يعرض 1 - 1 نتائج من 1 نتيجة بحث عن '"therapeutic use [Quinoxalines]"', وقت الاستعلام: 0.79s تنقيح النتائج
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    المصدر: Neuroscience 227, 313-326 (2012). doi:10.1016/j.neuroscience.2012.09.080

    مصطلحات موضوعية: Male, 1,1-dimethylbutyl-1-deoxy-Delta(9)-THC, Cannabinoid receptor, therapeutic use [Cannabinoids], Time Factors, medicine.medical_treatment, drug effects [Gene Expression Regulation], etiology [Brain Infarction], therapeutic use [Quinoxalines], Rats, Sprague-Dawley, Receptor, Cannabinoid, CB2, complications [Infarction, Middle Cerebral Artery], Receptor, Cannabinoid, CB1, Cannabinoid receptor type 2, anandamide, therapeutic use [Indans], Movement Disorders, Microglia, General Neuroscience, etiology [Movement Disorders], pharmacology [Polyunsaturated Alkamides], Brain, Infarction, Middle Cerebral Artery, pharmacology [Endocannabinoids], Ectodysplasins, therapeutic use [Neuroprotective Agents], Endocannabinoid system, ACEA 1011, metabolism [Receptor, Cannabinoid, CB1], medicine.anatomical_structure, Neuroprotective Agents, Indans, lipids (amino acids, peptides, and proteins), drug effects [Brain], drug effects [Psychomotor Performance], Brain Infarction, Agonist, pharmacology [Quinoxalines], medicine.drug_class, Polyunsaturated Alkamides, prevention & control [Brain Infarction], Ischemia, Arachidonic Acids, Biology, pharmacology [Arachidonic Acids], drug therapy [Movement Disorders], Neuroprotection, Quinoxalines, Glial Fibrillary Acidic Protein, medicine, Animals, ddc:610, metabolism [Receptor, Cannabinoid, CB2], Dose-Response Relationship, Drug, Cannabinoids, metabolism [Glial Fibrillary Acidic Protein], pathology [Movement Disorders], medicine.disease, Rats, therapeutic use [Sulfonic Acids], Disease Models, Animal, Gene Expression Regulation, nervous system, metabolism [Brain], metabolism [Ectodysplasins], Leukocyte Common Antigens, Cannabinoid, Sulfonic Acids, Neuroscience, metabolism [Leukocyte Common Antigens], Psychomotor Performance, 3-(2-hydroxymethylindanyl-4-oxy)phenyl-4,4,4-trifluoro-1-sulfonate, Endocannabinoids