يعرض 1 - 3 نتائج من 3 نتيجة بحث عن '"pathology [Myocytes, Cardiac]"', وقت الاستعلام: 0.90s تنقيح النتائج
  1. 1

    المصدر: The journal of clinical investigation 124(5), 2136-2146 (2014). doi:10.1172/JCI70577

  2. 2

    المصدر: Cell reports 25(3), 561-570.e6 (2018). doi:10.1016/j.celrep.2018.09.057
    Cell Reports
    Cell Reports, Vol 25, Iss 3, Pp 561-570.e6 (2018)

    مصطلحات موضوعية: 0301 basic medicine, Mitochondrial ROS, Respiratory chain, Tafazzin, Mitochondrion, pathology [Mitochondria], Hif1 alpha, Mice, chemistry.chemical_compound, Cardiolipin, Myocytes, Cardiac, metabolism [Reactive Oxygen Species], metabolism [Transcription Factors], genetics [Cardiolipins], Hypoxia, lcsh:QH301-705.5, Cells, Cultured, pathology [Myocytes, Cardiac], Mice, Knockout, biology, NF-kappa B, High-Throughput Nucleotide Sequencing, ROS, Barth syndrome, genetics [Transcription Factors], pathology [Induced Pluripotent Stem Cells], Mitochondria, Cell biology, metabolism [Induced Pluripotent Stem Cells], NF-κB signaling, metabolism [NF-kappa B], medicine.symptom, metabolism [Biomarkers], Signal Transduction, Cell signaling, Cardiolipins, respiratory chain, Induced Pluripotent Stem Cells, metabolism [Barth Syndrome], Mice, Transgenic, Article, General Biochemistry, Genetics and Molecular Biology, 03 medical and health sciences, lipid, genetics [Hypoxia-Inducible Factor 1, alpha Subunit], Taz protein, mouse, medicine, Animals, pathology [Barth Syndrome], Humans, ddc:610, metabolism [Hypoxia-Inducible Factor 1, alpha Subunit], metabolism [Cardiolipins], Hypoxia (medical), metabolism [Mitochondria], Hypoxia-Inducible Factor 1, alpha Subunit, medicine.disease, physiology [Transcription Factors], metabolism [Hypoxia], Mice, Inbred C57BL, TAZ protein, human, genetics [Barth Syndrome], 030104 developmental biology, lcsh:Biology (General), Gene Expression Regulation, pathology [Hypoxia], chemistry, Barth Syndrome, metabolism [Myocytes, Cardiac], biology.protein, cardiolipin, Reactive Oxygen Species, Acyltransferases, Biomarkers, Transcription Factors

    وصف الملف: application/pdf; application/vnd.openxmlformats-officedocument.spreadsheetml.sheet

  3. 3

    المساهمون: Institut de Génomique Fonctionnelle (IGF), Université de Montpellier (UM)-Université Montpellier 1 (UM1)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Université Montpellier 2 - Sciences et Techniques (UM2)-Centre National de la Recherche Scientifique (CNRS), Experimental Neuropediatrics, Universitaetsklinikum Hamburg-Eppendorf = University Medical Center Hamburg-Eppendorf [Hamburg] (UKE), Department of Cellular and Integrative Physiology, Institut de génétique humaine (IGH), Université de Montpellier (UM)-Centre National de la Recherche Scientifique (CNRS), Center for Experimental Medicine, Department of Experimental Pharmacology and Toxicology, Institut de Biologie du Développement de Marseille (IBDM), Aix Marseille Université (AMU)-Centre National de la Recherche Scientifique (CNRS), Cardiovascular Research Center Hamburg (CVRC), University Hamburg, Department of Pharmacology, University of Minnesota [Twin Cities] (UMN), University of Minnesota System-University of Minnesota System, Université Montpellier 1 (UM1)-Université Montpellier 2 - Sciences et Techniques (UM2)-Institut National de la Santé et de la Recherche Médicale (INSERM)-Centre National de la Recherche Scientifique (CNRS)

    المصدر: Nature Communications 5(1), 4664 (2014). doi:10.1038/ncomms5664
    Nature Communications
    Nature Communications, Nature Publishing Group, 2014, 5, pp.4664. ⟨10.1038/ncomms5664⟩
    Nature communications
    Nature Communications, 2014, 5, pp.4664. ⟨10.1038/ncomms5664⟩

    مصطلحات موضوعية: Male, Patch-Clamp Techniques, Potassium Channels, sinoatrial node, Xenopus, General Physics and Astronomy, Muscle Proteins, 030204 cardiovascular system & hematology, genetics [Muscle Proteins], physiology [Oocytes], Mice, 0302 clinical medicine, Kcnj5 protein, mouse, Heart Rate, Pregnancy, Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels, Myocytes, Cardiac, Ivabradine, ComputingMilieux_MISCELLANEOUS, Calcium signaling, pathology [Myocytes, Cardiac], 0303 health sciences, Multidisciplinary, genetics [Potassium Channels], ion channels, pharmacology [Benzazepines], drug therapy [Arrhythmias, Cardiac], genetics [G Protein-Coupled Inwardly-Rectifying Potassium Channels], metabolism [Potassium Channels], Potassium channel, 3. Good health, HCN4 protein, human, medicine.anatomical_structure, Female, ddc:500, drug effects [Heart Rate], medicine.drug, medicine.medical_specialty, pacemaker activity, metabolism [Muscle Proteins], Mice, Transgenic, Biology, arrhythmia, General Biochemistry, Genetics and Molecular Biology, Article, 03 medical and health sciences, genetics [Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels], Internal medicine, Heart rate, medicine, metabolism [G Protein-Coupled Inwardly-Rectifying Potassium Channels], Gene silencing, Animals, Humans, Calcium Signaling, 030304 developmental biology, [SDV.GEN]Life Sciences [q-bio]/Genetics, physiopathology [Arrhythmias, Cardiac], Sinoatrial node, Cardiac arrhythmia, Arrhythmias, Cardiac, General Chemistry, Benzazepines, electrophysiology, Mice, Inbred C57BL, Autonomic nervous system, Disease Models, Animal, Endocrinology, genetics [Arrhythmias, Cardiac], metabolism [Hyperpolarization-Activated Cyclic Nucleotide-Gated Channels], G Protein-Coupled Inwardly-Rectifying Potassium Channels, Oocytes, metabolism [Myocytes, Cardiac], genetics [Calcium Signaling], Neuroscience