المؤلفون: Southerland, EM, Milhorn, DM, Foreman, RD, Linderoth, B, DeJongste, MJL, Armour, JA, Subramanian, V, Singh, M, Singh, K, Ardell, JL

المصدر: American journal of physiology. Heart and circulatory physiology. 292(1):H311-H317

مصطلحات موضوعية: Medicin och hälsovetenskap

URL الوصول: http://kipublications.ki.se/Default.aspx?queryparsed=id:13908441

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المؤلفون: Ardell JL; UCLA Neurocardiology Research Center of Excellence and UCLA Cardiac Arrhythmia Center, Los Angeles, Los Angeles, CA, USA., Nier H; Biomedical Sciences, East Tennessee State University, Johnson City, TN, USA., Hammer M; UCLA Neurocardiology Research Center of Excellence and UCLA Cardiac Arrhythmia Center, Los Angeles, Los Angeles, CA, USA., Southerland EM; Biomedical Sciences, East Tennessee State University, Johnson City, TN, USA., Ardell CL; Biomedical Sciences, East Tennessee State University, Johnson City, TN, USA., Beaumont E; Biomedical Sciences, East Tennessee State University, Johnson City, TN, USA., KenKnight BH; New Ventures, LivaNova plc, Houston, TX, USA., Armour JA; UCLA Neurocardiology Research Center of Excellence and UCLA Cardiac Arrhythmia Center, Los Angeles, Los Angeles, CA, USA.

المصدر: The Journal of physiology [J Physiol] 2017 Nov 15; Vol. 595 (22), pp. 6887-6903. Date of Electronic Publication: 2017 Sep 30.

نوع المنشور: Journal Article; Research Support, Non-U.S. Gov't

بيانات الدورية: Publisher: Cambridge Univ. Press Country of Publication: England NLM ID: 0266262 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1469-7793 (Electronic) Linking ISSN: 00223751 NLM ISO Abbreviation: J Physiol Subsets: MEDLINE

مواضيع طبية MeSH: Heart Rate* , Vagus Nerve Stimulation*, Heart/*physiology , Vagus Nerve/*physiology, Adrenergic beta-Antagonists/pharmacology ; Angiotensin-Converting Enzyme Inhibitors/pharmacology ; Animals ; Benzazepines/pharmacology ; Dogs ; Female ; Heart/innervation ; Ivabradine ; Male ; Muscarinic Antagonists/pharmacology ; Vagus Nerve/drug effects

المؤلفون: Beaumont E; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Wright GL; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Southerland EM; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Li Y; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Chui R; Molecular, Cellular, and Integrative Physiology Program, University of California, Los Angeles, California;, KenKnight BH; Cyberonics, Inc., Houston, Texas; and., Armour JA; UCLA Neurocardiology Research Center of Excellence and UCLA Cardiac Arrhythmia Center, Los Angeles, California., Ardell JL; Molecular, Cellular, and Integrative Physiology Program, University of California, Los Angeles, California; UCLA Neurocardiology Research Center of Excellence and UCLA Cardiac Arrhythmia Center, Los Angeles, California jardell@mednet.ucla.edu.

المصدر: American journal of physiology. Heart and circulatory physiology [Am J Physiol Heart Circ Physiol] 2016 May 15; Vol. 310 (10), pp. H1349-59. Date of Electronic Publication: 2016 Mar 18.

نوع المنشور: Journal Article

بيانات الدورية: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901228 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1539 (Electronic) Linking ISSN: 03636135 NLM ISO Abbreviation: Am J Physiol Heart Circ Physiol Subsets: MEDLINE

مواضيع طبية MeSH: Vagus Nerve Stimulation* , Ventricular Function, Left* , Ventricular Pressure* , Ventricular Remodeling*, Heart/*innervation , Hypertrophy, Left Ventricular/*therapy , Vagus Nerve/*physiopathology, Animals ; Apoptosis ; Disease Models, Animal ; Glycogen Synthase/metabolism ; Guinea Pigs ; Hypertrophy, Left Ventricular/etiology ; Hypertrophy, Left Ventricular/metabolism ; Hypertrophy, Left Ventricular/physiopathology ; Male ; Myocytes, Cardiac/metabolism ; Myocytes, Cardiac/pathology ; Phosphorylation ; Synaptic Transmission ; Time Factors

