المؤلفون: Carlos Spuch, Cristina Ugalde, Eva Carro, José Antonio Molina, Isidro Ferrer, Félix Bermejo-Pareja, Desire E. Antequera, Te O. Vargas

المصدر: Neurobiology of Disease, Vol 36, Iss 1, Pp 42-50 (2009)

مصطلحات موضوعية: Male, Cytoplasm, Apoptosis, Choroid plexus, Amyloid beta-Protein Precursor, Mice, RNA interference, Apoptotic mitochondrial changes, Cells, Cultured, Electron Transport Complex II, Brain, Middle Aged, Alzheimer's disease, musculoskeletal system, Mitochondria, Cell biology, medicine.anatomical_structure, Neurology, Cerebral cortex, Female, RNA Interference, Genetically modified mouse, Programmed cell death, Transgene, Genetic Vectors, Enzyme-Linked Immunosorbent Assay, Mice, Transgenic, DNA Fragmentation, macromolecular substances, Biology, Transfection, lcsh:RC321-571, Electron Transport Complex IV, Alzheimer Disease, mental disorders, Presenilin-1, medicine, Animals, Humans, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, Gelsolin, Aged, Amyloid beta-Peptides, Epithelial Cells, NADH Dehydrogenase, Nitric oxide, Lentiviral vectors, Molecular biology, Peptide Fragments, nervous system diseases, Disease Models, Animal, Animals, Newborn, Mitochondrial dysfunction

URL الوصول: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::89e159423a60b9293f4599bf88a971ec
http://www.sciencedirect.com/science/article/pii/S0969996109001624