دورية أكاديمية
أعرض في EDS

The Modifier Model of Autism and Social Development in Higher Functioning Children.

التفاصيل البيبلوغرافية
العنوان: The Modifier Model of Autism and Social Development in Higher Functioning Children.
المؤلفون: Mundy PC; University of Miami, Department of Psychology, Marino Autism Research Institute., Henderson HA, Inge AP, Coman DC
المصدر: Research and practice for persons with severe disabilities : the journal of TASH [Res Pract Persons Severe Disabl] 2007 Jun 01; Vol. 32 (2), pp. 124-139.
نوع المنشور: Journal Article
اللغة: English
بيانات الدورية: Publisher: Sage Publications Country of Publication: United States NLM ID: 101151385 Publication Model: Print Cited Medium: Print ISSN: 1540-7969 (Print) Linking ISSN: 15407969 NLM ISO Abbreviation: Res Pract Persons Severe Disabl Subsets: PubMed not MEDLINE
أسماء مطبوعة: Publication: Thousand Oaks, CA : Sage Publications
Original Publication: Baltimore, Md. : TASH, c2002-
مستخلص: The study of phenotypic variability in social impairments and comorbid emotional disorders in autism is important because it provides information on phenotypic differences that currently complicate diagnosis, research, and treatment of this disorder. Currently, though, relatively little is known about the processes that contribute to individual differences in social impairments and comorbidity in autism. In this paper, we present a model that suggests modifier processes (MPs), which are not necessarily specific to the syndrome refractor alter the expression of autism and contribute to fundamental behavioral and psychological differences in children diagnosed with this disorder. One MPs involves the somewhat surprising tendency of some children with higher functioning autism (HFA) to make attributions about other peoples thoughts, although they have social cognitive deficits Just as in other children, the attributions of children with HFA are linked to some of their behavioral problems Another MP involves the influence of differences in motivation associated with the behavioral activation and inhibition systems that can be assessed with measures of anterior EEG asymmetry. This dimension of motivation may be linked to how active but inappropriate and withdrawn children with HFA may appear. Third, differences in the self-monitoring of errors among children with HFA appear to be related to individual differences in IQ and social symptom severity in these children. The possible role of these MPs in diagnostic subgroups and differences in treatment responses among children with HFA are discussed. In addition, the role of MPs in understanding the effects associated with specific genetic functions in autism, such as those associated with the serotonin transporter gene (5-HTTLPR), is discussed. A conclusion of this paper is that the varied expression of autism may require that we understand how autism interacts with other non-syndrome-specific processes that are related to individual differences in all people.
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معلومات مُعتمدة: R01 MH071273 United States MH NIMH NIH HHS; R01 MH071273-03 United States MH NIMH NIH HHS
تواريخ الأحداث: Date Created: 20091110 Latest Revision: 20240312
رمز التحديث: 20240312
مُعرف محوري في PubMed: PMC2773555
DOI: 10.2511/rpsd.32.2.124
PMID: 19898685
قاعدة البيانات: MEDLINE
الوصف
تدمد:1540-7969
DOI:10.2511/rpsd.32.2.124