Peerschke, Ellinor I., Ghebrehiwet, Berhane, Derer, Stefanie, Sina, Christian

المصدر: In Cellular and Molecular Gastroenterology and Hepatology 2021 12(1):229-250

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المؤلفون: Raschdorf, Annika, Sünderhauf, Annika, Skibbe, Kerstin, Ghebrehiwet, Berhane, Peerschke, Ellinor I., Sina, Christian, Derer, Stefanie

المصدر: Frontiers in Oncology; 2/8/2021, Vol. 11, pN.PAG-N.PAG, 17p

مصطلحات موضوعية: OXIDATIVE phosphorylation, COLON cancer, SINGLE nucleotide polymorphisms, MITOCHONDRIA, MITOCHONDRIAL proteins, CANCER cell differentiation

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المؤلفون: Skibbe, Kerstin, Ebbert, Karen, Preisker, Sophie, Sünderhauf, Annika, Langenstrassen, Petra, Sina, Christian, Derer, Stefanie

المصدر: In Immunobiology October 2016 221(10):1154-1154

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المؤلفون: Sünderhauf A; Institute of Nutritional Medicine, Molecular Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Skibbe K; Institute of Nutritional Medicine, Molecular Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Preisker S; Institute of Nutritional Medicine, Molecular Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Ebbert K; Institute of Nutritional Medicine, Molecular Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Verschoor A; Institute for Systemic Inflammation Research, University of Lübeck, Lübeck, Germany., Karsten CM; Institute for Systemic Inflammation Research, University of Lübeck, Lübeck, Germany., Kemper C; Institute for Systemic Inflammation Research, University of Lübeck, Lübeck, Germany; Division of Transplant Immunology and Mucosal Biology, Medical Research Council Centre for Transplantation, King's College London, Guy's Hospital, Great Maze Pond, London SE1 9RT, UK; Laboratory of Molecular Immunology and the Immunology Center, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, MD 20892, USA., Huber-Lang M; Institute for Clinical and Experimental Trauma-Immunology, University Hospital Ulm, Ulm, Germany., Basic M; Institute for Laboratory Animal Science and Central Animal Facility, Hannover Medical School, Hannover, Germany., Bleich A; Institute for Laboratory Animal Science and Central Animal Facility, Hannover Medical School, Hannover, Germany., Büning J; 1st Department of Medicine, Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Fellermann K; 1st Department of Medicine, Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Sina C; Institute of Nutritional Medicine, Molecular Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany; 1st Department of Medicine, Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany; 1st Department of Medicine, Section of Nutritional Medicine, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany., Derer S; Institute of Nutritional Medicine, Molecular Gastroenterology, University Hospital Schleswig-Holstein, Campus Lübeck, Lübeck, Germany. Electronic address: Stefanie.derer@uksh.de.

المصدر: Molecular immunology [Mol Immunol] 2017 Oct; Vol. 90, pp. 227-238. Date of Electronic Publication: 2017 Aug 24.

نوع المنشور: Journal Article

بيانات الدورية: Publisher: Pergamon Press Country of Publication: England NLM ID: 7905289 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1872-9142 (Electronic) Linking ISSN: 01615890 NLM ISO Abbreviation: Mol Immunol Subsets: MEDLINE

مواضيع طبية MeSH: Colitis, Ulcerative/*pathology , Complement C3a/*biosynthesis , Complement C3b/*biosynthesis , Epithelial Cells/*metabolism , Intestinal Mucosa/*pathology, Animals ; Bacteria/immunology ; Cell Line ; Colitis, Ulcerative/chemically induced ; Complement C3a/metabolism ; Complement C3b/metabolism ; Dextran Sulfate/toxicity ; Humans ; Inflammation/pathology ; Intestinal Mucosa/immunology ; Lipopolysaccharides/pharmacology ; Mice ; Mice, Inbred C57BL ; Signal Transduction/immunology ; Toll-Like Receptor 1/biosynthesis ; Toll-Like Receptor 2/biosynthesis ; Toll-Like Receptor 4/biosynthesis