المؤلفون: Ning Liang, Anastasius Damdimopoulos, Saioa Goñi, Zhiqiang Huang, Lise-Lotte Vedin, Tomas Jakobsson, Marco Giudici, Osman Ahmed, Matteo Pedrelli, Serena Barilla, Fawaz Alzaid, Arturo Mendoza, Tarja Schröder, Raoul Kuiper, Paolo Parini, Anthony Hollenberg, Philippe Lefebvre, Sven Francque, Luc Van Gaal, Bart Staels, Nicolas Venteclef, Eckardt Treuter, Rongrong Fan

المصدر: Nature Communications, Vol 10, Iss 1, Pp 1-14 (2019)

مصطلحات موضوعية: Science

وصف الملف: electronic resource

Relation: https://doaj.org/toc/2041-1723

URL الوصول: https://doaj.org/article/90508a91d07f4ebf99f099ffd68ebc8f

المؤلفون: Tarja Schröder, Matteo Pedrelli, Luc Van Gaal, Anthony N. Hollenberg, Arturo Mendoza, Philippe Lefebvre, Ning Liang, Saioa Goñi, Zhiqiang Huang, Bart Staels, Raoul Kuiper, Paolo Parini, Serena Barilla, Lise Lotte Vedin, Eckardt Treuter, Nicolas Venteclef, Sven Francque, Tomas Jakobsson, Anastasius Damdimopoulos, Marco Giudici, Rongrong Fan, Fawaz Alzaid, Osman Ahmed

المساهمون: Department of Biosciences and Nutrition [Karolinska Institutet], Karolinska Institutet [Stockholm], Department of Laboratory Medicine [Karolinska Institutet], Centre de Recherche des Cordeliers (CRC (UMR_S_1138 / U1138)), École pratique des hautes études (EPHE), Université Paris sciences et lettres (PSL)-Université Paris sciences et lettres (PSL)-Université Paris Diderot - Paris 7 (UPD7)-Université Paris Descartes - Paris 5 (UPD5)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Sorbonne Université (SU), Weill Medical College of Cornell University [New York], Department of Medicine Huddinge [Karolinska Institutet], Inflammation and Infection Theme [Karolinska University Hospital], Karolinska University Hospital [Stockholm], Récepteurs nucléaires, maladies cardiovasculaires et diabète - U 1011 (RNMCD), Institut Pasteur de Lille, Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille)-Institut National de la Santé et de la Recherche Médicale (INSERM), Department of Gastroenterology and Hepatology, University of Antwerp (UA), Laboratory of Experimental Medicine and Pediatrics, Department of Endocrinology, Diabetology and Metabolism, E.T. was supported by grants from the Center for Innovative Medicine (CIMED) at the Karolinska Institutet, the Swedish Research Council, the Swedish Cancer Society, the Swedish Diabetes Foundation, the Novo Nordisk Foundation, and the European Union FP7 HEALTH project HUMAN. R.F. was supported by the European Foundation for the Study of Diabetes (EFSD)/Lilly research fellowship, Junior Diabetes Wellness Sverige grant and KI research foundation grants. R.K. and T.S. were supported by KI/SL and grants from CIMED. N.L. received a doctoral education grant (KID) from Karolinska Institutet. Z.H. was supported by Chinese Government Scholarship (CSC). N.V. was supported by grants from the French National Agency of Research (CONRAD and PROVIDE), Region Ile de France (CORDDIM), Paris city (EMERGENCE), the French Foundation for Diabetes (SFD), and the European Union H2020 framework (ERC-EpiFAT 725790). The human liver transcriptome study performed by P.L., F.A., N.V., and B.S. was supported by EU [HEPADIP (Contract LSHM-CT-2005-018734) and RESOLVE (Contract FP7-305707)], the European Research Council (ERC Grant Immunobile, contract 694717), Fondation pour la Recherche Médicale (Equipe labellisée, DEQ20150331724) and Agence Nationale pour la Recherche (ANR-10-LBEX-46). B.S. is a recipient of an Advanced ERC Grant (694717), ANR-12-JSV1-0007,CONRAD,Le complexe répresseur SMRT-GPS2: senseur moléculaire de l'inflammation du tissu adipeux dans l'obésité humaine(2012), ANR-15-CE14-0029,PROVIDE,Compréhension des mécanismes inflammatoires protecteurs dans le diabète(2015), European Project: 694717,H2020-EU.1.1. - EXCELLENT SCIENCE - European Research Council (ERC) ,ImmunoBile(2016), École Pratique des Hautes Études (EPHE), Réseau International des Instituts Pasteur (RIIP)-Réseau International des Instituts Pasteur (RIIP)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université de Lille-Centre Hospitalier Régional Universitaire [Lille] (CHRU Lille), Derudas, Marie-Hélène, Jeunes Chercheuses et Jeunes Chercheurs - Le complexe répresseur SMRT-GPS2: senseur moléculaire de l'inflammation du tissu adipeux dans l'obésité humaine - - CONRAD2012 - ANR-12-JSV1-0007 - JC - VALID, Compréhension des mécanismes inflammatoires protecteurs dans le diabète - - PROVIDE2015 - ANR-15-CE14-0029 - AAPG2015 - VALID, Bile acid, immune-metabolism, lipid and glucose homeostasis - ImmunoBile - - H2020-EU.1.1. - EXCELLENT SCIENCE - European Research Council (ERC) 2016-09-01 - 2021-08-31 - 694717 - VALID

المصدر: Nature Communications
Nature Communications, Nature Publishing Group, 2019, 10 (1), pp.1684. ⟨10.1038/s41467-019-09524-z⟩
Nature Communications, 2019, 10 (1), pp.1684. ⟨10.1038/s41467-019-09524-z⟩
Nature communications
Nature Communications, Vol 10, Iss 1, Pp 1-14 (2019)

مصطلحات موضوعية: Male, 0301 basic medicine, Biopsy, [SDV]Life Sciences [q-bio], Datasets as Topic, General Physics and Astronomy, 02 engineering and technology, MESH: Mice, Knockout, Epigenesis, Genetic, MESH: Hepatocytes, Mice, MESH: Biopsy, Non-alcoholic Fatty Liver Disease, MESH: Animals, MESH: Datasets as Topic, MESH: Epigenesis, Genetic, lcsh:Science, MESH: PPAR alpha, MESH: Lipid Metabolism, Mice, Knockout, Multidisciplinary, Chemistry, Fatty liver, Intracellular Signaling Peptides and Proteins, 021001 nanoscience & nanotechnology, 3. Good health, GPS2, [SDV] Life Sciences [q-bio], Liver, Cistrome, MESH: HEK293 Cells, MESH: Fibrosis, Disease Progression, MESH: Disease Progression, Peroxisome proliferator-activated receptor alpha, 0210 nano-technology, Engineering sciences. Technology, Science, Diet, High-Fat, Article, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, MESH: Mice, Inbred C57BL, MESH: Intracellular Signaling Peptides and Proteins, medicine, Animals, Humans, PPAR alpha, MESH: Mice, MESH: Humans, MESH: Non-alcoholic Fatty Liver Disease, General Chemistry, Lipid Metabolism, medicine.disease, HDAC3, Fibrosis, MESH: Male, Mice, Inbred C57BL, Disease Models, Animal, MESH: Diet, High-Fat, HEK293 Cells, 030104 developmental biology, Nuclear receptor, Hepatocytes, Cancer research, lcsh:Q, Steatosis, Steatohepatitis, MESH: Disease Models, Animal, MESH: Liver

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URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bda4f2121737c067e7c9c5471aeeb6a3
https://www.hal.inserm.fr/inserm-02154801/document