المؤلفون: Beaumont E; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Southerland EM; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Hardwick JC; Department of Biology, Ithaca College, Ithaca, New York;, Wright GL; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Ryan S; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Li Y; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, KenKnight BH; Cyberonics, Houston, Texas; and., Armour JA; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee; Department of Medicine, University of California Los Angeles Health System, Los Angeles, California., Ardell JL; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee; Department of Medicine, University of California Los Angeles Health System, Los Angeles, California jardell@mednet.ucla.edu.

المصدر: American journal of physiology. Heart and circulatory physiology [Am J Physiol Heart Circ Physiol] 2015 Oct; Vol. 309 (7), pp. H1198-206. Date of Electronic Publication: 2015 Aug 14.

نوع المنشور: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't

بيانات الدورية: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901228 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1539 (Electronic) Linking ISSN: 03636135 NLM ISO Abbreviation: Am J Physiol Heart Circ Physiol Subsets: MEDLINE

مواضيع طبية MeSH: Vagus Nerve Stimulation*, Autonomic Nervous System/*physiopathology , Heart/*innervation , Myocardial Infarction/*physiopathology , Myocytes, Cardiac/*metabolism , Neuronal Plasticity/*physiology , Ventricular Dysfunction, Left/*physiopathology, Animals ; Evoked Potentials ; Glycogenolysis ; Guinea Pigs ; Membrane Potentials ; Norepinephrine/metabolism ; Stroke Volume/physiology ; Synaptic Transmission ; Ventricular Function, Left ; bcl-2-Associated X Protein/metabolism

المؤلفون: Hardwick JC; Department of Biology, Ithaca College, Ithaca, New York; jhardwick@ithaca.edu., Ryan SE; Department of Biology, Ithaca College, Ithaca, New York;, Powers EN; Department of Biology, Ithaca College, Ithaca, New York;, Southerland EM; Department of Biomedical Sciences, Quillen College of Medicine, East Tennessee State University, Johnson City, Tennessee;, Ardell JL; University of California at Los Angeles (UCLA) Neurocardiology Research Center of Excellence, Los Angeles, California; and UCLA Cardiac Arrhythmia Center, Los Angeles, California.

المصدر: American journal of physiology. Regulatory, integrative and comparative physiology [Am J Physiol Regul Integr Comp Physiol] 2015 Jul 15; Vol. 309 (2), pp. R179-88. Date of Electronic Publication: 2015 May 06.

نوع المنشور: Journal Article; Research Support, N.I.H., Extramural

بيانات الدورية: Publisher: American Physiological Society Country of Publication: United States NLM ID: 100901230 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1522-1490 (Electronic) Linking ISSN: 03636119 NLM ISO Abbreviation: Am J Physiol Regul Integr Comp Physiol Subsets: MEDLINE

مواضيع طبية MeSH: Heart/*innervation , Myocardial Infarction/*metabolism , Presynaptic Terminals/*metabolism , Receptor, Angiotensin, Type 1/*metabolism , Receptor, Angiotensin, Type 2/*metabolism , Receptors, Presynaptic/*metabolism, Action Potentials ; Angiotensin II/pharmacology ; Angiotensin II Type 1 Receptor Blockers/pharmacology ; Angiotensin II Type 2 Receptor Blockers/pharmacology ; Animals ; Disease Models, Animal ; Electric Stimulation ; Evoked Potentials ; Guinea Pigs ; Male ; Myocardial Infarction/pathology ; Myocardial Infarction/physiopathology ; Norepinephrine/pharmacology ; Presynaptic Terminals/drug effects ; Receptor, Angiotensin, Type 1/drug effects ; Receptor, Angiotensin, Type 2/drug effects ; Receptors, Presynaptic/antagonists & inhibitors ; Signal Transduction ; Time Factors