يعرض 1 - 9 نتائج من 9 نتيجة بحث عن '"pathology [Movement Disorders]"', وقت الاستعلام: 1.22s تنقيح النتائج
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    المصدر: Experimental neurology 335, 113513-(2021). doi:10.1016/j.expneurol.2020.113513

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    المصدر: NeuroImage : Clinical
    NeuroImage: Clinical, Vol 14, Iss C, Pp 641-647 (2017)
    Stewart, Jill Campbell; Dewanjee, Pritha; Tran, George; Quinlan, Erin Burke; Dodakian, Lucy; McKenzie, Alison; et al.(2017). Role of corpus callosum integrity in arm function differs based on motor severity after stroke.. NeuroImage. Clinical, 14, 641-647. UC Irvine: Institute for Clinical and Translational Science. Retrieved from: http://www.escholarship.org/uc/item/3p2508n0

    وصف الملف: application/pdf

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    المصدر: Cell Reports, Vol 17, Iss 4, Pp 1071-1086 (2016)
    Cell reports 17(4), 1071-1086 (2016). doi:10.1016/j.celrep.2016.09.058
    Cell Reports

    مصطلحات موضوعية: Male, Aging, genetics [SKP Cullin F-Box Protein Ligases], Transcription, Genetic, Muscle Proteins, Apoptosis, metabolism [Hippocampus], genetics [Muscle Proteins], Hippocampus, pathology [Ataxia], metabolism [Forkhead Box Protein O3], genetics [Homeostasis], Mice, pathology [Aging], genetics [Parkinson Disease], metabolism [Ubiquitin], complications [Movement Disorders], Fbxo32 protein, mouse, Homeostasis, lcsh:QH301-705.5, Mice, Knockout, protein homeostasis, Movement Disorders, Forkhead Box Protein O3, neurodegeneration, Brain, pathology [Nerve Degeneration], Parkinson Disease, genetics [Ataxia], metabolism [Autophagosomes], Up-Regulation, metabolism [Acyltransferases], Protein Binding, Proteasome Endopeptidase Complex, autophagy, metabolism [Muscle Proteins], FBXO32 protein, human, genetics [Mutation], Article, FBXO32, endophilin, Autophagy, deficiency [Acyltransferases], Animals, Humans, endocytosis, FoxO3 protein, mouse, ddc:610, metabolism [Proteasome Endopeptidase Complex], SKP Cullin F-Box Protein Ligases, Endophilin-A, Ubiquitin-Proteasome System, Ubiquitin, ataxia, Autophagosomes, metabolism [SKP Cullin F-Box Protein Ligases], pathology [Movement Disorders], pathology [Parkinson Disease], complications [Nerve Degeneration], pathology [Hippocampus], lcsh:Biology (General), metabolism [Brain], Mutation, Nerve Degeneration, Parkinson’s disease, genetics [Forkhead Box Protein O3], 2-acylglycerophosphate acyltransferase, next-generation sequencing, ubiquitin-proteasome system, Acyltransferases, HeLa Cells

    وصف الملف: Print-Electronic; application/pdf

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    المصدر: Neurobiology of disease 74, 76-88 (2015). doi:10.1016/j.nbd.2014.11.003
    Neurobiology of Disease, Vol 74, Iss, Pp 76-88 (2015)

    مصطلحات موضوعية: Methyltransferase, PRMT, genetics [Transcription Factor TFIID], Tnpo protein, Drosophila, Art1 protein, Drosophila, physiology [Active Transport, Cell Nucleus], Eye, Animals, Genetically Modified, genetics [Drosophila Proteins], metabolism [Transcription Factor TFIID], Drosophila Proteins, genetics [RNA-Binding Proteins], pathology [Eye], metabolism [RNA-Binding Protein FUS], Motor Neurons, Gene knockdown, Movement Disorders, metabolism [Karyopherins], physiopathology [Movement Disorders], pathology [Neurodegenerative Diseases], RNA-Binding Proteins, Neurodegenerative Diseases, Methylation, Cell biology, Drosophila melanogaster, Neurology, Gene Knockdown Techniques, Caz, RNA Interference, FTLD, pathology [Motor Neurons], Transportin, Transgene, Active Transport, Cell Nucleus, metabolism [Drosophila Proteins], Biology, Karyopherins, metabolism [RNA-Binding Proteins], lcsh:RC321-571, physiology [DNA Methylation], ddc:570, metabolism [Eye], caz protein, Drosophila, NLS, Animals, Humans, lcsh:Neurosciences. Biological psychiatry. Neuropsychiatry, metabolism [Methyltransferases], FUS, physiopathology [Neurodegenerative Diseases], Wild type, metabolism [Motor Neurons], Methyltransferases, pathology [Movement Disorders], FUS protein, human, DNA Methylation, Disease Models, Animal, genetics [Methyltransferases], Mutation, Cancer research, RNA-Binding Protein FUS, Transcription Factor TFIID, Nuclear transport, ALS, genetics [RNA-Binding Protein FUS], Nuclear localization sequence

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    المصدر: Neuroscience 227, 313-326 (2012). doi:10.1016/j.neuroscience.2012.09.080

    مصطلحات موضوعية: Male, 1,1-dimethylbutyl-1-deoxy-Delta(9)-THC, Cannabinoid receptor, therapeutic use [Cannabinoids], Time Factors, medicine.medical_treatment, drug effects [Gene Expression Regulation], etiology [Brain Infarction], therapeutic use [Quinoxalines], Rats, Sprague-Dawley, Receptor, Cannabinoid, CB2, complications [Infarction, Middle Cerebral Artery], Receptor, Cannabinoid, CB1, Cannabinoid receptor type 2, anandamide, therapeutic use [Indans], Movement Disorders, Microglia, General Neuroscience, etiology [Movement Disorders], pharmacology [Polyunsaturated Alkamides], Brain, Infarction, Middle Cerebral Artery, pharmacology [Endocannabinoids], Ectodysplasins, therapeutic use [Neuroprotective Agents], Endocannabinoid system, ACEA 1011, metabolism [Receptor, Cannabinoid, CB1], medicine.anatomical_structure, Neuroprotective Agents, Indans, lipids (amino acids, peptides, and proteins), drug effects [Brain], drug effects [Psychomotor Performance], Brain Infarction, Agonist, pharmacology [Quinoxalines], medicine.drug_class, Polyunsaturated Alkamides, prevention & control [Brain Infarction], Ischemia, Arachidonic Acids, Biology, pharmacology [Arachidonic Acids], drug therapy [Movement Disorders], Neuroprotection, Quinoxalines, Glial Fibrillary Acidic Protein, medicine, Animals, ddc:610, metabolism [Receptor, Cannabinoid, CB2], Dose-Response Relationship, Drug, Cannabinoids, metabolism [Glial Fibrillary Acidic Protein], pathology [Movement Disorders], medicine.disease, Rats, therapeutic use [Sulfonic Acids], Disease Models, Animal, Gene Expression Regulation, nervous system, metabolism [Brain], metabolism [Ectodysplasins], Leukocyte Common Antigens, Cannabinoid, Sulfonic Acids, Neuroscience, metabolism [Leukocyte Common Antigens], Psychomotor Performance, 3-(2-hydroxymethylindanyl-4-oxy)phenyl-4,4,4-trifluoro-1-sulfonate, Endocannabinoids

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