المؤلفون: Kroon, Tobias, Harms, M., Maurer, S., Bonnet, Laurianne, Alexandersson, Ida, Lindblom, A., Ahnmark, A., Nilsson, Daniel, Gennemark, P., O'Mahony, G., Osinski, V., McNamara, C., Boucher, Jeremie

المصدر: Molecular Metabolism. 36

مصطلحات موضوعية: Clinical Medicine, Klinisk medicin, Beige adipocytes, Brown adipocytes, FGF21, PPAR, Thermogenesis, UCP1, aleglitazar, chiglitazar, fatty acid binding protein 4, fenofibrate, fibroblast growth factor 21, imiglitazar, insulin, messenger RNA, muraglitazar, neutral insulin, peroxisome proliferator activated receptor alpha, peroxisome proliferator activated receptor gamma, pirinixic acid, ragaglitazar, reglitazar, rosiglitazone, saroglitazar, tesaglitazar, uncoupling protein 1, animal cell, animal experiment, animal model, animal tissue, Article, body weight loss, brown adipocyte, brown adipose tissue, Cidea gene, controlled study, drug dose comparison, drug mechanism, drug megadose, Ehhadh gene, energy expenditure, Fabp4 gene, fatty liver, female, gene, gene expression, high-fat diet-induced fatty liver, human, human cell, in vitro study, in vivo study, insulin sensitivity, lean body weight, low drug dose, male, mouse, nonhuman, obesity, Pdk4 gene, priority journal, proadipocyte, protein blood level, protein expression, protein function, Ucp1 gene, white adipocyte, white adipose tissue

URL الوصول: https://gup.ub.gu.se/publication/294738